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View article: Supplementary Table 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Table 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
XLS file - 8K, describing KEGG pathway analysis results
View article: Data from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Data from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
Patients with the genetic skin blistering disease recessive dystrophic epidermolysis bullosa (RDEB) develop aggressive cutaneous squamous cell carcinoma (cSCC). Metastasis leading to mortality is greater in RDEB than in other patient group…
View article: Supplementary Figure 3 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 3 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 409K, Invasion of SCCRDEB4 keratinocytes into RDEB cell derived matrix is increased after 1 week compared with equivalent non-RDEB cell derived matrix. Representative images from pan-keratin stained images of 1-week invasion of …
View article: Supplementary Table 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Table 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
XLS file - 36K, Table describing the patient samples used in this study
View article: Supplementary Table 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Table 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
XLS file - 36K, Table describing the patient samples used in this study
View article: Supplementary Methods from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Methods from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 382K, Includes Microarray and Expression Analysis, BRB Array Clustering Analysis, and Tumorigenicity assays
View article: Supplementary Figure 4 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 4 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 79K, A: Average array signal intensities �SD for the single probe representing COL7A1, n=3-5. B: Western blotting shows the level of type VII collagen varies between sample groups, increased in both UVSCCF samples compared with …
View article: Data from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Data from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
Patients with the genetic skin blistering disease recessive dystrophic epidermolysis bullosa (RDEB) develop aggressive cutaneous squamous cell carcinoma (cSCC). Metastasis leading to mortality is greater in RDEB than in other patient group…
View article: Supplementary Figure 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 190K, A: RDEBSCCF derived matrix promotes adhesion of keratinocytes compared with matrix derived from other fibroblast groups (* = p<0.01). All data normalized to collagen I adhesion, results are the mean of three separate exper…
View article: Supplementary Figure 4 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 4 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 79K, A: Average array signal intensities �SD for the single probe representing COL7A1, n=3-5. B: Western blotting shows the level of type VII collagen varies between sample groups, increased in both UVSCCF samples compared with …
View article: Supplementary Figure 3 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 3 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 409K, Invasion of SCCRDEB4 keratinocytes into RDEB cell derived matrix is increased after 1 week compared with equivalent non-RDEB cell derived matrix. Representative images from pan-keratin stained images of 1-week invasion of …
View article: Supplementary Figure 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 56K, Unsupervised clustering of the core serum response gene set is able to cluster NHF but not RDEBSCCF sample groups in culture
View article: Supplementary Figure 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 1 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 56K, Unsupervised clustering of the core serum response gene set is able to cluster NHF but not RDEBSCCF sample groups in culture
View article: Supplementary Methods from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Methods from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 382K, Includes Microarray and Expression Analysis, BRB Array Clustering Analysis, and Tumorigenicity assays
View article: Supplementary Table 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Table 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
XLS file - 8K, describing KEGG pathway analysis results
View article: Supplementary Figure 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa
Supplementary Figure 2 from Fibroblast-Derived Dermal Matrix Drives Development of Aggressive Cutaneous Squamous Cell Carcinoma in Patients with Recessive Dystrophic Epidermolysis Bullosa Open
PDF file - 190K, A: RDEBSCCF derived matrix promotes adhesion of keratinocytes compared with matrix derived from other fibroblast groups (* = p<0.01). All data normalized to collagen I adhesion, results are the mean of three separate exper…
View article: Intracytoplasmic Retention of Type VII Collagen and Dominant Dystrophic Epidermolysis Bullosa: Reversal of Defect Following Cessation of or Marked Improvement in Disease Activity
Intracytoplasmic Retention of Type VII Collagen and Dominant Dystrophic Epidermolysis Bullosa: Reversal of Defect Following Cessation of or Marked Improvement in Disease Activity Open
It has been recently shown that the presence of perinuclear "stellate bodies" within the epidermis in patients with a form of dominant dystrophic epidermolysis bullosa named "transient bullous dermolysis of the newborn" corresponds to coll…
View article: Epitope Mapping of the Laminin Molecule in Murine Skin Basement Membrane Zone: Demonstration of Spatial Differences in Ultrastructural Localization
Epitope Mapping of the Laminin Molecule in Murine Skin Basement Membrane Zone: Demonstration of Spatial Differences in Ultrastructural Localization Open
Results of studies performed to date with polyclonal anti-laminin antibodies have been conflicting as to the ultrastructural localization of this glycoprotein in skin basement membrane zone (BMZ). Whereas initial reports suggested its pres…
View article: Genetic Basis of Dominantly Inherited Transient Bullous Dermolysis of the Newborn: A Splice Site Mutation in the Type VII Collagen Gene
Genetic Basis of Dominantly Inherited Transient Bullous Dermolysis of the Newborn: A Splice Site Mutation in the Type VII Collagen Gene Open
Transient bullous dermolysis of the newborn (TBDN) is a blistering disease evident at birth or shortly thereafter, but the blistering tendency decreases with advancing age. The tissue separation in TBDN is below the lamina densa, and elect…
View article: Normal Molecular Weight of Type VII Collagen Produced by Recessive Dystrophic Epidermolysis Bullosa Keratinocytes
Normal Molecular Weight of Type VII Collagen Produced by Recessive Dystrophic Epidermolysis Bullosa Keratinocytes Open
Studies of the recessive dystrophic form of epidermolysis bullosa (RDEB) have suggested that an abnormality in type VII collagen may be involved in the pathogenesis of this disorder. Indirect immunofluorescence studies have shown that the …
View article: The Presence of Intra-lamina Lucida Blister Formation in Epidermolysis Bullosa Acquisita: Possible Role of Leukocytes
The Presence of Intra-lamina Lucida Blister Formation in Epidermolysis Bullosa Acquisita: Possible Role of Leukocytes Open
In evaluating patients we have noted disparity between the locations of bound immunoreactants and the level of blistering in epidermolysis bullosa acquisita (EBA). We examined 10 consecutive EBA patients by routine histology, direct (DIF) …
View article: Consensus reclassification of inherited epidermolysis bullosa and other disorders with skin fragility
Consensus reclassification of inherited epidermolysis bullosa and other disorders with skin fragility Open
The proposed classification scheme should be of value both to clinicians and researchers, emphasizing both clinical and genetic features of EB. What is already known about this topic? Epidermolysis bullosa (EB) is a group of genetic disord…
View article: Drug-induced linear IgA bullous dermatosis in a patient with a vancomycin-impregnated cement spacer.
Drug-induced linear IgA bullous dermatosis in a patient with a vancomycin-impregnated cement spacer. Open
Linear IgA bullous dermatosis (LABD) is an autoimmune blistering rash caused by IgA autoantibodies against the epidermal basement membrane zone. It commonly is drug induced, often in association with systemic vancomycin. We report a case o…
View article: Epidemiology of Inherited Epidermolysis Bullosa Based on Incidence and Prevalence Estimates From the National Epidermolysis Bullosa Registry
Epidemiology of Inherited Epidermolysis Bullosa Based on Incidence and Prevalence Estimates From the National Epidermolysis Bullosa Registry Open
Precise estimates of the incidence and prevalence of each major subtype of inherited EB in the United States are now available that should assist investigators in choosing which subtypes are amenable to properly designed, large-scale, clin…