Christine E. Sheehan
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View article: Figure S3(A-C) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S3(A-C) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Increased expression of SNX1
View article: Figure S7 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S7 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
IGFBP3 in culture medium
View article: Figure S6 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S6 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Increased expression of IGFBP3 mRNA
View article: Figure S1 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S1 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
TNFAIP8 knockdown inhibits cell migration
View article: Supplementary materials from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Supplementary materials from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Supplementary methods, references and figure legends
View article: Data from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Data from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Aberrant regulation of EGFR is common in non–small cell lung carcinomas (NSCLC), and tumor resistance to targeted therapies has been attributed to emergence of other co-occurring oncogenic events, parallel bypass receptor tyrosine kinase p…
View article: Figure S7 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S7 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
IGFBP3 in culture medium
View article: Figure S5(A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S5(A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Suppression of pIGF1R
View article: Figure S2 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S2 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
qRT-PCR assay
View article: Figure S4 (A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S4 (A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
EGFR localization to early endosomes
View article: Figure S4 (A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S4 (A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
EGFR localization to early endosomes
View article: Supplementary materials from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Supplementary materials from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Supplementary methods, references and figure legends
View article: Figure S5(A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S5(A,B) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Suppression of pIGF1R
View article: Figure S3(A-C) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S3(A-C) from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Increased expression of SNX1
View article: Figure S1 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S1 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
TNFAIP8 knockdown inhibits cell migration
View article: Data from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Data from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Aberrant regulation of EGFR is common in non–small cell lung carcinomas (NSCLC), and tumor resistance to targeted therapies has been attributed to emergence of other co-occurring oncogenic events, parallel bypass receptor tyrosine kinase p…
View article: Figure S6 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S6 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
Increased expression of IGFBP3 mRNA
View article: Figure S2 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells
Figure S2 from Dual Targeting of EGFR and IGF1R in the TNFAIP8 Knockdown Non–Small Cell Lung Cancer Cells Open
qRT-PCR assay
View article: Data from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma
Data from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma Open
Purpose: Micropapillary urothelial carcinoma (MPUC) is a rare and aggressive form of bladder cancer. We conducted genomic analyses [next-generation sequencing (NGS)] of MPUC and non-micropapillary urothelial bladder carcinomas (non-MPUC) t…
View article: Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival Open
Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-XL and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
View article: Supplementary Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations
Supplementary Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations Open
Supplementary Data from Relapsed Classic E-Cadherin (CDH1)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of HER2 (ERBB2) Gene Mutations
View article: Supplementary Table 1 from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma
Supplementary Table 1 from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma Open
PDF file - 207K, Supplementary Table Genomic Alterations in 64 Cases of Relapsed/Metastatic Non-Micropapillary Urothelial Carcinoma of the Bladder.
View article: Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations
Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations Open
Purpose: We queried whether comprehensive genomic profiling using a next-generation sequencing–based assay could identify novel and unanticipated targets of therapy for patients with relapsed invasive lobular carcinoma (ILC).Experimental D…
View article: Supplementary Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations
Supplementary Data from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations Open
Supplementary Data from Relapsed Classic E-Cadherin (CDH1)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of HER2 (ERBB2) Gene Mutations
View article: Supplementary Table S1. from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations
Supplementary Table S1. from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations Open
Mutant Allele Frequencies of the Mutated Genes in 22 cases of ILC
View article: Supplementary Table S1. from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations
Supplementary Table S1. from Relapsed Classic E-Cadherin (<i>CDH1</i>)–Mutated Invasive Lobular Breast Cancer Shows a High Frequency of <i>HER2</i> (<i>ERBB2</i>) Gene Mutations Open
Mutant Allele Frequencies of the Mutated Genes in 22 cases of ILC
View article: Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival Open
Purpose: BLID is a BH3-like motif containing apoptotic member of the Bcl-2 family of proteins. This study was designed to investigate the mechanism of BLID-induced apoptosis and to assess the significance of BLID expression in breast cance…
View article: Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival Open
Supplementary Data from The Proapoptotic Molecule BLID Interacts with Bcl-XL and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
View article: Supplementary Table 1 from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma
Supplementary Table 1 from A High Frequency of Activating Extracellular Domain <i>ERBB2</i> (<i>HER2</i>) Mutation in Micropapillary Urothelial Carcinoma Open
PDF file - 207K, Supplementary Table Genomic Alterations in 64 Cases of Relapsed/Metastatic Non-Micropapillary Urothelial Carcinoma of the Bladder.
View article: Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival
Data from The Proapoptotic Molecule BLID Interacts with Bcl-X<sub>L</sub> and Its Downregulation in Breast Cancer Correlates with Poor Disease-Free and Overall Survival Open
Purpose: BLID is a BH3-like motif containing apoptotic member of the Bcl-2 family of proteins. This study was designed to investigate the mechanism of BLID-induced apoptosis and to assess the significance of BLID expression in breast cance…