Christiane Chéreau
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View article: Supplementary Figure 4 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 4 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Immunoblots showing the effect of DMF on NRF2 along with its associated proteins in various cancer cells.
View article: Supplementary Figure 3 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 3 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Concentration-dependant cellular effects of DMF in OVCAR3 cells.
View article: Data from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i>
Data from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i> Open
Sorafenib is presently the only effective therapy in advanced hepatocellular carcinoma (HCC). Because most anticancer drugs act, at least in part, through the generation of reactive oxygen species, we investigated whether sorafenib can ind…
View article: Supplementary Table S2. Comparison of morphometric parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Supplementary Table S2. Comparison of morphometric parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Mean values {plus minus} SD of morphometric parameters of single myelinated axons isolated from sciatic nerves of mice injected with vehicle, oxaliplatin, oxaliplatin plus niclosamide or niclosamide alone, for 5 weeks (271-319 axons from 4…
View article: Supplementary Figure 5 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 5 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Modulation of DMF-induced cell death by DJ1 levels in OVCAR3 cells.
View article: Supplementary Figure 3 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 3 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Concentration-dependant cellular effects of DMF in OVCAR3 cells.
View article: Data from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Data from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
The transcription factor NRF2 (NFE2L2), regulates important antioxidant and cytoprotective genes. It enhances cancer cell proliferation and promotes chemoresistance in several cancers. Dimethyl fumarate (DMF) is known to promote NRF2 activ…
View article: Supplementary Table S2. Comparison of morphometric parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Supplementary Table S2. Comparison of morphometric parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Mean values {plus minus} SD of morphometric parameters of single myelinated axons isolated from sciatic nerves of mice injected with vehicle, oxaliplatin, oxaliplatin plus niclosamide or niclosamide alone, for 5 weeks (271-319 axons from 4…
View article: Figure Legends from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Figure Legends from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Supplementary Figure Legends
View article: Supplementary Figure 2 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 2 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
High concentrations of DMF causes caspase 3 and PARP cleavages in several cancer cell lines.
View article: Supplementary Figure 6 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 6 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Experimental protocol for establishment of inflammation-related mouse colon carcinogenesis along with representative IHC images of NRF2 expression in mice tumor.
View article: Figure S1. In Vivo Effects Of Oxaliplatin And Niclosamide On Mouse Motor Excitability from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Figure S1. In Vivo Effects Of Oxaliplatin And Niclosamide On Mouse Motor Excitability from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Excitability waveforms (means {plus minus} SD) were recorded at the plantar muscle in response to motor sciatic nerve stimulation of mice treated for 5 weeks with vehicle (black circles, n = 6), oxaliplatin (white circles, n = 7), oxalipla…
View article: Supplementary Figure 1 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 1 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Supplementary figure showing that high concentrations of DMF display cytotoxicity in several cancer cell lines
View article: Supplementary Figure 4 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 4 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Immunoblots showing the effect of DMF on NRF2 along with its associated proteins in various cancer cells.
View article: Supplementary Figure 2 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 2 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
High concentrations of DMF causes caspase 3 and PARP cleavages in several cancer cell lines.
View article: Supplementary Table S1. Comparison of neuromuscular excitability parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Supplementary Table S1. Comparison of neuromuscular excitability parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Derived excitability parameters (means {plus minus} SD) from plantar muscle recordings in response to motor sciatic nerve stimulation of mice treated for 5 weeks with vehicle (n = 6), oxaliplatin (n = 7), oxaliplatin plus niclosamide (n = …
View article: Figure S1. In Vivo Effects Of Oxaliplatin And Niclosamide On Mouse Motor Excitability from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Figure S1. In Vivo Effects Of Oxaliplatin And Niclosamide On Mouse Motor Excitability from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Excitability waveforms (means {plus minus} SD) were recorded at the plantar muscle in response to motor sciatic nerve stimulation of mice treated for 5 weeks with vehicle (black circles, n = 6), oxaliplatin (white circles, n = 7), oxalipla…
View article: Data from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Data from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Neuropathic pain is a limiting factor of platinum-based chemotherapies. We sought to investigate the neuroprotective potential of niclosamide in peripheral neuropathies induced by oxaliplatin. Normal neuron-like and cancer cells were treat…
View article: Supplementary Table 1 from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i>
Supplementary Table 1 from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i> Open
PDF file, 82K, Relationship between the dosage of sorafenib, serum AOPP levels and drug toxicity.
View article: Supplementary Table S1. Comparison of neuromuscular excitability parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Supplementary Table S1. Comparison of neuromuscular excitability parameters from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Derived excitability parameters (means {plus minus} SD) from plantar muscle recordings in response to motor sciatic nerve stimulation of mice treated for 5 weeks with vehicle (n = 6), oxaliplatin (n = 7), oxaliplatin plus niclosamide (n = …
View article: Data from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Data from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
The transcription factor NRF2 (NFE2L2), regulates important antioxidant and cytoprotective genes. It enhances cancer cell proliferation and promotes chemoresistance in several cancers. Dimethyl fumarate (DMF) is known to promote NRF2 activ…
View article: Supplementary Figure 5 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 5 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Modulation of DMF-induced cell death by DJ1 levels in OVCAR3 cells.
View article: Supplementary Table 1 from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i>
Supplementary Table 1 from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i> Open
PDF file, 82K, Relationship between the dosage of sorafenib, serum AOPP levels and drug toxicity.
View article: Figure Legends from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Figure Legends from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Supplementary Figure Legends
View article: Data from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i>
Data from Sorafenib-Induced Hepatocellular Carcinoma Cell Death Depends on Reactive Oxygen Species Production <i>In Vitro</i> and <i>In Vivo</i> Open
Sorafenib is presently the only effective therapy in advanced hepatocellular carcinoma (HCC). Because most anticancer drugs act, at least in part, through the generation of reactive oxygen species, we investigated whether sorafenib can ind…
View article: Supplementary Figure 6 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications
Supplementary Figure 6 from Dimethyl Fumarate Controls the NRF2/DJ-1 Axis in Cancer Cells: Therapeutic Applications Open
Experimental protocol for establishment of inflammation-related mouse colon carcinogenesis along with representative IHC images of NRF2 expression in mice tumor.
View article: Data from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response
Data from Niclosamide Inhibits Oxaliplatin Neurotoxicity while Improving Colorectal Cancer Therapeutic Response Open
Neuropathic pain is a limiting factor of platinum-based chemotherapies. We sought to investigate the neuroprotective potential of niclosamide in peripheral neuropathies induced by oxaliplatin. Normal neuron-like and cancer cells were treat…
View article: Supplementary Figure 2 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species
Supplementary Figure 2 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species Open
Supplementary Figure 2 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species
View article: Data from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species
Data from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species Open
Paradoxically, reactive oxygen species (ROS) can promote normal cellular proliferation and carcinogenesis, and can also induce apoptosis of tumor cells. In this report, we study the contribution of ROS to various cellular signals depending…
View article: Supplementary Figure 1 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species
Supplementary Figure 1 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species Open
Supplementary Figure 1 from Controlling Tumor Growth by Modulating Endogenous Production of Reactive Oxygen Species