Alon Harmelin
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View article: Dual targeting of histone deacetylases and MYC as potential treatment strategy for H3-K27M pediatric gliomas
Dual targeting of histone deacetylases and MYC as potential treatment strategy for H3-K27M pediatric gliomas Open
Diffuse midline gliomas (DMGs) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in…
View article: Author response: Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas
Author response: Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas Open
Diffuse midline gliomas (DMG) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in …
View article: Reviewer #2 (Public Review): Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas
Reviewer #2 (Public Review): Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas Open
Diffuse midline gliomas (DMG) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in …
View article: Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas
Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas Open
Diffuse midline gliomas (DMG) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in …
View article: Establishing a Mouse Genotyping Core Facility Based on Automation, High-resolution Melting Analysis, and Purpose-developed MATLAB Applications for Sample Tracking, Data Analysis, and Report Generation
Establishing a Mouse Genotyping Core Facility Based on Automation, High-resolution Melting Analysis, and Purpose-developed MATLAB Applications for Sample Tracking, Data Analysis, and Report Generation Open
An in-house genotyping facility should aim to be more cost-effective than outsourced service and more reliable than genotyping performed by short-term employees or students of individual research groups. Reliable genotyping allows efficien…
View article: Dual targeting of histone deacetylases and MYC as potential treatment strategy for H3-K27M pediatric gliomas
Dual targeting of histone deacetylases and MYC as potential treatment strategy for H3-K27M pediatric gliomas Open
Diffuse midline gliomas (DMGs) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in…
View article: Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas
Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas Open
Diffuse midline gliomas (DMG) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in …
View article: Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas
Dual Targeting of Histone Deacetylases and MYC as Potential Treatment Strategy for H3-K27M Pediatric Gliomas Open
Diffuse midline gliomas (DMG) are aggressive and fatal pediatric tumors of the central nervous system that are highly resistant to treatments. Lysine to methionine substitution of residue 27 on histone H3 (H3-K27M) is a driver mutation in …
View article: Lung dendritic-cell metabolism underlies susceptibility to viral infection in diabetes
Lung dendritic-cell metabolism underlies susceptibility to viral infection in diabetes Open
People with diabetes feature a life-risking susceptibility to respiratory viral infection, including influenza and SARS-CoV-2 (ref. 1 ), whose mechanism remains unknown. In acquired and genetic mouse models of diabetes, induced with an acu…
View article: Supplementary Figure 1 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Figure 1 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Figure 1 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Movie 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Supplementary Movie 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Supplementary Movie 1 from In vivo Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
View article: Data from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Data from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Tumor-associated stroma, in general, and tumor fibroblasts and myofibroblasts, in particular, play a role in tumor progression. We previously reported that myofibroblast infiltration into implanted ovarian carcinoma spheroids marked the ex…
View article: Supplementary Text 3 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 3 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 3 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Movie 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Supplementary Movie 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Supplementary Movie 1 from In vivo Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
View article: Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Table 2 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Table 3 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Table 3 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Table 3 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor
View article: Data from Cell Lineage Analysis of a Mouse Tumor
Data from Cell Lineage Analysis of a Mouse Tumor Open
Revealing the lineage relations among cancer cells can shed light on tumor growth patterns and metastasis formation, yet cell lineages have been difficult to come by in the absence of a suitable method. We previously developed a method for…
View article: Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor
View article: Data from The <i>SIL</i> Gene Is Essential for Mitotic Entry and Survival of Cancer Cells
Data from The <i>SIL</i> Gene Is Essential for Mitotic Entry and Survival of Cancer Cells Open
Although mitosis is a general physiologic process, cancer cells are unusually sensitive to mitotic inhibitors. Therefore, there is an interest in the identification of novel mitotic inhibitors. Here, we report the novel discovery of the SI…
View article: Supplementary Figures 1 - 6, Table 1 from The <i>SIL</i> Gene Is Essential for Mitotic Entry and Survival of Cancer Cells
Supplementary Figures 1 - 6, Table 1 from The <i>SIL</i> Gene Is Essential for Mitotic Entry and Survival of Cancer Cells Open
Supplementary Figures 1 - 6, Table 1 from The SIL Gene Is Essential for Mitotic Entry and Survival of Cancer Cells
View article: Supplementary Figure 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Supplementary Figure 1 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Supplementary Figure 1 from In vivo Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
View article: Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 1 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Text 4 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 4 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 4 from Cell Lineage Analysis of a Mouse Tumor
View article: Supplementary Figure 2 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Supplementary Figure 2 from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Supplementary Figure 2 from In vivo Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
View article: Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 5 from Cell Lineage Analysis of a Mouse Tumor
View article: Data from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors
Data from <i>In vivo</i> Imaging of the Systemic Recruitment of Fibroblasts to the Angiogenic Rim of Ovarian Carcinoma Tumors Open
Tumor-associated stroma, in general, and tumor fibroblasts and myofibroblasts, in particular, play a role in tumor progression. We previously reported that myofibroblast infiltration into implanted ovarian carcinoma spheroids marked the ex…
View article: Supplementary Text 2 from Cell Lineage Analysis of a Mouse Tumor
Supplementary Text 2 from Cell Lineage Analysis of a Mouse Tumor Open
Supplementary Text 2 from Cell Lineage Analysis of a Mouse Tumor