Smad Signaling and Promote Epithelial–Mesenchymal Transition Article Swipe

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Satoshi Sakai , Tatsuya Ohhata , Kyoko Kitagawa , Chiharu Uchida , Takuya Aoshima , Hiroyuki Niida , Tetsuro Suzuki , Yasumichi Inoue , Keiji Miyazawa , Masatoshi Kitagawa ·

YOU? · · 2023 · Open Access · · DOI: https://doi.org/10.1158/0008-5472.22421268.v1

FS1:Representative photographs of Fig. 1A; FS2:EMT-related gene expression; FS3:Snail induction by TGF-ÃŸ; FS4:Contribution of ELIT-1 on Snail expression; FS5:Effects on cell proliferation; FS6: EMT-related gene expression by ELIT-1; FS7:Smad3 binding to promoters; FS8:Effects of ELIT-1 overexpression (expression); FS9:Effect of ELIT-1 on heterodimer formation and translocation; FS10: Effects of ELIT-1 overexpression (ChIP)

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SMAD Chemistry Gene silencing Snail Gene Gene expression Cell biology Cancer research Biology Biochemistry Ecology

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Type: preprint
Language: en
Landing Page: https://doi.org/10.1158/0008-5472.22421268.v1
OA Status: gold
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DOI: https://doi.org/10.1158/0008-5472.22421268.v1

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Title: Supplementary Figure S1-S10 from Long Noncoding RNA <i>ELIT-1</i> Acts as a Smad3 Cofactor to Facilitate TGFβ/Smad Signaling and Promote Epithelial–Mesenchymal Transition

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Type: preprint

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Language: en

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Publication year: 2023

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Publication date: 2023-03-31

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Authors: Satoshi Sakai, Tatsuya Ohhata, Kyoko Kitagawa, Chiharu Uchida, Takuya Aoshima, Hiroyuki Niida, Tetsuro Suzuki, Yasumichi Inoue, Keiji Miyazawa, Masatoshi Kitagawa

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Landing page: https://doi.org/10.1158/0008-5472.22421268.v1

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OA URL: https://doi.org/10.1158/0008-5472.22421268.v1

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Concepts: SMAD, Chemistry, Gene silencing, Snail, Gene, Gene expression, Cell biology, Cancer research, Biology, Biochemistry, Ecology

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