Benjamin J. Staudinger
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View article: Cystic Fibrosis Lung Function Decline after Within-Host Evolution Increases Virulence of Infecting <i>Pseudomonas aeruginosa</i>
Cystic Fibrosis Lung Function Decline after Within-Host Evolution Increases Virulence of Infecting <i>Pseudomonas aeruginosa</i> Open
"Cystic Fibrosis Lung Function Decline after Within-Host Evolution Increases Virulence of Infecting Pseudomonas aeruginosa." American Journal of Respiratory and Critical Care Medicine, 203(5), pp. 637–640
View article: In Vivo Proteome of <i>Pseudomonas aeruginosa</i> in Airways of Cystic Fibrosis Patients
In Vivo Proteome of <i>Pseudomonas aeruginosa</i> in Airways of Cystic Fibrosis Patients Open
Chronic airway infection with P. aeruginosa (PA) is a hallmark of cystic fibrosis (CF) disease. The mechanisms producing PA persistence in CF therapies remain poorly understood. To gain insight on PA physiology in patient airways and bette…
View article: Regional Isolation Drives Bacterial Diversification within Cystic Fibrosis Lungs
Regional Isolation Drives Bacterial Diversification within Cystic Fibrosis Lungs Open
View article: Dynamic Proteome Response of Pseudomonas aeruginosa to Tobramycin Antibiotic Treatment
Dynamic Proteome Response of Pseudomonas aeruginosa to Tobramycin Antibiotic Treatment Open
Genetically susceptible bacteria become antibiotic tolerant during chronic infections, and the mechanisms responsible are poorly understood. One factor that may contribute to differential sensitivity in vitro and in vivo is differences in …
View article: General and condition-specific essential functions of <i>Pseudomonas aeruginosa</i>
General and condition-specific essential functions of <i>Pseudomonas aeruginosa</i> Open
Significance There is an urgent need for new antibiotics active against resistant bacterial pathogens like Pseudomonas aeruginosa . Target-directed drug development provides a potential path to such drugs, and essential gene products repre…