Abedul Haque
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View article: Photovoltaic Performance Improvement of Dilute Nitrides GaAs<sub>1-<i>x</i></sub>N<sub><i>x </i></sub>-Based Thin-Film Solar Cell Structure Using SCAPS-1D Software
Photovoltaic Performance Improvement of Dilute Nitrides GaAs<sub>1-<i>x</i></sub>N<sub><i>x </i></sub>-Based Thin-Film Solar Cell Structure Using SCAPS-1D Software Open
The recent industrial revolution has increased the demand for the possible use of renewable energy sources to meet the World’s high energy requirements and to minimize the quantity of green-house gases (GHGs) in the atmosphere at once in a…
View article: Supplementary Table S1 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S1 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S1: Characteristics of Cell Lines Used
View article: Supplementary Table S3 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S3 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S3: IC50 values for BKM120 and Erlotinib at 72 h
View article: Supplementary Table S6 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S6 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S6: Pair-wise p values for animal studies
View article: Supplementary Table S2 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S2 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Table S2: Source and dilution factors of antibodies used
View article: Supplementary Table S4 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S4 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S4: Dose reduction indices for BKM120
View article: Supplementary Table S5 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S5 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supp lementary Table S5: Dose reduction indices for erlotinib
View article: Supplementary Figures S1-S10 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Figures S1-S10 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
This file contains supplementary information for publication
View article: Supplementary Table S4 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S4 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S4: Dose reduction indices for BKM120
View article: Supplementary Table S2 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S2 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Table S2: Source and dilution factors of antibodies used
View article: Supplementary Figures S1-S10 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Figures S1-S10 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
This file contains supplementary information for publication
View article: Supplementary Table S3 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S3 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S3: IC50 values for BKM120 and Erlotinib at 72 h
View article: Data from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Data from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
We previously reported that the EGFR-targeted inhibitor erlotinib induces G1 arrest of squamous cell carcinoma of the head and neck (SCCHN) cell lines without inducing significant apoptosis. Large-scale genomic studies suggest that >50% of…
View article: Supplementary Table S1 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S1 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S1: Characteristics of Cell Lines Used
View article: Supplementary Table S5 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S5 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supp lementary Table S5: Dose reduction indices for erlotinib
View article: Data from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Data from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
We previously reported that the EGFR-targeted inhibitor erlotinib induces G1 arrest of squamous cell carcinoma of the head and neck (SCCHN) cell lines without inducing significant apoptosis. Large-scale genomic studies suggest that >50% of…
View article: Supplementary Table S6 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance
Supplementary Table S6 from <i>In Vitro</i> and <i>In Vivo</i> Synergistic Antitumor Activity of the Combination of BKM120 and Erlotinib in Head and Neck Cancer: Mechanism of Apoptosis and Resistance Open
Supplementary Table S6: Pair-wise p values for animal studies
View article: Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Purpose:Molecular profiling has been used to select patients for targeted therapy and determine prognosis. Noninvasive strategies are critical to hepatocellular carcinoma (HCC) given the challenge of obtaining liver tissue biopsies.Experim…
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 3
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 3
View article: Supplementary Figure 1 from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Figure 1 from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplementary Figure 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 2
View article: Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Purpose:Molecular profiling has been used to select patients for targeted therapy and determine prognosis. Noninvasive strategies are critical to hepatocellular carcinoma (HCC) given the challenge of obtaining liver tissue biopsies.Experim…
View article: Supplementary Figure 1 from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Figure 1 from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplementary Figure 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 1
View article: Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA
Supplementary Data from Molecular Profiling of Hepatocellular Carcinoma Using Circulating Cell-Free DNA Open
Supplemental table 2