Dina Stepanova
YOU?
Author Swipe
View article: SONOCHEMICAL APPROACH TO OBTAIN BIOMASS OF YEAST SACCHAROMYCES CEREVISIAE WITH HIGH ERGOSTEROL CONTENT
SONOCHEMICAL APPROACH TO OBTAIN BIOMASS OF YEAST SACCHAROMYCES CEREVISIAE WITH HIGH ERGOSTEROL CONTENT Open
Chemical synthesis of vitamin D2, an important nutrient for the human body, is dif ficult due to the asymmetric structure of the molecule of this vitamin. For this reason, the pro spects for the development of biotechnological approaches f…
View article: Intensification of the process of ergosterol accumulation by yeast Saccharomyces cerevisiae by changing the composition of the culture medium and using ultrasound
Intensification of the process of ergosterol accumulation by yeast Saccharomyces cerevisiae by changing the composition of the culture medium and using ultrasound Open
The chemical synthesis of vitamin D2, an important nutrient for living organisms, is complex due to the asymmetric structure of the vitamin molecule. This opens up significant prospects for the development of biotechnological approaches to…
View article: Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis
Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis Open
Neurofibromatosis type 2 (NF2) is an autosomal dominant disorder characterized by the development of multiple tumors in the central nervous system, most notably schwannomas, and meningiomas. Mutational inactivation of the NF2 gene e…
View article: Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis
Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis Open
Neurofibromatosis type 2 (NF2) is an autosomal dominant disorder characterized by the development of multiple tumors in the central nervous system, most notably schwannomas, and meningiomas. Mutational inactivation of the NF2 gene e…
View article: Supplementary Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis
Supplementary Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis Open
Supplemental files describing metabolic alterations in NF2-deficient cells, supplementary materials and methods, and supportive data regarding the effects of FASN inhibitors or knockdown on various metabolic processes in WT and NF2-deficie…
View article: Supplementary Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis
Supplementary Data from An Essential Role for the Tumor-Suppressor Merlin in Regulating Fatty Acid Synthesis Open
Supplemental files describing metabolic alterations in NF2-deficient cells, supplementary materials and methods, and supportive data regarding the effects of FASN inhibitors or knockdown on various metabolic processes in WT and NF2-deficie…
View article: Supplementary Table 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 58K, Specificity of Pak inhibitors
View article: Supplementary Figure Legends 1-5, Table Legends 1-3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure Legends 1-5, Table Legends 1-3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 131K
View article: Supplementary Figure 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 126K, Activity of FRAX-597 against group A and group B Paks
View article: Supplementary Figure 4 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 4 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 162K, Proliferation and apoptosis in drug-treated animals
View article: Supplementary Table 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 99K, Immunostaining for Pak1 and Pak2 in mouse skin
View article: Data from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Data from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
The RAS genes are the most commonly mutated oncogenes in human cancer and present a particular therapeutic dilemma, as direct targeting of Ras proteins by small molecules has proved difficult. Signaling pathways downstream of Ras, i…
View article: Supplementary Figure 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 219K, Immunostaining for Pak1 and Pak2 in mouse skin
View article: Supplementary Figure 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 232K, Immunohistochemical analysis of P-Erk, P-Akt, P-S6, and beta-catenin in tumors arising in K5-tTR::tet-Kras mice
View article: Supplementary Methods from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Methods from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 153K
View article: Data from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Data from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
The RAS genes are the most commonly mutated oncogenes in human cancer and present a particular therapeutic dilemma, as direct targeting of Ras proteins by small molecules has proved difficult. Signaling pathways downstream of Ras, i…
View article: Supplementary Figure 5 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 5 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 60K, Schematic presentation of Ras-mediated Pak1 signaling
View article: Supplementary Table 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 99K, Immunostaining for Pak1 and Pak2 in mouse skin
View article: Supplementary Figure 5 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 5 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 60K, Schematic presentation of Ras-mediated Pak1 signaling
View article: Supplementary Figure 4 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 4 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 162K, Proliferation and apoptosis in drug-treated animals
View article: Supplementary Figure 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 126K, Activity of FRAX-597 against group A and group B Paks
View article: Supplementary Methods from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Methods from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 153K
View article: Supplementary Table 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 289K, Immunostaining of Pak1 and proliferative signaling markers in SCC
View article: Supplementary Table 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 289K, Immunostaining of Pak1 and proliferative signaling markers in SCC
View article: Supplementary Figure Legends 1-5, Table Legends 1-3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure Legends 1-5, Table Legends 1-3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 131K
View article: Supplementary Figure 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 2 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 232K, Immunohistochemical analysis of P-Erk, P-Akt, P-S6, and beta-catenin in tumors arising in K5-tTR::tet-Kras mice
View article: Supplementary Figure 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Figure 1 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 219K, Immunostaining for Pak1 and Pak2 in mouse skin
View article: Supplementary Table 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model
Supplementary Table 3 from p21-Activated Kinase 1 Is Required for Efficient Tumor Formation and Progression in a Ras-Mediated Skin Cancer Model Open
PDF file - 58K, Specificity of Pak inhibitors
View article: Composite phospholipid-gold nanoparticles with targeted fragment for tumor imaging
Composite phospholipid-gold nanoparticles with targeted fragment for tumor imaging Open
Gold nanoparticles and their conjugates have significant potential in the field of diagnosis of various diseases due to their SPR, which enhances light scattering and absorption. Conjugates of gold nanoparticles with various ligands can be…
View article: Усиление активатором пируватдегидрогеназы дихлорацетатом натрия антипролиферативного действия доксорубицина на опухолевых клетках
Усиление активатором пируватдегидрогеназы дихлорацетатом натрия антипролиферативного действия доксорубицина на опухолевых клетках Open
Выявлено достоверное усиление цитотоксических свойств доксорубицина под влиянием дихлорацетата натрия (ДХА) в концентрации 100 мкМ и не выявлено усиление ДХА антипролиферативной активности цисплатина, винкристина и этопозида на клетках лин…