David F. Vincent
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View article: SERRATUS ANTERIOR PLANE BLOCK FOR MULTIPLE RIB FRACTURES-A NOVEL PAIN MANAGEMENT TECHNIQUE
SERRATUS ANTERIOR PLANE BLOCK FOR MULTIPLE RIB FRACTURES-A NOVEL PAIN MANAGEMENT TECHNIQUE Open
View article: Keeping Community During a Pandemic
Keeping Community During a Pandemic Open
The purpose of this qualitative exploratory study was to better understand how LGBTQ+ senior centers created virtual communities and implemented organizational adaptation in the earliest stages of the pandemic. Three focus groups (n=22) we…
View article: Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
ECT2 is an activator of RHO GTPases that is essential for cytokinesis. In addition, ECT2 was identified as an oncoprotein when expressed ectopically in NIH/3T3 fibroblasts. However, oncogenic activation of ECT2 resulted from N-terminal tru…
View article: Supplementary Figures from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Supplementary Figures from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
Supplementary Figures S1-S4
View article: Supplementary Figures from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Supplementary Figures from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
Supplementary Figures S1-S4
View article: Supplementary Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Supplementary Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
Supplementary Tables and Methods.
View article: Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
ECT2 is an activator of RHO GTPases that is essential for cytokinesis. In addition, ECT2 was identified as an oncoprotein when expressed ectopically in NIH/3T3 fibroblasts. However, oncogenic activation of ECT2 resulted from N-terminal tru…
View article: Supplementary Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Supplementary Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
Supplementary Tables and Methods.
View article: Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Data from Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
ECT2 is an activator of RHO GTPases that is essential for cytokinesis. In addition, ECT2 was identified as an oncoprotein when expressed ectopically in NIH/3T3 fibroblasts. However, oncogenic activation of ECT2 resulted from N-terminal tru…
View article: Supplementary Figure S3 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S3 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in HEK-293T and HepG2 cells rapidly results in cell cycle blockade in G2/M phases and mitotic catastrophe events.
View article: Supplementary Table S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Table S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
List of all up-regulated genes in Tif1γ-/--primary MEFs compared to control-primary MEFs (fold {greater than or equal to}1.70).
View article: Supplementary Figure S3 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S3 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in HEK-293T and HepG2 cells rapidly results in cell cycle blockade in G2/M phases and mitotic catastrophe events.
View article: Supplementary Table S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Table S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
List of all up-regulated genes in Tif1γ-/--primary MEFs compared to control-primary MEFs (fold {greater than or equal to}1.70).
View article: Supplementary Figure S2 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S2 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in primary mouse embryonic fibroblasts rapidly results in cell cycle blockade in G2/M phases, spindle checkpoint activation and mitotic catastrophe events.
View article: Data from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Data from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
The transcription accessory factor TIF1γ/TRIM33/RFG7/PTC7/Ectodermin functions as a tumor suppressor that promotes development and cellular differentiation. However, its precise function in cancer has been elusive. In the present st…
View article: Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
View article: Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Supplementary Figure Legends from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
View article: Supplementary Figure S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in spontaneously immortalized mouse embryonic fibroblasts rapidly results in cell cycle blockade in G2/M phases, spindle checkpoint activation and mitotic catastrophe events.
View article: Supplementary Methods and References from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Methods and References from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Description of additional methods and procedures used in the study. Also includes Supplementary References.
View article: Data from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Data from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
The transcription accessory factor TIF1γ/TRIM33/RFG7/PTC7/Ectodermin functions as a tumor suppressor that promotes development and cellular differentiation. However, its precise function in cancer has been elusive. In the present st…
View article: Supplementary Figure S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S1 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in spontaneously immortalized mouse embryonic fibroblasts rapidly results in cell cycle blockade in G2/M phases, spindle checkpoint activation and mitotic catastrophe events.
View article: Supplementary Methods and References from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Methods and References from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Description of additional methods and procedures used in the study. Also includes Supplementary References.
View article: Supplementary Figure S2 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability
Supplementary Figure S2 from TIF1γ Suppresses Tumor Progression by Regulating Mitotic Checkpoints and Chromosomal Stability Open
Inactivation of Tif1γ in primary mouse embryonic fibroblasts rapidly results in cell cycle blockade in G2/M phases, spindle checkpoint activation and mitotic catastrophe events.
View article: Author Correction: Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features
Author Correction: Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features Open
View article: Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features
Epithelial TGFβ engages growth-factor signalling to circumvent apoptosis and drive intestinal tumourigenesis with aggressive features Open
The pro-tumourigenic role of epithelial TGFβ signalling in colorectal cancer (CRC) is controversial. Here, we identify a cohort of born to be bad early-stage (T1) colorectal tumours, with aggressive features and a propensity to disseminate…
View article: Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth
Aberrant Expression and Subcellular Localization of ECT2 Drives Colorectal Cancer Progression and Growth Open
ECT2 is an activator of RHO GTPases that is essential for cytokinesis. In addition, ECT2 was identified as an oncoprotein when expressed ectopically in NIH/3T3 fibroblasts. However, oncogenic activation of ECT2 resulted from N-terminal tru…
View article: Oncogenic BRAF, unrestrained by TGFβ-receptor signalling, drives right-sided colonic tumorigenesis
Oncogenic BRAF, unrestrained by TGFβ-receptor signalling, drives right-sided colonic tumorigenesis Open
View article: A RAC-GEF network critical for early intestinal tumourigenesis
A RAC-GEF network critical for early intestinal tumourigenesis Open
View article: Generation of an Fsp1 (fibroblast‐specific protein 1)‐Flpo transgenic mouse strain
Generation of an Fsp1 (fibroblast‐specific protein 1)‐Flpo transgenic mouse strain Open
Summary Recombination systems represent a major breakthrough in the field of genetic model engineering. The Flp recombinases (Flp, Flpe, and Flpo) bind and cleave DNA Frt sites. We created a transgenic mouse strain ([Fsp1‐Flpo]) expressing…