Dilated Cardiomyopathy Secondary to Toxicity from Anthracycline Chemotherapy in a Patient with a History of Acute Lymphoblastic Leukemia: A Case Report Article Swipe
Physician assigned to the Internal Medicine Service at the Zone 3 General Hospital, Jesús María, Aguascalientes, Mexican Social Security Institute.
,
Miguel Ángel Galindo López
,
Fernando Ortiz-Anaya
,
Four-year resident physician in the Internal Medicine Service at the Zone 3 General Hospital, Jesús María, Aguascalientes, Mexican Social Security Institute.
·
YOU?
·
· 2025
· Open Access
·
· DOI: https://doi.org/10.47191/ijmscrs/v5-i11-03
YOU?
·
· 2025
· Open Access
·
· DOI: https://doi.org/10.47191/ijmscrs/v5-i11-03
Dilated cardiomyopathy is defined by presence of left ventricular or biventricular dilation and left systolic dysfunction in absence of hypertension, coronary disease, or valvular disease that would justify it. Its causes can be divided into two large groups: those of genetic origin and those of nongenetic origin, which include infectious, toxic, and autoimmune disease causes, among others. Its natural course varies significantly based on the different etiologies and its diagnosis can influence the disease's management and prognosis. Clinical case: 29-year-old male, with a history of having suffered from Acute Lymphoblastic Leukemia at the age of 4 with complete remission of the disease demonstrated at that time by bone marrow aspirate and immunophenotype, having been treated with anthracyclines. On this occasion, he attended due to the presence of sudden onset dyspnea and hemoptysis, for which in the first instance, due to the previously mentioned history, an attempt was made to rule out pulmonary thromboembolism with chest Angio tomography, which was corroborated by said study and demonstrating the presence of two intracavitary thrombi. Therefore, a transthoracic echocardiogram was performed, highlighting severe dilation of the left ventricle. Management for heart failure was adjusted based on structural alteration demonstrated by echocardiography, with favorable clinical evolution. Conclusions: The natural history of dilated cardiomyopathy is not easy to establish due to the wide heterogeneity of its etiology and the different rates of progression depending on it. In some cases, there may be functional recovery after acute myocardial damage, for example, in cardiomyopathy induced by tachycardia or in that induced by cardiotoxic drugs. In other cases, stabilization of the disease and systolic dysfunction and, in others, sudden death may be observed.
Related Topics
Concepts
Medicine
Cardiology
Internal medicine
Natural history
Dilated cardiomyopathy
Cardiomyopathy
Etiology
Heart failure
Anthracycline
Heart disease
Chemotherapy
Ventricular tachycardia
Family history
Lymphoblastic Leukemia
Disease
Pulmonary Veno-Occlusive Disease
valvular heart disease
Progressive disease
Tachycardia
Right heart failure
Sinus tachycardia
Myocarditis
Pulmonary hypertension
Pulmonary embolism
Transthoracic echocardiogram
Acute coronary syndrome
Metadata
- Type
- article
- Landing Page
- https://doi.org/10.47191/ijmscrs/v5-i11-03
- https://ijmscrs.com/index.php/ijmscrs/article/download/2521/2189
- OA Status
- hybrid
- OpenAlex ID
- https://openalex.org/W7105845890
All OpenAlex metadata
Raw OpenAlex JSON
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https://openalex.org/W7105845890Canonical identifier for this work in OpenAlex
- DOI
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https://doi.org/10.47191/ijmscrs/v5-i11-03Digital Object Identifier
- Title
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Dilated Cardiomyopathy Secondary to Toxicity from Anthracycline Chemotherapy in a Patient with a History of Acute Lymphoblastic Leukemia: A Case ReportWork title
- Type
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articleOpenAlex work type
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2025Year of publication
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2025-11-15Full publication date if available
- Authors
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Physician assigned to the Internal Medicine Service at the Zone 3 General Hospital, Jesús María, Aguascalientes, Mexican Social Security Institute., Miguel Ángel Galindo López, Fernando Ortiz-Anaya, Four-year resident physician in the Internal Medicine Service at the Zone 3 General Hospital, Jesús María, Aguascalientes, Mexican Social Security Institute.List of authors in order
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https://doi.org/10.47191/ijmscrs/v5-i11-03Publisher landing page
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https://ijmscrs.com/index.php/ijmscrs/article/download/2521/2189Direct link to full text PDF
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hybridOpen access status per OpenAlex
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https://ijmscrs.com/index.php/ijmscrs/article/download/2521/2189Direct OA link when available
- Concepts
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Medicine, Cardiology, Internal medicine, Natural history, Dilated cardiomyopathy, Cardiomyopathy, Etiology, Heart failure, Anthracycline, Heart disease, Chemotherapy, Ventricular tachycardia, Family history, Lymphoblastic Leukemia, Disease, Pulmonary Veno-Occlusive Disease, valvular heart disease, Progressive disease, Tachycardia, Right heart failure, Sinus tachycardia, Myocarditis, Pulmonary hypertension, Pulmonary embolism, Transthoracic echocardiogram, Acute coronary syndromeTop concepts (fields/topics) attached by OpenAlex
- Cited by
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0Total citation count in OpenAlex
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| abstract_inverted_index.acute | 239 |
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| abstract_inverted_index.based | 62, 190 |
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| abstract_inverted_index.severe | 178 |
| abstract_inverted_index.sudden | 127, 269 |
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| abstract_inverted_index.causes, | 54 |
| abstract_inverted_index.damage, | 241 |
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| abstract_inverted_index.disease | 24, 53, 101, 262 |
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| abstract_inverted_index.Clinical | 77 |
| abstract_inverted_index.Leukemia | 90 |
| abstract_inverted_index.adjusted | 189 |
| abstract_inverted_index.aspirate | 109 |
| abstract_inverted_index.attended | 121 |
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| abstract_inverted_index.dilation | 11, 179 |
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| abstract_inverted_index.recovery | 237 |
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| abstract_inverted_index.thrombi. | 170 |
| abstract_inverted_index.valvular | 23 |
| abstract_inverted_index.depending | 227 |
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| abstract_inverted_index.establish | 212 |
| abstract_inverted_index.favorable | 198 |
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| abstract_inverted_index.remission | 98 |
| abstract_inverted_index.Management | 184 |
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| abstract_inverted_index.alteration | 193 |
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| abstract_inverted_index.etiologies | 66 |
| abstract_inverted_index.evolution. | 200 |
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| abstract_inverted_index.management | 74 |
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| abstract_inverted_index.prognosis. | 76 |
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| abstract_inverted_index.ventricle. | 183 |
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| abstract_inverted_index.hemoptysis, | 131 |
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| abstract_inverted_index.tachycardia | 248 |
| abstract_inverted_index.tomography, | 156 |
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| abstract_inverted_index.highlighting | 177 |
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| abstract_inverted_index.thromboembolism | 152 |
| abstract_inverted_index.immunophenotype, | 111 |
| abstract_inverted_index.echocardiography, | 196 |
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