Andrey Brydun
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View article: Supplementary Figure S2 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S2 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Gene ontology, KEGG pathway and Gene set enrichment analysis of BACH1 silenced AsPC-1. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S4 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S4 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Immunofluorescence staining of BACH1 and E-cadherin in AsPC-1/sgBACH1 cells and the expression amounts of BACH1 in pancreatic cancer cell line models used. Details are described in methods and legends file (Supplementary_methods_and_legend…
View article: Supplementary Figure S3 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S3 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
The effects of BACH1 silencing on cellular migration in wild type KRAS cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Pancreatic ductal adenocarcinoma (PDAC) is among the cancers with the poorest prognoses due to its highly malignant features. BTB and CNC homology 1 (BACH1) has been implicated in RAS-driven tumor formation. We focused on the role of BACH1…
View article: Supplementary Table S1-6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Table S1-6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Table S1. Primer information Table S2. The list of genes which were changed expression of mRNA by BACH1 silencing Table S3. The list of genes which were changed in their mRNA expression by BACH1 deletion Table S4. Clinicopathological featu…
View article: Supplementary Figure S1 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S1 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
The expression amounts of BACH1 in pancreatic cancer cell lines and the effect of BACH1 silencing on cellular proliferation in wild type KRAS cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Pancreatic ductal adenocarcinoma (PDAC) is among the cancers with the poorest prognoses due to its highly malignant features. BTB and CNC homology 1 (BACH1) has been implicated in RAS-driven tumor formation. We focused on the role of BACH1…
View article: Supplementary Figure S4 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S4 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Immunofluorescence staining of BACH1 and E-cadherin in AsPC-1/sgBACH1 cells and the expression amounts of BACH1 in pancreatic cancer cell line models used. Details are described in methods and legends file (Supplementary_methods_and_legend…
View article: Supplementary Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Methods and legends for supplementary figures and tables
View article: Supplementary Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Data from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Methods and legends for supplementary figures and tables
View article: Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Induction of epithelial gene expression in AsPC1 cells lacking BACH1. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S5 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Induction of epithelial gene expression in AsPC1 cells lacking BACH1. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S7 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S7 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
BACH1 high expression is correlated with poor prognosis. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Examination of metastatic activity of BACH1 knockout AsPC1 cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S2 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S2 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Gene ontology, KEGG pathway and Gene set enrichment analysis of BACH1 silenced AsPC-1. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S1 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S1 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
The expression amounts of BACH1 in pancreatic cancer cell lines and the effect of BACH1 silencing on cellular proliferation in wild type KRAS cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S6 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
Examination of metastatic activity of BACH1 knockout AsPC1 cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: Supplementary Figure S3 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition
Supplementary Figure S3 from BACH1 Promotes Pancreatic Cancer Metastasis by Repressing Epithelial Genes and Enhancing Epithelial–Mesenchymal Transition Open
The effects of BACH1 silencing on cellular migration in wild type KRAS cells. Details are described in methods and legends file (Supplementary_methods_and_legends).
View article: MiR-196a regulates heme oxygenase-1 by silencing Bach1 in the neonatal mouse lung
MiR-196a regulates heme oxygenase-1 by silencing Bach1 in the neonatal mouse lung Open
In the lung, heme oxygenase-1 (HO-1) is developmentally regulated, with its highest expression in the first days of life. In addition, neonatal mice have limited HO-1 induction in hyperoxia compared with adults. However, few reports have a…
View article: Genomewide approaches for <scp>BACH</scp>1 target genes in mouse embryonic fibroblasts showed <scp>BACH</scp>1‐<i>Pparg</i> pathway in adipogenesis
Genomewide approaches for <span>BACH</span>1 target genes in mouse embryonic fibroblasts showed <span>BACH</span>1‐<i>Pparg</i> pathway in adipogenesis Open
The transcription repressor BTB and CNC homology 1 ( BACH 1) represses genes involved in heme metabolism and oxidative stress response. BACH 1 also suppresses the p53‐dependent cellar senescence in primary mouse embryonic fibroblasts ( MEF…
View article: Limb-girdle muscular dystrophy type 2A (calpainopathy)
Limb-girdle muscular dystrophy type 2A (calpainopathy) Open
The clinical presentation of progressive limb-girdle muscular dystrophy type 2A (LGMD2A) is due to loss of functionally active cal-pain-3, a skeletal muscle specific isoform of one of the calpain family proteins involved in the regulation …
View article: Glycogen storage disease type II (Pompe disease) in children
Glycogen storage disease type II (Pompe disease) in children Open
The paper gives the data available in the literature, which reflect the manifestations, diagnosis, and current treatments of the rare (orphan) inherited disease glycogen storage disease type II or Pomp disease in children, as well as its c…