Daniel Schäfer
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View article: CARIPRAZINE AUGMENTATION TO SSRIS IN TREATING A CASE OF TREATMENT-RESISTANT OBSESSIVE-COMPULSIVE DISORDER IN AN ADOLESCENT FEMALE
CARIPRAZINE AUGMENTATION TO SSRIS IN TREATING A CASE OF TREATMENT-RESISTANT OBSESSIVE-COMPULSIVE DISORDER IN AN ADOLESCENT FEMALE Open
Introduction: Obsessive-Compulsive Disorder (OCD) affects 1-3% of the population and often requires a combination of selective serotonin reuptake inhibitors and cognitive-behavioral therapy (CBT). However, 40-60% of patients remain treatme…
View article: Supplementary Figure Legends, Table Legends, Figures S1 - S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Figure Legends, Table Legends, Figures S1 - S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
The PDF includes legends for Supplementary Tables S1 - S5 as well as legends for Supplementary Figures S1 - S3. Supplementary Figure S1 indicates pathogens detected in mice held in the SPF and conventional animal facility. Supplementary Fi…
View article: Supplementary Table S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Pax5 target genes differentially express in p-BALL bearing Pax5+/- mice according to Delogu A. et al 2006, Schebesta A. et al 2007, and Revilla-i-Domingo R. et al 2012.
View article: Supplementary Table S1 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S1 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Health monitoring report of PAx5+/- held in the SPF and conventional animal facility.
View article: Data from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Data from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Earlier in the past century, infections were regarded as the most likely cause of childhood B-cell precursor acute lymphoblastic leukemia (pB-ALL). However, there is a lack of relevant biologic evidence supporting this hypothesis. We prese…
View article: Supplementary Table S1 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S1 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Health monitoring report of PAx5+/- held in the SPF and conventional animal facility.
View article: Supplementary Table S2 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S2 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Differentially expressed genes in bone marrow tumors from Pax5+/- mice compared to wild-type B220+ bone marrow B cells.
View article: Supplementary Table S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S3 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Pax5 target genes differentially express in p-BALL bearing Pax5+/- mice according to Delogu A. et al 2006, Schebesta A. et al 2007, and Revilla-i-Domingo R. et al 2012.
View article: Data from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Data from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Earlier in the past century, infections were regarded as the most likely cause of childhood B-cell precursor acute lymphoblastic leukemia (pB-ALL). However, there is a lack of relevant biologic evidence supporting this hypothesis. We prese…
View article: Supplementary Table S5 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S5 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
SNVs detected by whole exam sequencing in the three index Pax5+/- pB-ALL.
View article: Supplementary Table S5 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S5 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
SNVs detected by whole exam sequencing in the three index Pax5+/- pB-ALL.
View article: Supplementary Table S4 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S4 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Total numbers and percentage of hematopoietic subsets in young Pax5+/- and wild type mice.
View article: Supplementary Table S2 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S2 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Differentially expressed genes in bone marrow tumors from Pax5+/- mice compared to wild-type B220+ bone marrow B cells.
View article: Supplementary Table S4 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility
Supplementary Table S4 from Infection Exposure Is a Causal Factor in B-cell Precursor Acute Lymphoblastic Leukemia as a Result of <i>Pax5</i>-Inherited Susceptibility Open
Total numbers and percentage of hematopoietic subsets in young Pax5+/- and wild type mice.
View article: Supplementary Tables 1-3, 5-9,Supplementary Figures 1-5 from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases
Supplementary Tables 1-3, 5-9,Supplementary Figures 1-5 from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases Open
Contents: Supplementary Table 1: Characteristics of the pediatric patients enrolled in whole-genome- and whole-exome-sequencing. Supplementary Table 2: Conventional Facility Health Monitoring Report. Supplementary Table 3: Percentage of he…
View article: Data from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases
Data from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases Open
ETV6-RUNX1 is associated with the most common subtype of childhood leukemia. As few ETV6-RUNX1 carriers develop precursor B-cell acute lymphocytic leukemia (pB-ALL), the underlying genetic basis for development of full-blown leukemia remai…
View article: Data from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases
Data from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases Open
ETV6-RUNX1 is associated with the most common subtype of childhood leukemia. As few ETV6-RUNX1 carriers develop precursor B-cell acute lymphocytic leukemia (pB-ALL), the underlying genetic basis for development of full-blown leukemia remai…
View article: Supplementary Tables 1-3, 5-9,Supplementary Figures 1-5 from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases
Supplementary Tables 1-3, 5-9,Supplementary Figures 1-5 from Infection Exposure Promotes <i>ETV6-RUNX1</i> Precursor B-cell Leukemia via Impaired H3K4 Demethylases Open
Contents: Supplementary Table 1: Characteristics of the pediatric patients enrolled in whole-genome- and whole-exome-sequencing. Supplementary Table 2: Conventional Facility Health Monitoring Report. Supplementary Table 3: Percentage of he…
View article: Are Old Men Impotent? On a Sparse Discourse of Early Modern Medicine and Its Forensic Implications in Paolo Zacchia’s Quaestiones medico-legales
Are Old Men Impotent? On a Sparse Discourse of Early Modern Medicine and Its Forensic Implications in Paolo Zacchia’s Quaestiones medico-legales Open
In early modern medical literature, there are increasing references to sterility and impotence in older men. This is especially true of the Quaestiones medico-legales by the Roman physician Paolo Zacchia (1584–1659). In several books of th…
View article: Historical Considerations on the Ethics of Aging: Examples from the Sixteenth Century
Historical Considerations on the Ethics of Aging: Examples from the Sixteenth Century Open
The history of the ethics of agingi sac omplex and still neglectedt opic.From a scientifics tandpoint,wef irst need ad efinition: What does ethics mean today?(For instance, should we use anormative,consequentialist,ordeontological ethics?)…