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View article: Proteomic Profiles in Patients with Thrombosis Due to COVID-19 Are Distinct from Non-COVID-19 Thrombosis
Proteomic Profiles in Patients with Thrombosis Due to COVID-19 Are Distinct from Non-COVID-19 Thrombosis Open
BACKGROUND. COVID-19 is a prothrombotic disease, characterized by endotheliopathy, hypercoagulability, and thromboembolic complications. We hypothesized that the pathogenesis of thromboembolism associated with COVID-19 might differ from th…
View article: PRO-THROMBOTIC SIGNATURE FROM NEUTROPHIL ACTIVATION AND DECREASED ADAMTS13 TO VWF RATIO IS A KEY DRIVER OF CARDIAC INJURY IN HOSPITALIZED PATIENTS WITH COVID-19
PRO-THROMBOTIC SIGNATURE FROM NEUTROPHIL ACTIVATION AND DECREASED ADAMTS13 TO VWF RATIO IS A KEY DRIVER OF CARDIAC INJURY IN HOSPITALIZED PATIENTS WITH COVID-19 Open
View article: VWF/ADAMTS13 Ratios Are Potential Markers of Immunothrombotic Complications in Patients with COVID-19: A Cross-Sectional Study
VWF/ADAMTS13 Ratios Are Potential Markers of Immunothrombotic Complications in Patients with COVID-19: A Cross-Sectional Study Open
BACKGROUND. COVID-19 is a prothrombotic disease, characterized by both arterial and venous thrombosis. The pathogenesis of coagulopathy in COVID-19 is multifactorial, with activation of endothelial cells, platelets, neutrophils, and other …
View article: Dynamic intercellular redistribution of HIT antigen modulates heparin-induced thrombocytopenia
Dynamic intercellular redistribution of HIT antigen modulates heparin-induced thrombocytopenia Open
Key Points Development of thrombocytopenia in HIT is modulated by the (re)distribution of PF4 among hematopoietic and endothelial cell surfaces. Redistribution of PF4 from platelets to other hematopoietic cells may limit thrombocytopenia b…
View article: Platelet transactivation by monocytes promotes thrombosis in heparin-induced thrombocytopenia
Platelet transactivation by monocytes promotes thrombosis in heparin-induced thrombocytopenia Open
Key Points The procoagulant nature of HIT can be simulated in a microfluidic model using human blood and its components. PF4/glycosaminoglycans/immunoglobulin G complexes activate monocytes through FcγRIIA to generate TF and thrombin, lead…