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View article: Postoperative venous thromboembolism in transgender patients receiving estrogen-based hormone therapy
Postoperative venous thromboembolism in transgender patients receiving estrogen-based hormone therapy Open
View article: Issue Information
Issue Information Open
Transfusion requirements and complication rate in β-thalassemia intermedia due
View article: Estrogen‐based hormonal therapy and the risk of thrombosis in <scp>COVID</scp>‐19 patients
Estrogen‐based hormonal therapy and the risk of thrombosis in <span>COVID</span>‐19 patients Open
Objective Estrogen‐containing contraceptives and hormone replacement therapy are used commonly, however, the risks of venous and arterial thrombosis imparted by such medications during COVID‐19 infection or other similar viral infections r…
View article: Figure S6 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S6 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the investigations of tumorigenic phenotypes in the UT89 and UT42 isogenic cell line model systems
View article: Figure S5 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S5 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
PP2A Aα-, C-, and B55α-subunit protein abundance in PPP2R1A wildtype and P179-mutant patient EMCA tumor samples
View article: Figure S3 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S3 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data evaluating for disruption of PP2A holoenzyme assembly in a cell-free binding assay system
View article: Data from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Data from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Somatic mutation of the protein phosphatase 2A (PP2A) Aα-subunit gene PPP2R1A is highly prevalent in high-grade endometrial carcinoma. The structural, molecular, and biological basis by which the most recurrent endometrial carcinoma…
View article: Figure S5 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S5 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
PP2A Aα-, C-, and B55α-subunit protein abundance in PPP2R1A wildtype and P179-mutant patient EMCA tumor samples
View article: Figure S1 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S1 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data on PPP2R1A mutation in endometrial cancer (EMCA) and clinical and genomic features of P179 mutation-positive EMCA tumors, from the cBioportal cancer database
View article: Supplementary Figure Legends and Methods from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Supplementary Figure Legends and Methods from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplementary Figure Legends and Methods
View article: Figure S3 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S3 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data evaluating for disruption of PP2A holoenzyme assembly in a cell-free binding assay system
View article: Supplementary Figure Legends and Methods from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Supplementary Figure Legends and Methods from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplementary Figure Legends and Methods
View article: Figure S6 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S6 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the investigations of tumorigenic phenotypes in the UT89 and UT42 isogenic cell line model systems
View article: Figure S4 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S4 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the classical and enhanced sampling molecular dynamics simulations
View article: Figure S7 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S7 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the investigation of growth inhibition induced by SMAP treatment in EMCA
View article: Figure S7 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S7 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the investigation of growth inhibition induced by SMAP treatment in EMCA
View article: Data from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Data from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Somatic mutation of the protein phosphatase 2A (PP2A) Aα-subunit gene PPP2R1A is highly prevalent in high-grade endometrial carcinoma. The structural, molecular, and biological basis by which the most recurrent endometrial carcinoma…
View article: Figure S1 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S1 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data on PPP2R1A mutation in endometrial cancer (EMCA) and clinical and genomic features of P179 mutation-positive EMCA tumors, from the cBioportal cancer database
View article: Figure S4 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S4 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Supplemental data for the classical and enhanced sampling molecular dynamics simulations
View article: Figure S2 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S2 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Sequencing outcomes and PPP2R1A hotspot mutation frequency for the Case Comprehensive Cancer Center Gynecologic Tumor Biobank
View article: Figure S2 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis
Figure S2 from The Highly Recurrent PP2A Aα-Subunit Mutation P179R Alters Protein Structure and Impairs PP2A Enzyme Function to Promote Endometrial Tumorigenesis Open
Sequencing outcomes and PPP2R1A hotspot mutation frequency for the Case Comprehensive Cancer Center Gynecologic Tumor Biobank
View article: Re: Hormonal therapies and venous thrombosis: The estrogen matters! Morimont et al.
Re: Hormonal therapies and venous thrombosis: The estrogen matters! Morimont et al. Open
View article: Hormonal therapies and venous thrombosis: Considerations for prevention and management
Hormonal therapies and venous thrombosis: Considerations for prevention and management Open
When contemplating hormonal contraception or hormone replacement therapy, clinicians must consider a variety of factors including hormone type, dose, route, personal and family history of thrombosis, and other prothrombotic risk factors to…
View article: Loss of Forkhead box M1 promotes erythropoiesis through increased proliferation of erythroid progenitors
Loss of Forkhead box M1 promotes erythropoiesis through increased proliferation of erythroid progenitors Open
Forkhead box M1 (FOXM1) belongs to the forkhead/winged-helix family of transcription factors and regulates a network of proliferation-associated genes. Its abnormal upregulation has been shown to be a key driver of cancer progression and a…