Dmitry Malin
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View article: Maternal Chronic Ultrasound Stress Provokes Immune Activation and Behavioral Deficits in the Offspring: A Mouse Model of Neurodevelopmental Pathology
Maternal Chronic Ultrasound Stress Provokes Immune Activation and Behavioral Deficits in the Offspring: A Mouse Model of Neurodevelopmental Pathology Open
Neurodevelopmental disorders stemming from maternal immune activation can significantly affect a child’s life. A major limitation in pre-clinical studies is the scarcity of valid animal models that accurately mimic these challenges. Among …
View article: Analysis of the Serie of Cases of Neuroleptic Malignant Syndrome (Cross-Sectional Observational Study)
Analysis of the Serie of Cases of Neuroleptic Malignant Syndrome (Cross-Sectional Observational Study) Open
Background : neuroleptic malignant syndrome (NMS) is the most dangerous complication of therapy with neuroleptics with high mortality. The publications on MNS are the reviews articles or case reports. The aim of study : an identification of…
View article: Data from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Data from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
The initiation of androgen-deprivation therapy (ADT) induces susceptibilities in prostate cancer cells that make them vulnerable to synergistic treatment and enhanced cell death. Senescence results in cell-cycle arrest, but cells remain vi…
View article: Supplementary Table S1 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Supplementary Table S1 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
All supplementary table
View article: Supplementary Figure S1_S4 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Supplementary Figure S1_S4 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
All supplementery figures
View article: Supplementary Table S1 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Supplementary Table S1 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
All supplementary table
View article: Data from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Data from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
The initiation of androgen-deprivation therapy (ADT) induces susceptibilities in prostate cancer cells that make them vulnerable to synergistic treatment and enhanced cell death. Senescence results in cell-cycle arrest, but cells remain vi…
View article: Supplementary Figure S1_S4 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin
Supplementary Figure S1_S4 from Synthetic Lethal Metabolic Targeting of Androgen-Deprived Prostate Cancer Cells with Metformin Open
All supplementery figures
View article: Supplementary Figure S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Figure S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Figure S1. Methionine restriction inhibits cell proliferation.
View article: Supplementary Figures 1 - 5, Tables 1 - 4 from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain
Supplementary Figures 1 - 5, Tables 1 - 4 from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain Open
PDF file - 797K, Figure S1. Barrier phenotype of human brain microvascular endothelial cells and astrocytes in monocultures and co-cultures. Figure S2. alpha/beta-crystallin does not affect cell viability of TNBC cells in standard monolaye…
View article: Supplementary Table S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Table S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Table S1. Composition of diets.
View article: Supplementary Table S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Table S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Table S1. Composition of diets.
View article: Data from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain
Data from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain Open
Purpose: Basal-like breast tumors are typically (ER/PR/HER2) triple-negative and are associated with a high incidence of brain metastases and poor clinical outcomes. The molecular chaperone αB-crystallin is predominantly expressed in tripl…
View article: Data from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain
Data from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain Open
Purpose: Basal-like breast tumors are typically (ER/PR/HER2) triple-negative and are associated with a high incidence of brain metastases and poor clinical outcomes. The molecular chaperone αB-crystallin is predominantly expressed in tripl…
View article: Supplementary Figures 1 - 5, Tables 1 - 4 from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain
Supplementary Figures 1 - 5, Tables 1 - 4 from αB-Crystallin: A Novel Regulator of Breast Cancer Metastasis to the Brain Open
PDF file - 797K, Figure S1. Barrier phenotype of human brain microvascular endothelial cells and astrocytes in monocultures and co-cultures. Figure S2. alpha/beta-crystallin does not affect cell viability of TNBC cells in standard monolaye…
View article: Data from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Data from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Purpose: Many neoplasms are vulnerable to methionine deficiency by mechanisms that are poorly understood. Because gene profiling studies have revealed that methionine depletion increases TNF-related apoptosis-inducing ligand receptor-2 (TR…
View article: Supplementary Table S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Table S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Table S2. Identification of proteins differentially expressed in response to methionine deprivation.
View article: Supplementary Figure S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Figure S1 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Figure S1. Methionine restriction inhibits cell proliferation.
View article: Supplementary Figure S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Figure S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Figure S2. Methioninase sensitizes TNBC cells to lexatumumab.
View article: Supplementary methods and legends from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary methods and legends from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary methods and legends
View article: Data from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Data from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Purpose: Many neoplasms are vulnerable to methionine deficiency by mechanisms that are poorly understood. Because gene profiling studies have revealed that methionine depletion increases TNF-related apoptosis-inducing ligand receptor-2 (TR…
View article: Supplementary Table S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Table S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Table S2. Identification of proteins differentially expressed in response to methionine deprivation.
View article: Supplementary Figure S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary Figure S2 from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary Figure S2. Methioninase sensitizes TNBC cells to lexatumumab.
View article: Supplementary methods and legends from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression
Supplementary methods and legends from Methionine Deprivation Induces a Targetable Vulnerability in Triple-Negative Breast Cancer Cells by Enhancing TRAIL Receptor-2 Expression Open
Supplementary methods and legends
View article: Astrocytes promote progression of breast cancer metastases to the brain via a KISS1-mediated autophagy
Astrocytes promote progression of breast cancer metastases to the brain via a KISS1-mediated autophagy Open
Formation of metastases, also known as cancer dissemination, is an important stage of breast cancer (BrCa) development. KISS1 expression is associated with inhibition of metastases development. Recently we have demonstrated that BrCa metas…