Changzheng Liu
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View article: Nonlinear characteristic analysis and control of a DC motor
Nonlinear characteristic analysis and control of a DC motor Open
This paper focuses on the brushless DC motor system. Complexity analysis is used to study the basic chaotic dynamic behavior of the system. Second, based on the active-finite-time control theory, a feedback controller for the system is pro…
View article: Supplementary tables. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary tables. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
6 tables referenced in this manuscript is shown as supplementary files. Table S1. Characterization of 20 aired of PDAC patients. Table S2. Sequences of miRNAs and U6 snRNA primers. Table S3. Primer sequences of protein-coding genes. Table …
View article: Data from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Data from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
Mutant p53 (mutp53) proteins promote tumor invasion and metastasis in pancreatic ductal adenocarcinoma (PDAC). However, the mechanism underlying sustained activation of mutp53 oncogenic signaling is currently unclear. In this study, we rep…
View article: Supplementary Figures. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary Figures. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
10 supplementary figures are referenced in this manuscript. Fig.S1. The expression of NOP14 is positively correlated with the capacity of PDAC cells. Fig. S2. The expression of NOP14 is determined in PANC-1 cells. Fig. S3. NOP14 promotes M…
View article: Supplementary tables. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary tables. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
6 tables referenced in this manuscript is shown as supplementary files. Table S1. Characterization of 20 aired of PDAC patients. Table S2. Sequences of miRNAs and U6 snRNA primers. Table S3. Primer sequences of protein-coding genes. Table …
View article: Supplementary Figures. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary Figures. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
10 supplementary figures are referenced in this manuscript. Fig.S1. The expression of NOP14 is positively correlated with the capacity of PDAC cells. Fig. S2. The expression of NOP14 is determined in PANC-1 cells. Fig. S3. NOP14 promotes M…
View article: Supplementary Materials and Methods. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary Materials and Methods. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
Detailed information of the materials and methods referenced in this manuscript is shown as supplementary files.
View article: Supplementary Materials and Methods. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Supplementary Materials and Methods. from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
Detailed information of the materials and methods referenced in this manuscript is shown as supplementary files.
View article: Data from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14
Data from Pancreatic Cancer Progression Relies upon Mutant p53-Induced Oncogenic Signaling Mediated by NOP14 Open
Mutant p53 (mutp53) proteins promote tumor invasion and metastasis in pancreatic ductal adenocarcinoma (PDAC). However, the mechanism underlying sustained activation of mutp53 oncogenic signaling is currently unclear. In this study, we rep…
View article: Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Figure S3 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S3 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S3. Enforced expression of miR-29s in MGC803 and HGC27 cells.
View article: Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Figure Legends 1-3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure Legends 1-3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure Legends 1-3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure 2 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Tables S1-S4 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Tables S1-S4 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Tables S1-S4. Comparison of miR-29s expression levels with clinic-pathological features in patients with primary gastric cancer (S1); miR-29s associated with disease-free survival of patients with GC in the training set (S2);…
View article: Supplementary Figure S8 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S8 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S8. Correlation between mIR-29s expression and catenin-ÃŽ' in 66 cases of GCs.
View article: Supplementary Figure S1 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S1 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S1. miR-29s expression analyses in GC cells.
View article: Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure 3 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Figure 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Figure 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Figure 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Table 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Supplementary Table 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Supplementary Table 1 from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
View article: Supplementary Figure S1 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S1 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S1. miR-29s expression analyses in GC cells.
View article: Supplementary Figure S10 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S10 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S10. mIR-29c and catenin-ÃŽ' expression is determined in GC cells treated with imatinib.
View article: Supplementary Figure S9 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S9 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S9. E-cadherin expression is determined in GC cells with drugs treatment.
View article: Supplementary Figure S9 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S9 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S9. E-cadherin expression is determined in GC cells with drugs treatment.
View article: Data from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Data from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Chemotherapy has improved the survival of patients with gastric cancer by unknown mechanisms. In this study, we showed that cisplatin and docetaxel used in gastric cancer treatment increase the expression of miRNA-29 (miR-29) family member…
View article: Supplementary Figure S8 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S8 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S8. Correlation between mIR-29s expression and catenin-ÃŽ' in 66 cases of GCs.
View article: Data from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer
Data from miRNA-96 Suppresses KRAS and Functions as a Tumor Suppressor Gene in Pancreatic Cancer Open
Therapeutic applications of microRNA (miRNA) in KRAS-driven pancreatic cancers might be valuable, but few studies have explored this area. Here, we report that miR-96 directly targets the KRAS oncogene and functions as a tumor-suppr…
View article: Data from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Data from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Chemotherapy has improved the survival of patients with gastric cancer by unknown mechanisms. In this study, we showed that cisplatin and docetaxel used in gastric cancer treatment increase the expression of miRNA-29 (miR-29) family member…
View article: Supplementary Figure S5 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S5 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S5. The suppressive effect of miR-29c on GC formation.
View article: Supplementary Figure S6 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer
Supplementary Figure S6 from Chemotherapy-Induced miRNA-29c/Catenin-δ Signaling Suppresses Metastasis in Gastric Cancer Open
Supplementary Figure S6. miR-29s and catenin-ÃŽ' expression depicts an inverse correlation in 20 cases of GC tissues.