HIPOGONADISMO HIPERGONADOTRÓFICO: UM ESTUDO SOBRE AS PRINCIPAIS SÍNDROMES DA PUBERDADE TARDIA Article Swipe
Maria Isabel Giusti Moreira
,
Julia Bartolomeo Siqueira
,
Larissa Zuanetti Silva
,
Letícia Caetano da Silva
,
Bruna Bonifácio
,
Vitorya Vanini Cardoso
,
Rene Coulaud Santos da Costa Cruz
,
Antônio Job Torres del Fiaco
,
Adriana Silva Lima
,
H.S. Costa
,
David Viana Martín
,
Rômulo Batista Rodrígues
,
Lays Figueira Castelo Branco
,
Gabriel Gade Martins Mesquita
,
A. Miranda
,
Maria Júlia Ribeiro de Oliveira
,
Gabriella Rennisy de Sousa Silva
,
Márcio Silva da Conceição
,
Giovanna Larsen Yamaki
,
Emanuele Souza Dias
,
Ana Paula Mariano Kerber
,
Heladia García
,
Daniela Celis Severgnini
·
YOU?
·
· 2025
· Open Access
·
· DOI: https://doi.org/10.36557/2674-8169.2025v7n3p192-203
YOU?
·
· 2025
· Open Access
·
· DOI: https://doi.org/10.36557/2674-8169.2025v7n3p192-203
O hipogonadismo hipergonadotrófico (HH) é um distúrbio endocrinológico caracterizado pela falência gonadal primária, em que ocorre uma deficiência na produção de hormônios sexuais pelas gônadas, o que leva a um aumento compensatório dos níveis de gonadotrofinas. A puberdade tardia é um atraso no desenvolvimento das características sexuais secundárias e na maturação sexual, com base na avaliação de parâmetros como o crescimento, o desenvolvimento mamário nas mulheres e o aumento do volume testicular nos homens A relevância deste estudo reside na necessidade crescente de diagnóstico e manejo adequados do hipogonadismo hipergonadotrófico, especialmente devido ao impacto significativo que a puberdade tardia pode ter na qualidade de vida dos indivíduos afetados. Trata-se de uma revisão sistemática da literatura, a qual investigou sobre as formas clínicas da puberdade tardia relacionadas ao hipogonadismo hipergonadotrófico, pela coleta de dados nas plataformas PubMed, LILACS, Periódicos CAPES, EMBASE e Scielo, dos últimos 5 anos. Assim, o HH é um distúrbio endócrino complexo, manifestado pela falha nas gônadas e compensada por níveis elevados de gonadotrofinas (FSH e LH). Esse quadro clínico se reflete em puberdade tardia ou incompleta, sendo responsável por uma série de síndromes genéticas que afetam tanto os homens quanto as mulheres. As principais condições associadas ao hipogonadismo hipergonadotrófico incluem a Síndrome de Klinefelter, a Síndrome de Turner, a Síndrome de Swyer, o Hipogonadismo Hipergonadotrófico Idiopático e a Disgenesia Gonadal. Cada uma dessas síndromes apresenta características clínicas, diagnósticas e terapêuticas específicas, refletindo a diversidade dos mecanismos patológicos subjacentes.
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- OA Status
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All OpenAlex metadata
Raw OpenAlex JSON
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https://openalex.org/W4408489087Canonical identifier for this work in OpenAlex
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https://doi.org/10.36557/2674-8169.2025v7n3p192-203Digital Object Identifier
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HIPOGONADISMO HIPERGONADOTRÓFICO: UM ESTUDO SOBRE AS PRINCIPAIS SÍNDROMES DA PUBERDADE TARDIAWork title
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articleOpenAlex work type
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ptPrimary language
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2025Year of publication
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2025-03-05Full publication date if available
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Maria Isabel Giusti Moreira, Julia Bartolomeo Siqueira, Larissa Zuanetti Silva, Letícia Caetano da Silva, Bruna Bonifácio, Vitorya Vanini Cardoso, Rene Coulaud Santos da Costa Cruz, Antônio Job Torres del Fiaco, Adriana Silva Lima, H.S. Costa, David Viana Martín, Rômulo Batista Rodrígues, Lays Figueira Castelo Branco, Gabriel Gade Martins Mesquita, A. Miranda, Maria Júlia Ribeiro de Oliveira, Gabriella Rennisy de Sousa Silva, Márcio Silva da Conceição, Giovanna Larsen Yamaki, Emanuele Souza Dias, Ana Paula Mariano Kerber, Heladia García, Daniela Celis SevergniniList of authors in order
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https://doi.org/10.36557/2674-8169.2025v7n3p192-203Publisher landing page
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| abstract_inverted_index.sendo | 179 |
| abstract_inverted_index.sobre | 118 |
| abstract_inverted_index.tanto | 189 |
| abstract_inverted_index.Assim, | 146 |
| abstract_inverted_index.CAPES, | 138 |
| abstract_inverted_index.EMBASE | 139 |
| abstract_inverted_index.Swyer, | 214 |
| abstract_inverted_index.afetam | 188 |
| abstract_inverted_index.atraso | 41 |
| abstract_inverted_index.coleta | 130 |
| abstract_inverted_index.dessas | 225 |
| abstract_inverted_index.devido | 91 |
| abstract_inverted_index.estudo | 77 |
| abstract_inverted_index.formas | 120 |
| abstract_inverted_index.homens | 73, 191 |
| abstract_inverted_index.manejo | 85 |
| abstract_inverted_index.ocorre | 15 |
| abstract_inverted_index.quadro | 170 |
| abstract_inverted_index.quanto | 192 |
| abstract_inverted_index.reside | 78 |
| abstract_inverted_index.série | 183 |
| abstract_inverted_index.tardia | 38, 98, 124, 176 |
| abstract_inverted_index.volume | 70 |
| abstract_inverted_index.LILACS, | 136 |
| abstract_inverted_index.PubMed, | 135 |
| abstract_inverted_index.Scielo, | 141 |
| abstract_inverted_index.Turner, | 210 |
| abstract_inverted_index.aumento | 30, 68 |
| abstract_inverted_index.gonadal | 11 |
| abstract_inverted_index.impacto | 93 |
| abstract_inverted_index.incluem | 202 |
| abstract_inverted_index.níveis | 33, 162 |
| abstract_inverted_index.reflete | 173 |
| abstract_inverted_index.sexuais | 22, 46 |
| abstract_inverted_index.sexual, | 51 |
| abstract_inverted_index.Gonadal. | 222 |
| abstract_inverted_index.Trata-se | 108 |
| abstract_inverted_index.clínico | 171 |
| abstract_inverted_index.elevados | 163 |
| abstract_inverted_index.gônadas | 158 |
| abstract_inverted_index.mamário | 63 |
| abstract_inverted_index.mulheres | 65 |
| abstract_inverted_index.revisão | 111 |
| abstract_inverted_index.últimos | 143 |
| abstract_inverted_index.Síndrome | 204, 208, 212 |
| abstract_inverted_index.adequados | 86 |
| abstract_inverted_index.afetados. | 107 |
| abstract_inverted_index.apresenta | 227 |
| abstract_inverted_index.clínicas | 121 |
| abstract_inverted_index.complexo, | 153 |
| abstract_inverted_index.crescente | 81 |
| abstract_inverted_index.falência | 10 |
| abstract_inverted_index.gônadas, | 24 |
| abstract_inverted_index.mulheres. | 194 |
| abstract_inverted_index.puberdade | 37, 97, 123, 175 |
| abstract_inverted_index.qualidade | 102 |
| abstract_inverted_index.Disgenesia | 221 |
| abstract_inverted_index.associadas | 198 |
| abstract_inverted_index.clínicas, | 229 |
| abstract_inverted_index.compensada | 160 |
| abstract_inverted_index.distúrbio | 6, 151 |
| abstract_inverted_index.endócrino | 152 |
| abstract_inverted_index.genéticas | 186 |
| abstract_inverted_index.hormônios | 21 |
| abstract_inverted_index.investigou | 117 |
| abstract_inverted_index.mecanismos | 238 |
| abstract_inverted_index.primária, | 12 |
| abstract_inverted_index.principais | 196 |
| abstract_inverted_index.produção | 19 |
| abstract_inverted_index.refletindo | 234 |
| abstract_inverted_index.síndromes | 185, 226 |
| abstract_inverted_index.testicular | 71 |
| abstract_inverted_index.Idiopático | 218 |
| abstract_inverted_index.Periódicos | 137 |
| abstract_inverted_index.avaliação | 55 |
| abstract_inverted_index.condições | 197 |
| abstract_inverted_index.diversidade | 236 |
| abstract_inverted_index.incompleta, | 178 |
| abstract_inverted_index.indivíduos | 106 |
| abstract_inverted_index.literatura, | 114 |
| abstract_inverted_index.manifestado | 154 |
| abstract_inverted_index.maturação | 50 |
| abstract_inverted_index.necessidade | 80 |
| abstract_inverted_index.parâmetros | 57 |
| abstract_inverted_index.plataformas | 134 |
| abstract_inverted_index.relevância | 75 |
| abstract_inverted_index.Klinefelter, | 206 |
| abstract_inverted_index.crescimento, | 60 |
| abstract_inverted_index.deficiência | 17 |
| abstract_inverted_index.diagnóstico | 83 |
| abstract_inverted_index.patológicos | 239 |
| abstract_inverted_index.relacionadas | 125 |
| abstract_inverted_index.responsável | 180 |
| abstract_inverted_index.secundárias | 47 |
| abstract_inverted_index.sistemática | 112 |
| abstract_inverted_index.subjacentes. | 240 |
| abstract_inverted_index.Hipogonadismo | 216 |
| abstract_inverted_index.caracterizado | 8 |
| abstract_inverted_index.diagnósticas | 230 |
| abstract_inverted_index.especialmente | 90 |
| abstract_inverted_index.específicas, | 233 |
| abstract_inverted_index.hipogonadismo | 1, 88, 127, 200 |
| abstract_inverted_index.significativo | 94 |
| abstract_inverted_index.terapêuticas | 232 |
| abstract_inverted_index.compensatório | 31 |
| abstract_inverted_index.gonadotrofinas | 165 |
| abstract_inverted_index.desenvolvimento | 43, 62 |
| abstract_inverted_index.gonadotrofinas. | 35 |
| abstract_inverted_index.características | 45, 228 |
| abstract_inverted_index.endocrinológico | 7 |
| abstract_inverted_index.Hipergonadotrófico | 217 |
| abstract_inverted_index.hipergonadotrófico | 2, 201 |
| abstract_inverted_index.hipergonadotrófico, | 89, 128 |
| cited_by_percentile_year | |
| countries_distinct_count | 0 |
| institutions_distinct_count | 23 |
| citation_normalized_percentile.value | 0.08986555 |
| citation_normalized_percentile.is_in_top_1_percent | False |
| citation_normalized_percentile.is_in_top_10_percent | True |