Albert Roessner
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View article: Non-Coding RNAs in Diagnostic Pathology of High-Grade Central Osteosarcoma
Non-Coding RNAs in Diagnostic Pathology of High-Grade Central Osteosarcoma Open
A histological evaluation remains the cornerstone of diagnosing highly malignant osteosarcoma, having demonstrated its efficacy and reliability over several decades. However, despite these advancements, misdiagnoses with severe consequence…
View article: Non‐Coding RNAs in Diagnostic Pathology of High Grade Central Osteosarcoma
Non‐Coding RNAs in Diagnostic Pathology of High Grade Central Osteosarcoma Open
The histological evaluation remains the cornerstone of diagnosing highly malignant osteosarcoma, having demonstrated its efficacy and reliability over several decades. However, even in recent times misdiagnoses with severe consequences, in…
View article: Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Data from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Data from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
There are few reports describing the role of p53-dependent gene repression in apoptotic cell death. To identify such apoptosis-associated p53 target genes, we used the pro-oxidant plant-derived drug thymoquinone and compared p53+/+ and p53…
View article: Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Methods and Materials from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure 2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Table 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Data from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Data from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
There are few reports describing the role of p53-dependent gene repression in apoptotic cell death. To identify such apoptosis-associated p53 target genes, we used the pro-oxidant plant-derived drug thymoquinone and compared p53+/+ and p53…
View article: Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure Legends 1-2 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells
Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor <i>CHEK1</i> and Contributes to Apoptosis in Colorectal Cancer Cells Open
Supplementary Figure 1 from Thymoquinone Triggers Inactivation of the Stress Response Pathway Sensor CHEK1 and Contributes to Apoptosis in Colorectal Cancer Cells
View article: Combined Gastric and Colorectal Cancer Screening—A New Strategy
Combined Gastric and Colorectal Cancer Screening—A New Strategy Open
Background: Our aim was to evaluate the feasibility of a serological assessment of gastric cancer risk in patients undergoing colonoscopy in countries with low-to-moderate incidence rates. Methods: Serum samples were prospectively collecte…
View article: Inactivation of JNK2 as carcinogenic factor in colitis-associated and sporadic colorectal carcinogenesis
Inactivation of JNK2 as carcinogenic factor in colitis-associated and sporadic colorectal carcinogenesis Open
We recently reported that dysregulated c-Jun N-terminal kinases (JNK) activity causes defective cell cycle checkpoint control, inducing neoplastic transformation in a cellular ulcerative colitis (UC) model. In the quiescent chronic phase o…
View article: Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide‐Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding
Chk1 Promotes DNA Damage Response Bypass following Oxidative Stress in a Model of Hydrogen Peroxide‐Associated Ulcerative Colitis through JNK Inactivation and Chromatin Binding Open
Dysregulation of c‐Jun N ‐terminal kinase (JNK) activation promoted DNA damage response bypass and tumorigenesis in our model of hydrogen peroxide‐associated ulcerative colitis (UC) and in patients with quiescent UC (QUC), UC‐related dyspl…
View article: Additional file 1: of The prevalence of gastric heterotopia of the proximal esophagus is underestimated, but preneoplasia is rare - correlation with Barrettâ s esophagus
Additional file 1: of The prevalence of gastric heterotopia of the proximal esophagus is underestimated, but preneoplasia is rare - correlation with Barrettâ s esophagus Open
Gastric Heterotopia datasets. Contains demographic, endoscopic and histological data, as mentioned at the head of columns. (XLSX 53 kb)
View article: Ballooning osteolysis in 71 failed total ankle arthroplasties
Ballooning osteolysis in 71 failed total ankle arthroplasties Open
Background and purpose - Aseptic loosening is a major cause of failure in total ankle arthroplasty (TAA). In contrast to other total joint replacements, large periarticular cysts (ballooning osteolysis) have frequently been observed in thi…
View article: Histological evaluation of inflammation, hyperplasia, and glandular ectasia.
Histological evaluation of inflammation, hyperplasia, and glandular ectasia. Open
Blinded H&E-stained; gastric sections from n = 5–11 wt and ctsz−/− mice infected or non-infected with H. pylori SS1 for 24, 36, or 50 weeks were assessed. Sections were graded from 0–5 based on the criteria of Rogers et al. [23]. Compared …