Daniel Enríquez
YOU?
Author Swipe
View article: Ocular-adnexal non-Hodgkin lymphoma among hispanic Latino population: 18 years of experience
Ocular-adnexal non-Hodgkin lymphoma among hispanic Latino population: 18 years of experience Open
Background: Ocular-adnexal non-Hodgkin lymphoma (OANHL) is a rare lymphoid malignancy, accounting for only 1–2% of all non-Hodgkin lymphomas. Its development has been associated with endemic infections, such as Chlamydia species. However, …
View article: The effect of steroid-induced hyperglycemia in early mortality during induction treatment of Philadelphia-negative acute lymphoblastic leukemia in Latino young adults
The effect of steroid-induced hyperglycemia in early mortality during induction treatment of Philadelphia-negative acute lymphoblastic leukemia in Latino young adults Open
Introduction: Acute lymphoblastic leukemia (ALL) disproportionately affects Latino populations, who also exhibit a dismal prognosis. Especifically, early death has been described to have a high frequency in Latinos and adolescents/young ad…
View article: Survival outcomes and prognostic factors in children and adults with medulloblastoma from a Latin America country: A retrospective cohort
Survival outcomes and prognostic factors in children and adults with medulloblastoma from a Latin America country: A retrospective cohort Open
Background Few studies have evaluated the real-world outcomes of patients with medulloblastoma with contradictory results. Therefore, we aimed to compare the characteristics, survival outcomes, and prognostic factors between children and a…
View article: The Number of Extranodal Sites Matters! a Proposal for a Feasible Prognostic Model in Nodal PTCL. a <i>Collaborative Study</i> grupo De Estudio Latino-Americano De Linfoproliferativos <i>(GELL) & T-Cell Brazil Project (TCBP)</i>
The Number of Extranodal Sites Matters! a Proposal for a Feasible Prognostic Model in Nodal PTCL. a <i>Collaborative Study</i> grupo De Estudio Latino-Americano De Linfoproliferativos <i>(GELL) & T-Cell Brazil Project (TCBP)</i> Open
Introduction Peripheral T cell lymphoma (PTCL) accounts for 10-15% of all non-Hodgkin lymphomas. As previously described by our group (Fischer et al, 2023 ASH Meeting), Latin America has a distinct epidemiological PTCL distribution, with a…
View article: Leveraging a Novel Machine Learning Method to Predict Outcomes in Latin American (LATAM) Patients with Newly Diagnosed T and NK Cell Lymphomas (TNKL)
Leveraging a Novel Machine Learning Method to Predict Outcomes in Latin American (LATAM) Patients with Newly Diagnosed T and NK Cell Lymphomas (TNKL) Open
Introduction: Our PETAL consortium group recently characterized the largest global cohort of relapsed/refractory TNKL patients demonstrating statistically significant survival differences between patients in high-income and developing coun…
View article: CENTRAL NERVOUS SYSTEM INVOLVEMENT IN AGGRESSIVE ATLL: CAN WE PREDICT THE RISK IN A DEVASTATING COMPLICATION? EPIDEMIOLOGY AND CLINICAL FEATURES FROM LATIN AMERICA. A COLLABORATIVE STUDY FROM GRUPO DE ESTUDIO LATINO-AMERICANO DE LINFOPROLIFERATIVO (GELL) & T-CELL BRAZIL PROJECT (TCBP)
CENTRAL NERVOUS SYSTEM INVOLVEMENT IN AGGRESSIVE ATLL: CAN WE PREDICT THE RISK IN A DEVASTATING COMPLICATION? EPIDEMIOLOGY AND CLINICAL FEATURES FROM LATIN AMERICA. A COLLABORATIVE STUDY FROM GRUPO DE ESTUDIO LATINO-AMERICANO DE LINFOPROLIFERATIVO (GELL) & T-CELL BRAZIL PROJECT (TCBP) Open
Introduction: Adult T-cell leukemia/lymphoma (ATLL) is a mature, peripheral T-cell neoplasm caused by HTLV-1 and its lifetime risk is estimated as 4-7% among HTLV-1 carriers. Acute and lymphoma subtypes are highly aggressive diseases, char…
View article: Succinic semialdehyde derived from the gut microbiota can promote the proliferation of adult T-cell leukemia/lymphoma cells
Succinic semialdehyde derived from the gut microbiota can promote the proliferation of adult T-cell leukemia/lymphoma cells Open
Adult T-cell leukemia/lymphoma (ATLL) is a refractory blood cancer with severe immunodeficiency resulting from retroviral infection. ATLL develops in only 5 % of HTLV-1-infected individuals, but the entire mechanism of ATLL progression rem…
View article: THE NUMBER OF EXTRANODAL SITES IN NODAL PTCL: A PROPOSAL FOR A FEASIBLE PROGNOSTIC MARKER FOR OUTCOMES IN LOW-INCOME COUNTRIES. A COLLABORATIVE STUDY GRUPO DE ESTUDIO LATINO-AMERICANO DE LINFOPROLIFERATIVO (GELL) & T-CELL BRAZIL PROJECT (TCBP)
THE NUMBER OF EXTRANODAL SITES IN NODAL PTCL: A PROPOSAL FOR A FEASIBLE PROGNOSTIC MARKER FOR OUTCOMES IN LOW-INCOME COUNTRIES. A COLLABORATIVE STUDY GRUPO DE ESTUDIO LATINO-AMERICANO DE LINFOPROLIFERATIVO (GELL) & T-CELL BRAZIL PROJECT (TCBP) Open
Introduction: PTCL accounts for 10-15% of all NHL. As previously demonstrated, Latin America has its own epidemiological distribution, with a high frequency of ATLL and ENKT, likely influenced by distinct genetic profiles and viral epidemi…
View article: Geographic EBV variants confound disease-specific variant interpretation and predict variable immune therapy responses
Geographic EBV variants confound disease-specific variant interpretation and predict variable immune therapy responses Open
Epstein-Barr virus (EBV) is a potent carcinogen linked to hematologic and solid malignancies and causes significant global morbidity and mortality. Therapy using allogeneic EBV-specific lymphocytes shows promise in certain populations, but…
View article: Molecular-guided therapy for the treatment of patients with relapsed and refractory childhood cancers: a Beat Childhood Cancer Research Consortium trial
Molecular-guided therapy for the treatment of patients with relapsed and refractory childhood cancers: a Beat Childhood Cancer Research Consortium trial Open
Background Children with relapsed central nervous system (CNS tumors), neuroblastoma, sarcomas, and other rare solid tumors face poor outcomes. This prospective clinical trial examined the feasibility of combining genomic and transcriptomi…
View article: Brain Metastasis in Triple‐Negative Breast Cancer
Brain Metastasis in Triple‐Negative Breast Cancer Open
Background . Breast cancer is an important cause of cancer‐related death in women worldwide and represents the second most frequent cause of brain metastases after lung cancer. The aim of this study was to determine the characteristics and…
View article: Understanding the Immunopathology of HTLV-1-Associated Adult T-Cell Leukemia/Lymphoma: A Comprehensive Review
Understanding the Immunopathology of HTLV-1-Associated Adult T-Cell Leukemia/Lymphoma: A Comprehensive Review Open
Human T-cell leukemia virus type-1 (HTLV-1) causes adult T-cell leukemia/lymphoma (ATL). HTLV-1 carriers have a lifelong asymptomatic balance between infected cells and host antiviral immunity; however, 5–10% of carriers lose this balance …
View article: Real-World Outcomes of Adolescents and Young Adults with Diffuse Large B-Cell Lymphoma: A Multicenter Retrospective Cohort Study
Real-World Outcomes of Adolescents and Young Adults with Diffuse Large B-Cell Lymphoma: A Multicenter Retrospective Cohort Study Open
Purpose: Patients with diffuse large B-cell lymphoma (DLBCL) are typically treated with rituximab, cyclophosphamide, doxorubicin, vincristine, and prednisone (R-CHOP). However, a standard of care for managing adolescents and young adults (…
View article: Understanding the Immunopathology of HTLV-1-Associated Adult T-Cell Leukemia/Lymphoma: A Comprehensive Review
Understanding the Immunopathology of HTLV-1-Associated Adult T-Cell Leukemia/Lymphoma: A Comprehensive Review Open
Human T-cell leukemia virus type-1 (HTLV-1) is the causative agent of adult T-cell leukemia/lymphoma (ATL). HTLV-1 carriers have a life-long asymptomatic balance between infected cells and host antiviral immunity, but 5–10% of carriers los…
View article: Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Extranodal natural killer/T-cell lymphoma (ENKTL) is an aggressive, rare lymphoma of natural killer (NK) cell origin with poor clinical outcomes. Here we used phenotypic and molecular profiling, including epigenetic analyses, to investigat…
View article: Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Extranodal natural killer/T-cell lymphoma (ENKTL) is an aggressive, rare lymphoma of natural killer (NK) cell origin with poor clinical outcomes. Here we used phenotypic and molecular profiling, including epigenetic analyses, to investigat…
View article: Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Figure from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma Open
Supplementary Data from Identification and Targeting of the Developmental Blockade in Extranodal Natural Killer/T-cell Lymphoma
View article: Figure S6 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S6 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Additional Longitudinal Analysis of Relapsed and Refractory Childhood Solid Tumors.
View article: Figure S4 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S4 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Significantly Differentially Expressed Transcription Factors and Drug Targets in Relapsed and Refractory Childhood Solid Tumors.
View article: Figure S5 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S5 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Consensus Matrix of the GSVA Enrichment Scores Computed for 50 Hallmark Gene Sets. xCell Immune Score Values by GSVA Cluster.
View article: Figure S3 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S3 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Most Variably Expressed Genes in Relapsed and Refractory Childhood Solid Tumors.
View article: Figure S5 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S5 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Consensus Matrix of the GSVA Enrichment Scores Computed for 50 Hallmark Gene Sets. xCell Immune Score Values by GSVA Cluster.
View article: Figure S1 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S1 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
GISTIC Analysis of 184 Relapsed and Refractory Childhood Solid Tumors.
View article: Figure S3 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion
Figure S3 from Genomic and Transcriptomic Analysis of Relapsed and Refractory Childhood Solid Tumors Reveals a Diverse Molecular Landscape and Mechanisms of Immune Evasion Open
Most Variably Expressed Genes in Relapsed and Refractory Childhood Solid Tumors.