Catherine Lavau
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View article: Extracellular vesicles as a potential source of biomarkers for endocrine disruptors in MASLD: A short review on the case of DEHP
Extracellular vesicles as a potential source of biomarkers for endocrine disruptors in MASLD: A short review on the case of DEHP Open
Metabolic dysfunction-Associated Steatotic Liver Disease (MASLD) is a chronic disease with increasing prevalence and for which non-invasive biomarkers are needed. Environmental endocrine disruptors (EDs) are known to be involved in the ons…
View article: UDP-6-glucose dehydrogenase in hormonally responsive breast cancers
UDP-6-glucose dehydrogenase in hormonally responsive breast cancers Open
Survival for metastatic breast cancer is low and thus, continued efforts to treat and prevent metastatic progression are critical. Estrogen is shown to promote aggressive phenotypes in multiple cancer models irrespective of estrogen recept…
View article: Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine MLL Rearrangement Leukemias
View article: Data from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Data from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Important biological and pathologic properties are often conserved across species. Although several mouse leukemia models have been well established, the genes deregulated in both human and murine leukemia cells have not been studied syste…
View article: Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Supplementary Tables 1-4 from Consistent Deregulation of Gene Expression between Human and Murine MLL Rearrangement Leukemias
View article: Data from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Data from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Important biological and pathologic properties are often conserved across species. Although several mouse leukemia models have been well established, the genes deregulated in both human and murine leukemia cells have not been studied syste…
View article: Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine MLL Rearrangement Leukemias
View article: Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias
Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine <i>MLL</i> Rearrangement Leukemias Open
Supplementary Tables 5-8 from Consistent Deregulation of Gene Expression between Human and Murine MLL Rearrangement Leukemias
View article: Fusion of the CRM1 nuclear export receptor to AF10 causes leukemia and transcriptional activation of HOXA genes
Fusion of the CRM1 nuclear export receptor to AF10 causes leukemia and transcriptional activation of HOXA genes Open
View article: Original uncropped and unaltered images underlying the blots shown in Figs 1D, 2A and 2F.
Original uncropped and unaltered images underlying the blots shown in Figs 1D, 2A and 2F. Open
View article: The SQSTM1-NUP214 fusion protein interacts with Crm1, activates Hoxa and Meis1 genes, and drives leukemogenesis in mice
The SQSTM1-NUP214 fusion protein interacts with Crm1, activates Hoxa and Meis1 genes, and drives leukemogenesis in mice Open
The NUP98 and NUP214 nucleoporins (NUPs) are recurrently fused to heterologous proteins in leukemia. The resulting chimeric oncoproteins retain the phenylalanine-glycine (FG) repeat motifs of the NUP moiety that mediate interaction with th…
View article: The SQSTM1-NUP214 Fusion Protein Cooperates with Crm1 to Activate Hoxa Genes and Drives Leukemogenesis in Mice
The SQSTM1-NUP214 Fusion Protein Cooperates with Crm1 to Activate Hoxa Genes and Drives Leukemogenesis in Mice Open
Background: The NUP98 and NUP214 nucleoporins (NUPs) are recurrently fused to heterologous proteins in leukemia. The resulting chimeric oncoproteins retain the NUP phenylalanine-glycine (FG) repeat motifs that mediate interaction with the …
View article: The human HOXA9 protein uses paralog-specific residues of the homeodomain to interact with TALE-class cofactors
The human HOXA9 protein uses paralog-specific residues of the homeodomain to interact with TALE-class cofactors Open
HOX proteins interact with PBX and MEIS cofactors, which belong to the TALE-class of homeodomain (HD)-containing transcription factors. Although the formation of HOX-PBX complexes depends on a unique conserved HOX motif called hexapeptide …
View article: Modulation of PICALM Levels Perturbs Cellular Cholesterol Homeostasis
Modulation of PICALM Levels Perturbs Cellular Cholesterol Homeostasis Open
PICALM (Phosphatidyl Inositol Clathrin Assembly Lymphoid Myeloid protein) is a ubiquitously expressed protein that plays a role in clathrin-mediated endocytosis. PICALM also affects the internalization and trafficking of SNAREs and modulat…
View article: Correction: Corrigendum: PICALM modulates autophagy activity and tau accumulation
Correction: Corrigendum: PICALM modulates autophagy activity and tau accumulation Open