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Corrigendum to “Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation” [J. Mol. Cell. Cardiol. 117 (2018) 88–99] Open

Amanda Soler, Ian Hunter, Gregory Joseph, Rebecca Hutcheson, Brenda Hutcheson , et al. · 2018

Elevated 20-HETE in metabolic syndrome regulates arterial stiffness and systolic hypertension via MMP12 activation Open

Amanda Soler, Ian Hunter, Gregory Joseph, Rebecca Hutcheson, Brenda Hutcheson , et al. · 2018

Cardiovascular function in male and female JCR:LA-cp rats: effect of high-fat/high-sucrose diet Open

Ian Hunter, Amanda Soler, Gregory Joseph, Brenda Hutcheson, Chastity Bradford , et al. · 2017

Thirty percent of the world population is diagnosed with metabolic syndrome. High-fat/high-sucrose (HF/HS) diet (Western diet) correlates with metabolic syndrome prevalence. We characterized effects of the HF/HS diet on vascular (arterial …

Elevated 20-HETE impairs coronary collateral growth in metabolic syndrome via endothelial dysfunction Open

Gregory Joseph, Amanda Soler, Rebecca Hutcheson, Ian Hunter, Chastity Bradford , et al. · 2016

Coronary collateral growth (CCG) is impaired in metabolic syndrome (MetS). microRNA-145 (miR-145-Adv) delivery to our rat model of MetS (JCR) completely restored and neutrophil depletion significantly improved CCG. We determined whether lo…

miR-21-mediated decreased neutrophil apoptosis is a determinant of impaired coronary collateral growth in metabolic syndrome Open

Rebecca Hutcheson, Russell Terry, Brenda Hutcheson, Rashmi Jadhav, Jennifer A. Chaplin , et al. · 2015

Coronary collateral growth (CCG) is impaired in metabolic syndrome. microRNA-21 (miR-21) is a proproliferative and antiapoptotic miR, which we showed to be elevated in metabolic syndrome. Here we investigate whether impaired CCG in metabol…

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