Anders Valind
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View article: Early Toxicity in Childhood Acute Lymphoblastic Leukemia: A Comparison of NOPHO ALL2008 and ALLTogether Protocols in Sweden
Early Toxicity in Childhood Acute Lymphoblastic Leukemia: A Comparison of NOPHO ALL2008 and ALLTogether Protocols in Sweden Open
Background The European ALLTogether protocol for childhood acute lymphoblastic leukemia, initiated in Sweden 2019, introduced earlier asparaginase during induction, dexamethasone instead of prednisone for all patients, and omitted anthracy…
View article: Infectious Complications During Early Treatment of Childhood Acute Lymphoblastic Leukemia—A Comparison Between the ALLTogether and NOPHO ALL‐2008 Protocols
Infectious Complications During Early Treatment of Childhood Acute Lymphoblastic Leukemia—A Comparison Between the ALLTogether and NOPHO ALL‐2008 Protocols Open
Introduction Infection remains the most common treatment‐related toxicity of childhood ALL, emphasizing the need to identify patients at risk and to tailor treatment strategies accordingly. Aims The primary aim was to compare infectious to…
View article: Table S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 1. Overview of the study cohort and the clinical data. This table contains basic clinical information, including age, sex, whether tumors were uni- or bilateral, histology, stage, risk group, treatment to which the rese…
View article: Figure S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Figure S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 1 Geographic overview of histological and genetic features.
View article: Table S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 3. Table. Sample list with TP53 status. All 20 WT cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Figure S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Figure S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 1 Geographic overview of histological and genetic features.
View article: Table S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 3. Table. Sample list with TP53 status. All 20 WT cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Fig S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Fig S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 3. Relationship between anaplasia, copy number aberrations, regression and mitotic rate.
View article: Table S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 2. Representativeness of study participants. Details on how the patients with Wilms tumor in the present study compare to the general population of patients diagnosed with with Wilms tumor.
View article: Fig S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Fig S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 2. Complexity of phylogenetic tree, comparing WT DA to IR and BT WT.
View article: Fig S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Fig S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 2. Complexity of phylogenetic tree, comparing WT DA to IR and BT WT.
View article: Data from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Data from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Purpose: While patients with intermediate-risk Wilms tumors (WT) now have an overall survival rate of almost 90%, those affected by high-stage tumors with diffuse anaplasia (DA) have an overall survival of only around 50%. We here identify…
View article: Table S4 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S4 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 4. Segmental aberrations. All 20 Wilms tumor (WT) cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Table S4 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S4 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 4. Segmental aberrations. All 20 Wilms tumor (WT) cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Table S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S1 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 1. Overview of the study cohort and the clinical data. This table contains basic clinical information, including age, sex, whether tumors were uni- or bilateral, histology, stage, risk group, treatment to which the rese…
View article: Table S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Table S2 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Table 2. Representativeness of study participants. Details on how the patients with Wilms tumor in the present study compare to the general population of patients diagnosed with with Wilms tumor.
View article: Fig S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution
Fig S3 from Resolving the pathogenesis of anaplastic Wilms tumors through spatial mapping of cancer cell evolution Open
Supplementary Figure 3. Relationship between anaplasia, copy number aberrations, regression and mitotic rate.
View article: Haemostasis during early treatment of childhood acute lymphoblastic leukaemia with the <scp>ALLTogether</scp> protocol
Haemostasis during early treatment of childhood acute lymphoblastic leukaemia with the <span>ALLTogether</span> protocol Open
Funding Agencies|Ellen Bachrach Memorial Fund; Swedish Childhood Cancer Fund
View article: A Gradual Transition Toward Anaplasia in Wilms Tumor Through Tolerance to Genetic Damage
A Gradual Transition Toward Anaplasia in Wilms Tumor Through Tolerance to Genetic Damage Open
Patients with Wilms tumor (WT) in general have excellent survival, but the prognosis of patients belonging to the subgroup of WT with diffuse anaplasia (DA) is poor due to frequent resistance to chemotherapy. We hypothesized that DA WT cel…
View article: Table S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Table S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Table 2. Representativeness of study participants. Details on how the patients with Wilms tumor in the present study compare to the general population of patients diagnosed with with Wilms tumor.
View article: Figure S1 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Figure S1 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Figure 1 Geographic overview of histological and genetic features.
View article: Table S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Table S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Table 2. Representativeness of study participants. Details on how the patients with Wilms tumor in the present study compare to the general population of patients diagnosed with with Wilms tumor.
View article: Table S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Table S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Table 3. Table. Sample list with TP53 status. All 20 WT cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Fig S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Fig S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Figure 2. Complexity of phylogenetic tree, comparing WT DA to IR and BT WT.
View article: Data from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Data from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Purpose:While patients with intermediate-risk (IR) Wilms tumors now have an overall survival (OS) rate of almost 90%, those affected by high-stage tumors with diffuse anaplasia have an OS of only around 50%. We here identify key events in …
View article: Fig S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Fig S2 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Figure 2. Complexity of phylogenetic tree, comparing WT DA to IR and BT WT.
View article: Table S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Table S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Table 3. Table. Sample list with TP53 status. All 20 WT cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…
View article: Fig S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Fig S3 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Figure 3. Relationship between anaplasia, copy number aberrations, regression and mitotic rate.
View article: Table S4 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution
Table S4 from Resolving the Pathogenesis of Anaplastic Wilms Tumors through Spatial Mapping of Cancer Cell Evolution Open
Supplementary Table 4. Segmental aberrations. All 20 Wilms tumor (WT) cases and their corresponding samples are listed. ‘Sample position’ refers to the individual sample and its position in a certain paraffin block (see Figure 1 for detail…