Hongyan Diao
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View article: Study on human papillomavirus (HPV) infection, typing, peripheral blood immune factor expression and vaginal microenvironment balance in cervical cancer screening population
Study on human papillomavirus (HPV) infection, typing, peripheral blood immune factor expression and vaginal microenvironment balance in cervical cancer screening population Open
HPV, particularly HPV16/18, is associated with immune imbalance and vaginal microenvironment disruptions, highlighting the need for tailored strategies to improve CC prevention and diagnosis.
View article: S100A8/A9-activated IFNγ+ NK cells trigger β-cell necroptosis in hepatitis B virus-associated liver cirrhosis
S100A8/A9-activated IFNγ+ NK cells trigger β-cell necroptosis in hepatitis B virus-associated liver cirrhosis Open
Together, the data indicate that IFN-γ produced by NK cells induces β cell necroptosis via the S100A8/A9-RAGE-p38 MAPK axis in patients with LC and DM. Reduced levels of S100A8/A9, NK cells, and IFN-γ could be valuable for the treatment of…
View article: Figure S4 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S4 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S4 shows the extended data of "ALKBH5-mediated m6A demethylation in 3’UTR region of SNAI2 mRNA is recognized by YTHDF2"
View article: Supplementary Table 3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 3 shows primer sequences
View article: Supplementary Table 2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 2 shows the Sequences of oligonucleotides for siRNA/shRNA
View article: Figure S5 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S5 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S5 shows the extended data of "SLUG reversed the stemness properties and immune escape induced by ALKBH5"
View article: Figure S1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S1 shows the extended data of "Enhanced malignancy and declined m6A levels in HBV-related HCC"
View article: Supplementary Table 3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 3 shows primer sequences
View article: Supplementary Table 1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 1 shows the general description of patients
View article: Figure S2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S2 shows the extended data of "HBV up-regulated ALKBH5 in HBV-positive HCC"
View article: Figure S2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S2 shows the extended data of "HBV up-regulated ALKBH5 in HBV-positive HCC"
View article: Figure S1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S1 shows the extended data of "Enhanced malignancy and declined m6A levels in HBV-related HCC"
View article: Supplementary Table 2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 2 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 2 shows the Sequences of oligonucleotides for siRNA/shRNA
View article: Figure S5 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S5 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S5 shows the extended data of "SLUG reversed the stemness properties and immune escape induced by ALKBH5"
View article: Figure S4 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S4 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S4 shows the extended data of "ALKBH5-mediated m6A demethylation in 3’UTR region of SNAI2 mRNA is recognized by YTHDF2"
View article: Data from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Data from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Hepatitis B viral (HBV) persistent infection plays a significant role in hepatocellular carcinoma (HCC) tumorigenesis. Many studies have revealed the pivotal roles of N6-methyladenosine (m6A) in multiple cancers, while the regulatory mecha…
View article: Data from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Data from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Hepatitis B viral (HBV) persistent infection plays a significant role in hepatocellular carcinoma (HCC) tumorigenesis. Many studies have revealed the pivotal roles of N6-methyladenosine (m6A) in multiple cancers, while the regulatory mecha…
View article: Supplementary Table 1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Supplementary Table 1 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Table 1 shows the general description of patients
View article: Figure S3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S3 shows the extended data of "ALKBH5 is required for the maintenance of hepatoma cell self-renewal in HBV-positive HCC"
View article: Figure S3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Figure S3 from Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Supplementary Figure S3 shows the extended data of "ALKBH5 is required for the maintenance of hepatoma cell self-renewal in HBV-positive HCC"
View article: Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape
Hepatitis B Virus–Mediated m6A Demethylation Increases Hepatocellular Carcinoma Stemness and Immune Escape Open
Hepatitis B viral (HBV) persistent infection plays a significant role in hepatocellular carcinoma (HCC) tumorigenesis. Many studies have revealed the pivotal roles of N6-methyladenosine (m6A) in multiple cancers, while the regulatory mecha…
View article: Faecal hsa-miR-7704 inhibits the growth and adhesion of Bifidobacterium longum by suppressing ProB and aggravates hepatic encephalopathy
Faecal hsa-miR-7704 inhibits the growth and adhesion of Bifidobacterium longum by suppressing ProB and aggravates hepatic encephalopathy Open
Both gut microbiome and microRNAs (miRNAs) play a role in the development of hepatic encephalopathy (HE). However, the functional link between the microbiome and host-derived miRNAs in faeces remains poorly understood. In the present study…
View article: M1 macrophage‐derived exosomes promote autoimmune liver injury by transferring long noncoding RNA H19 to hepatocytes
M1 macrophage‐derived exosomes promote autoimmune liver injury by transferring long noncoding RNA H19 to hepatocytes Open
Exosomes mediate intercellular communication by transmitting active molecules. The function of long noncoding RNA (lncRNA) H19 in autoimmune liver injury is unclear. Concanavalin A (ConA)‐induced liver injury is well‐characterized immune‐m…
View article: WCN23-0949 PGC1α MODULATES MITOCHONDRIAL HOMEOSTASIS IN TUBULAR CELLS OF DIABETIC KIDNEY DISEASE
WCN23-0949 PGC1α MODULATES MITOCHONDRIAL HOMEOSTASIS IN TUBULAR CELLS OF DIABETIC KIDNEY DISEASE Open
Tubular injury and dysregulated mitochondrial dynamics play pivotal roles in the pathogenesis of diabetic kidney disease (DKD). However, the potential mechanism by which mitochondrial dysfunction-initiated tubulopathy triggers intrarenal s…