Christian Rupp
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View article: Proteome Analysis of Biliary Extracellular Vesicles by High-Precision Mass Spectrometry Reveals Potential Biomarkers for Cholangiocarcinoma and Primary Sclerosing Cholangitis
Proteome Analysis of Biliary Extracellular Vesicles by High-Precision Mass Spectrometry Reveals Potential Biomarkers for Cholangiocarcinoma and Primary Sclerosing Cholangitis Open
View article: Promising Outcomes of Modified ALPPS for Staged Hepatectomy in Cholangiocarcinoma
Promising Outcomes of Modified ALPPS for Staged Hepatectomy in Cholangiocarcinoma Open
Associating liver partition and portal vein ligation for staged hepatectomy (ALPPS) is a two-stage procedure that can potentially cure patients with large cholangiocarcinoma. The current study evaluates the impact of modifications on the o…
View article: JNKs protect from cholestatic liver disease progression by modulating Apelin signalling
JNKs protect from cholestatic liver disease progression by modulating Apelin signalling Open
The cell-specific function of Jnk genes during cholestasis has not been explicitly explored. In this study, we showed that combined Jnk1/2, but not Jnk2 deficiency, in hepatocytes exacerbates liver damage and fibrosis …
View article: Supplementary Table S4 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S4 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Predicted risk group (BRBarray)_LMedits
View article: Supplementary Table S3 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S3 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
TLR4 signature genes
View article: Supplementary Table S2 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S2 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Key Resources Table
View article: Supplementary Table S7 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S7 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Control and cirrhosis patients
View article: Supplementary Table S6 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S6 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
MDSC signiture genes
View article: Supplementary table S8 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary table S8 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
16s rRNA sequencing of PSC patients
View article: Supplementary Table S6 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S6 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
MDSC signiture genes
View article: Supplementary Table S3 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S3 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
TLR4 signature genes
View article: Data from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Data from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Gut dysbiosis is commonly observed in patients with cirrhosis and chronic gastrointestinal disorders; however, its effect on antitumor immunity in the liver is largely unknown. Here we studied how the gut microbiome affects antitumor immun…
View article: Supplementary Table S5 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S5 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
PSC patients information
View article: Supplementary Table S4 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S4 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Predicted risk group (BRBarray)_LMedits
View article: Data from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Data from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Gut dysbiosis is commonly observed in patients with cirrhosis and chronic gastrointestinal disorders; however, its effect on antitumor immunity in the liver is largely unknown. Here we studied how the gut microbiome affects antitumor immun…
View article: Supplementary Table S5 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S5 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
PSC patients information
View article: Supplementary Table S1 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S1 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
LD1 CNV_Sequencing
View article: Supplementary figures and figure legends from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary figures and figure legends from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Supplementary figures and figure legends
View article: Supplementary Table S7 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S7 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Control and cirrhosis patients
View article: Supplementary Table S1 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S1 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
LD1 CNV_Sequencing
View article: Supplementary figures and figure legends from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary figures and figure legends from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Supplementary figures and figure legends
View article: Supplementary Table S2 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary Table S2 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
Key Resources Table
View article: Supplementary table S8 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma
Supplementary table S8 from Gut Microbiome Directs Hepatocytes to Recruit MDSCs and Promote Cholangiocarcinoma Open
16s rRNA sequencing of PSC patients
View article: Supplementary Figure 5 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 5 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 213K, C‐Raf, but not B‐Raf ablation, prevents tumor development in GDC‐0879 treated K5‐ SOS‐F+ mice.
View article: Supplementary Figure 8 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 8 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 185K, Effect of Rok inhibitors on K5‐SOS‐F+; C‐RafKO epithelia.
View article: Supplementary Figure 7 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 7 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 74K, B‐ and C‐Raf are required for PLX‐4032, but not GDC‐0879‐induced ERK phosphorylation in primary wild‐type keratinocytes.
View article: Supplementary Figure 4 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 4 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 680K, B‐Raf or C‐Raf ablation prevents tumor development in PLX‐4032‐treated KS‐SOS‐F+ mice.
View article: Data from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Data from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
RAF inhibitors achieve unprecedented but mainly transient clinical responses in patients with melanoma whose tumors harbor an activating BRAF mutation. One notable side-effect of RAF inhibitors is the stimulation of cutaneous skin tumors, …
View article: Supplementary Figure 3 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 3 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 1000K, The Mek inhibitor trametinib prevents Ras‐ and Ras+Raf inhibitor‐induced tumorigenesis.
View article: Supplementary Figure 2 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK
Supplementary Figure 2 from Skin Tumorigenesis Stimulated by Raf Inhibitors Relies Upon Raf Functions That Are Dependent and Independent of ERK Open
PDF file - 82K, Raf inhibitors activate ERK in human keratinocyte (HaCat) and squamous cell carcinoma (SCC) cell lines.