Alyson Endicott
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View article: Data from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Data from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Tobacco smoke exposure has been associated with risk of childhood acute lymphoblastic leukemia (ALL). Understanding the relationship between tobacco exposures and specific mutations may yield etiologic insights. We carried out a case-only …
View article: Supplemental Figures from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Figures from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Supplemental Figures S1-S7
View article: Table S3 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S3 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, stratified by B-ALL and cytogenetic subtypes ETV6-RUNX1 fusion and high hyperdiploidy.
View article: Table S7 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S7 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses of joint effects of prenatal and postnatal tobacco smoke exposures on the number of deletions in the child's leukemia using multivariable Poisson regressions.
View article: Data from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Data from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Genome-wide association studies (GWAS) have identified SNPs in six genes that are associated with childhood acute lymphoblastic leukemia (ALL). A lead SNP was found to occur on chromosome 9p21.3, a region that is deleted in 30% of childhoo…
View article: Table S8 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S8 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses stratified by sex between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, the California Childhood Leukemia Study, 1995-2015.
View article: Data from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Data from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Tobacco smoke exposure has been associated with risk of childhood acute lymphoblastic leukemia (ALL). Understanding the relationship between tobacco exposures and specific mutations may yield etiologic insights. We carried out a case-only …
View article: Table S6 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S6 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, with maternal smoking adjusted for paternal smoking and vice-versa.
View article: Table S6 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S6 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, with maternal smoking adjusted for paternal smoking and vice-versa.
View article: Table S4 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S4 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Number of cases with 0, 1, or {greater than or equal to}2 deletions in relation to the binary tobacco exposure variables.
View article: Table S4 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S4 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Number of cases with 0, 1, or {greater than or equal to}2 deletions in relation to the binary tobacco exposure variables.
View article: Supplemental Table S2 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Table S2 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Distribution of deletions by gene and race/ethnicity in childhood ALL cases from the CCLS.
View article: Supplementary Table S2 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Supplementary Table S2 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Top SNP associations in the 9p21.3 region evaluated in Hispanic ALL patients from CCLS and validated in European-ancestry ALL patients from COG, with functional annotation.
View article: Data from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Data from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Genome-wide association studies (GWAS) have identified SNPs in six genes that are associated with childhood acute lymphoblastic leukemia (ALL). A lead SNP was found to occur on chromosome 9p21.3, a region that is deleted in 30% of childhoo…
View article: Supplemental Table S1 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Table S1 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Number of deletions in each gene tested amongst 559 ALL cases from the CCLS, assessed by MLPA.
View article: Table S3 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S3 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, stratified by B-ALL and cytogenetic subtypes ETV6-RUNX1 fusion and high hyperdiploidy.
View article: Supplementary Table S1 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Supplementary Table S1 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Subject characteristics of cases and controls undergoing genetic association analyses from the California Childhood Leukemia Study, The Children's Oncology Group, The Wellcome Trust Consortium, the GENEVA Melanoma Study, The University of …
View article: Supplementary Table S1 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Supplementary Table S1 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Subject characteristics of cases and controls undergoing genetic association analyses from the California Childhood Leukemia Study, The Children's Oncology Group, The Wellcome Trust Consortium, the GENEVA Melanoma Study, The University of …
View article: Supplemental Table S1 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Table S1 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Number of deletions in each gene tested amongst 559 ALL cases from the CCLS, assessed by MLPA.
View article: Table S5 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S5 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Correlation matrix for the parental reported tobacco smoke exposure variables. P-values and correlation coefficients were calculated using the Spearman's rank correlation coefficient test.
View article: Supplemental Table S2 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Table S2 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Distribution of deletions by gene and race/ethnicity in childhood ALL cases from the CCLS.
View article: Supplemental Material from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Material from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Revised Supplemental Material
View article: Supplemental Figures from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Figures from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Supplemental Figures S1-S7
View article: Table S8 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S8 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses stratified by sex between tobacco smoke exposures and the number of deletions in the child's leukemia using multivariable Poisson regressions, the California Childhood Leukemia Study, 1995-2015.
View article: Supplementary Table S2 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution
Supplementary Table S2 from A Heritable Missense Polymorphism in <i>CDKN2A</i> Confers Strong Risk of Childhood Acute Lymphoblastic Leukemia and Is Preferentially Selected during Clonal Evolution Open
Top SNP associations in the 9p21.3 region evaluated in Hispanic ALL patients from CCLS and validated in European-ancestry ALL patients from COG, with functional annotation.
View article: Table S5 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S5 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Correlation matrix for the parental reported tobacco smoke exposure variables. P-values and correlation coefficients were calculated using the Spearman's rank correlation coefficient test.
View article: Table S7 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Table S7 from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Association analyses of joint effects of prenatal and postnatal tobacco smoke exposures on the number of deletions in the child's leukemia using multivariable Poisson regressions.
View article: Supplemental Material from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia
Supplemental Material from Correlates of Prenatal and Early-Life Tobacco Smoke Exposure and Frequency of Common Gene Deletions in Childhood Acute Lymphoblastic Leukemia Open
Revised Supplemental Material
View article: Mapping of Segmental and Partial Segmental Infantile Hemangiomas of the Face and Scalp
Mapping of Segmental and Partial Segmental Infantile Hemangiomas of the Face and Scalp Open
This cohort study provides an updated map of facial segmental IHs with redefined maxillary (S2) and mandibular (S3) segment borders. It provides evidence that indeterminate hemangiomas are partial segmental hemangiomas respecting anatomic …
View article: Toxic epidermal necrolysis-like toxic erythema of chemotherapy: 2 illustrative cases
Toxic epidermal necrolysis-like toxic erythema of chemotherapy: 2 illustrative cases Open