Charlie Zhong
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View article: Physical health outcomes and step cadence improvements: a digital physical activity intervention in cancer survivors
Physical health outcomes and step cadence improvements: a digital physical activity intervention in cancer survivors Open
Background Cancer survivors experience symptoms that can often be managed with moderate-to-vigorous physical activity (MVPA). Most studies are short, small, and in-person, limiting scalability. This study evaluated a 12-month digital MVPA …
View article: 0334 Chronotype, Sleep Timing, Sleep Regularity, and Cancer Risk: A Systematic Review
0334 Chronotype, Sleep Timing, Sleep Regularity, and Cancer Risk: A Systematic Review Open
Introduction Sleep is a multidimensional modifiable lifestyle factor related to cancer risk. Prior research has primarily focused on sleep duration, despite the increasing importance of sleep timing and sleep regularity in the health resea…
View article: Overnight Fasting and Body Weight: Emulated Target Trial Using Cancer Prevention Study-3 Data
Overnight Fasting and Body Weight: Emulated Target Trial Using Cancer Prevention Study-3 Data Open
Background/Objectives: Intermittent fasting has gained attention in managing weight, yet its long-term effects remain unclear. We examined the impact of overnight, before-sleep, and after-sleep fasting on body weight over two years using d…
View article: Perinatal Exposures to Ambient Fine Particulate Matter and Outdoor Artificial Light at Night and Risk of Pediatric Papillary Thyroid Cancer
Perinatal Exposures to Ambient Fine Particulate Matter and Outdoor Artificial Light at Night and Risk of Pediatric Papillary Thyroid Cancer Open
This study provides new evidence suggesting associations between early-life exposure to and O-ALAN and pediatric papillary thyroid cancer. Given that O-ALAN may also represent other air pollutants or broader urbanization patterns, further…
View article: Electronic Screen Use and Sleep Duration and Timing in Adults
Electronic Screen Use and Sleep Duration and Timing in Adults Open
Importance Electronic screen use before bed may disrupt circadian rhythms, leading to poorer sleep. Objective To investigate the association between screen use before bed and sleep outcomes among adults. Design, Setting, and Participants T…
View article: Chronotype, sleep timing, sleep regularity, and cancer risk: A systematic review
Chronotype, sleep timing, sleep regularity, and cancer risk: A systematic review Open
Sleep is a multidimensional modifiable lifestyle factor related to cancer risk. Prior research has primarily focused on sleep duration, despite the increasing importance of sleep timing and sleep regularity in the health research field. Th…
View article: Supplementary Table S7 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S7 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S7 shows meta-analyzed minP permutation p values from the Latino and non-Latino datasets used for creating tables S5 and S6.
View article: Supplementary Table S5 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S5 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S5 shows minP adjusted P values for tobacco x gene interaction in Latinos only.
View article: Supplementary Table S6 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S6 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S6 shows minP adjusted P values for tobacco x gene interaction in non-Latino whites only.
View article: Supplementary Table S3 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S3 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S3 shows a full list of G x E interaction p-values in the two main study sets (Latinos only), and meta-analyzed together.
View article: Supplementary Figure S1 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S1 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure 1 shows bulk tissue expression for RECQL. This gene is expressed highly in pre-B lymphoblasts which is the target for transformation in childhood ALL.
View article: Supplementary Figure S2 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S2 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S2 shows bulk tissue expression for TDG. This gene is expressed highly in pre-B lymphoblasts (more than any other tissue) which is the target for transformation in childhood ALL.
View article: Supplementary Table S1 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S1 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S1 lists all DNA repair genes examined in this study.
View article: Supplementary Figure S3 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S3 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S3 shows bulk tissue expression for MLH3. This gene is expressed highly in pre-B lymphoblasts which is the target for transformation in childhood ALL.
View article: Supplementary Table S2 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S2 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S2 shows "transcripts of each gene per million transcripts" showing the relative high expression of these genes in pre-B lymphoblasts in GTEX data.
View article: Data from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Data from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Background:Acute lymphoblastic leukemia (ALL) is the most common type of cancer among children. Tobacco exposure during gestation has been investigated as a potential risk factor, but its role remains undefined. Given tobacco’s toxicologic…
View article: Supplementary Figure S8 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S8 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S8 shows a "coefficient effect size" comparison between Non-Latino white and Latino children with ALL compared to controls. The effect sizes generally are not associated as a group.
View article: Supplementary Table S4 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Table S4 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Table S4 shows a full list of G x E interaction p-values in the two main study sets (non-Latino whites only), and meta-analyzed together.
View article: Supplementary Figure S4 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S4 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S4 shows bulk tissue expression for SETMAR. This gene is expressed substantially in pre-B lymphoblasts which is the target for transformation in childhood ALL.
View article: Supplementary Figure S7 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S7 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S7 shows a "interaction effect size of G x E" comparison between Non-Latino white and Latino children with ALL compared to controls. The effect sizes generally are not associated as a group.
View article: Supplementary Figure S5 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S5 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S5 shows bulk tissue expression for PDS5B. This gene is expressed highly in pre-B lymphoblasts which is the target for transformation in childhood ALL.
View article: Supplementary Figure S6 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study
Supplementary Figure S6 from The Influence of DNA Repair Genes and Prenatal Tobacco Exposure on Risk of Childhood Acute Lymphoblastic Leukemia: A Gene–Environment Interaction Study Open
Supplementary Figure S6 shows bulk tissue expression for BARD1. This gene is expressed highly in pre-B lymphoblasts (more than any other tissue) which is the target for transformation in childhood ALL.
View article: Nomogram based on the log odds of negative lymph node/T stage can predict the prognosis of patients with colorectal cancer: a retrospective study based on SEER database and external validation in China
Nomogram based on the log odds of negative lymph node/T stage can predict the prognosis of patients with colorectal cancer: a retrospective study based on SEER database and external validation in China Open
Objectives This study investigated the prognostic role of log odds of negative lymph node/T stage (LONT) and established a nomogram based on LONT to predict the prognosis in colorectal cancer (CRC) patients. Design A retrospective cohort s…
View article: Validity of self-reported sleep duration in the Cancer Prevention Study– 3
Validity of self-reported sleep duration in the Cancer Prevention Study– 3 Open
Purpose We examined the one-year test re-test reliability and validity criterion of survey-assessed sleep duration collected from two separate questions. Methods The Activity Validation Sub Study included 751 participants of the Cancer Pre…
View article: Exposures to Artificial Light at Night and Ambient Fine Particulate Matter and Risk of Pediatric Papillary Thyroid Cancer
Exposures to Artificial Light at Night and Ambient Fine Particulate Matter and Risk of Pediatric Papillary Thyroid Cancer Open
View article: Environmental exposures and residential mobility among Hispanic and non-Hispanic White families in California
Environmental exposures and residential mobility among Hispanic and non-Hispanic White families in California Open
View article: Supplemental Figure 6 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia
Supplemental Figure 6 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia Open
QQ-plot for CCLS study, non-Hispanic White participants. As Supp. Fig. 4, but for the CCLS non-Hispanic White participant strata.
View article: Supplemental Figure 6 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia
Supplemental Figure 6 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia Open
QQ-plot for CCLS study, non-Hispanic White participants. As Supp. Fig. 4, but for the CCLS non-Hispanic White participant strata.
View article: Supplemental Figure 1 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia
Supplemental Figure 1 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia Open
Manhattan plot of disease-gene associations and ALL risk. The figure displays P-values across the genome for gene-ALL genotype additive model results.
View article: Supplemental Figure 4 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia
Supplemental Figure 4 from Gene–Environment Analyses Reveal Novel Genetic Candidates with Prenatal Tobacco Exposure in Relation to Risk for Childhood Acute Lymphoblastic Leukemia Open
QQ-plot for CCRLP study, non-Hispanic White participants. Expected P-value distribution is placed on the X-axis, and observed P-values on the Y axis. The plots are displayed as follows: a. 3-degree of freedom test incorporating gene-, envi…