Anna E. Prizment
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View article: Supplementary Table 1 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC)
Supplementary Table 1 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC) Open
Supplemental Table 1 shows the results from previous studies that found significant associations between the pre-selected SNPs and risk of cancers or autoimmune diseases.
View article: Supplementary Table 7 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC)
Supplementary Table 7 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC) Open
Supplemental Table 7 shows the association of sMICA and sMICB levels (continuous variables) and pre-selected SNPsa with risk of CRC stratified by family history of CRC.
View article: Supplementary Table 9 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC)
Supplementary Table 9 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC) Open
Supplemental Table 9 shows the associations between pre-selected SNPs and mean of Visit 2 and Visit 3 sMICA and sMICB levels (continuous variables).
View article: Supplementary Table 3 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC)
Supplementary Table 3 from Associations between MICA and MICB Genetic Variants, Protein Levels, and Colorectal Cancer: Atherosclerosis Risk in Communities (ARIC) Open
Supplemental Table 3 shows the associations between pre-selected SNPs and CRC risk stratified by sex, race, or diabetes status.
View article: Supplementary Tables S1 to S6 from Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA
Supplementary Tables S1 to S6 from Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA Open
Table S1: Genes and regions interrogated by the targeted mutation panel, assuming 75bp amplicon size. Table S2: Mutations detected with targeted driver gene panel in Early and Very Early plasma samples. Table S3: Clinical characteristics o…
View article: Data from Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA
Data from Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA Open
To explore how early cancers can be detected prior to clinical signs or symptoms, we assessed prospectively collected serial plasma samples from the Atherosclerosis Risk in Communities study, including 26 participants diagnosed with cancer…
View article: Proteomics-based aging clocks in midlife or late-life and their associated risk of dementia
Proteomics-based aging clocks in midlife or late-life and their associated risk of dementia Open
Accelerated biological age is associated with lower cognition and a higher risk of dementia in midlife and more prominently in late life.
View article: Associations of proteomic age with mortality and incident chronic diseases in the European Prospective Investigation into Cancer and Nutrition (EPIC)
Associations of proteomic age with mortality and incident chronic diseases in the European Prospective Investigation into Cancer and Nutrition (EPIC) Open
View article: Aging measures and cancer in the Health and Retirement Study (HRS)
Aging measures and cancer in the Health and Retirement Study (HRS) Open
View article: Associations Between Cancer and Atrial Fibrillation: The Atherosclerosis Risk in Communities Study
Associations Between Cancer and Atrial Fibrillation: The Atherosclerosis Risk in Communities Study Open
In adult cancer patients, AF risk is the highest within 3 months after diagnosis and remains significantly elevated throughout survivorship but could be due to detection bias. Cancer risk is strongest within 3 months of AF diagnosis but si…
View article: Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA
Detection of Cancers Three Years prior to Diagnosis Using Plasma Cell-Free DNA Open
To explore how early cancers can be detected prior to clinical signs or symptoms, we assessed prospectively collected serial plasma samples from the Atherosclerosis Risk in Communities study, including 26 participants diagnosed with cancer…
View article: Alcohol intake and pancreatic cancer risk: An analysis from 30 prospective studies across Asia, Australia, Europe, and North America
Alcohol intake and pancreatic cancer risk: An analysis from 30 prospective studies across Asia, Australia, Europe, and North America Open
Background Alcohol is a known carcinogen, yet the evidence for an association with pancreatic cancer risk is considered as limited or inconclusive by international expert panels. We examined the association between alcohol intake and pancr…
View article: Cross-sectional associations of proteomic age acceleration with self-reported physical and mental health and depression symptoms among those with and without cancer
Cross-sectional associations of proteomic age acceleration with self-reported physical and mental health and depression symptoms among those with and without cancer Open
Purpose Accelerated aging may affect quality of life (QOL), and having cancer may further complicate that relationship. We assessed the cross-sectional association of a novel proteomic aging clock (PAC) with self-reported physical and ment…
View article: 59 Does cytomegalovirus (CMV) infection contribute to social-related health disparities among cancer survivors?
59 Does cytomegalovirus (CMV) infection contribute to social-related health disparities among cancer survivors? Open
Objectives/Goals: We aim to explore the associations of race/ethnicity and socioeconomic status (SES): 1) with grip strength, walking speed, and comorbidity index cross-sectionally and 2) with the change in comorbidity index and mortality …
View article: Supplementary Table S5 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S5 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S5: The association between current ethanol intake and breast cancer incidence stratified by routine examinations frequency and by race in ARIC (1990-2015)
View article: Supplementary Figure S3 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S3 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S3: Natural logarithm hazard ratios of breast cancer for P-spline term scaled 313-SNP PRS with 4 degrees of freedom estimated by fully-adjusted Cox proportional hazards model in ARIC (1990-2015)
View article: Supplementary Figure S5 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S5 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S5: Natural logarithm hazard ratios of breast cancer for P-spline term scaled 313-SNP PRS with 4 degrees of freedom estimated by fully-adjusted Cox proportional hazards model stratified by alcohol consumption category …
View article: Supplementary Figure S4 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S4 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S4: Natural logarithm hazard ratios of breast cancer for P-spline term current ethanol intake with 4 degrees of freedom estimated by fully-adjusted Cox proportional hazards model in ARIC (1990-2015)
View article: Supplementary Table S2 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S2 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S2: The association between 313-SNP PRS and breast cancer incidence stratified by maternal history of breast cancer and by race in ARIC (1990-2015)
View article: Supplementary Table S1 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S1 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S1: Baseline characteristics of women in ARIC at Visit 2 (1990-1992)
View article: Data from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Data from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
High genetic risk and alcohol consumption ≥1 drink/day are associated with increased breast cancer risk. However, the interaction between alcohol and genetics on breast cancer risk is poorly understood, including in populations not enriche…
View article: Supplementary Figure S6 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S6 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S6: Natural logarithm hazard ratios of breast cancer for P-spline term scaled 313-SNP PRS with 4 degrees of freedom estimated by fully-adjusted Cox proportional hazards model stratified by alcohol consumption category …
View article: Supplementary Table S4 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S4 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S4: The association between current ethanol intake and breast cancer incidence restricted to those had a health insurance, stratified by race in ARIC (1990-2015)
View article: Supplementary Table S6 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S6 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S6: The association between current ethanol intake and breast cancer incidence stratified by whether the participants ever had a mammogram and by race in ARIC (1990-2015)
View article: Supplementary Table S3 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Table S3 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Table S3: The association between current ethanol intake and breast cancer incidence excluding extreme ethanol intake values, stratified by race in ARIC (1990-2015)
View article: Supplementary Figure S1 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S1 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S1: Time-dependent coefficients of scaled 313-SNP PRS for breast cancer incidence estimated by fully adjusted Cox proportional hazards model in ARIC (1990-2015)
View article: Supplementary Figure S2 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Supplementary Figure S2 from Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
Supplementary Figure S2: Time-dependent coefficients of current ethanol intake for breast cancer incidence estimated by fully adjusted Cox proportional hazards model in ARIC (1990-2015)
View article: Proteomics-based aging clocks in midlife and late-life and risk of dementia
Proteomics-based aging clocks in midlife and late-life and risk of dementia Open
View article: The Association between Proteomic Aging Clocks and the Risk of Cancer in Midlife Individuals
The Association between Proteomic Aging Clocks and the Risk of Cancer in Midlife Individuals Open
Background To measure the aging process before a cancer diagnosis, we developed the first cancer-specific proteomic aging clock (CaPAC) and examined its association with cancer risk in the Atherosclerosis Risk in Communities (ARIC) and Mul…
View article: Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study
Alcohol Consumption Does not Modify the Polygenic Risk Score–Based Genetic Risk of Breast Cancer in Postmenopausal Women: Atherosclerosis Risk in Communities Study Open
High genetic risk and alcohol consumption ≥1 drink/day are associated with increased breast cancer risk. However, the interaction between alcohol and genetics on breast cancer risk is poorly understood, including in populations not enriche…