Dylan R. Edwards
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View article: Brk/PTK6 and Involucrin Expression May Predict Breast Cancer Cell Responses to Vitamin D3
Brk/PTK6 and Involucrin Expression May Predict Breast Cancer Cell Responses to Vitamin D3 Open
The process of human embryonic mammary development gives rise to the structures in which mammary cells share a developmental lineage with skin epithelial cells such as keratinocytes. As some breast carcinomas have previously been shown to …
View article: Supplementary Figure Legends from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure Legends from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Supplementary Figure Legends
View article: Supplementary Figure S2 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure S2 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Knock-down of MMP-9 did not promote invasive growth of Panc02 pancreatic cancer cells in vitro.
View article: Supplementary Figure Legends from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure Legends from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Supplementary Figure Legends
View article: Data from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Data from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Matrix metalloproteinase 9 (MMP-9/Gelatinase B) is overexpressed in pancreatic ductal adenocarcinoma (PDAC) and plays a central role in tumor cell invasion and metastasis. Here we complemented mechanistic insights in the cancer biology of …
View article: Visual Overview from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Visual Overview from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Visual Overview
View article: Supplementary Figure S1 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure S1 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
MMP2 in KPC mice.
View article: Supplementary Figure S1 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure S1 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
MMP2 in KPC mice.
View article: Supplementary Figure S2 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Supplementary Figure S2 from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Knock-down of MMP-9 did not promote invasive growth of Panc02 pancreatic cancer cells in vitro.
View article: Data from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Data from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Matrix metalloproteinase 9 (MMP-9/Gelatinase B) is overexpressed in pancreatic ductal adenocarcinoma (PDAC) and plays a central role in tumor cell invasion and metastasis. Here we complemented mechanistic insights in the cancer biology of …
View article: Visual Overview from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow
Visual Overview from Systemic Ablation of MMP-9 Triggers Invasive Growth and Metastasis of Pancreatic Cancer via Deregulation of IL6 Expression in the Bone Marrow Open
Visual Overview
View article: Data from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Data from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
Purpose: This study investigated the functional and clinical significance of integrin αvβ6 upregulation in myoepithelial cells of ductal carcinoma in situ (DCIS).Experimental Design: Archival samples of DCIS and DCIS w…
View article: Supplementary Figure 3 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 3 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 134K, Supplementary Figure 3. Organotypic of MDA-MB-231 + beta6-1089 staining.
View article: Supplementary Table 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Table 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 55K, Supplementary Table 1. Details relating to; treatment received, patient age, hormone replacement therapy, mean follow up and pathological characteristics for cases and controls.
View article: Supplementary Figure 2 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 2 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 108K, Supplementary Figure 2. alphavbeta6 functional assays in the beta6-1089 cell line using RNAi.
View article: Supplementary Figure 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 106K, Supplementary Figure 1. Transient transfection of alphavbeta6 in primary myoepithelial cells.
View article: Supplementary Figure 3 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 3 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 134K, Supplementary Figure 3. Organotypic of MDA-MB-231 + beta6-1089 staining.
View article: Data from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Data from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
Purpose: This study investigated the functional and clinical significance of integrin αvβ6 upregulation in myoepithelial cells of ductal carcinoma in situ (DCIS).Experimental Design: Archival samples of DCIS and DCIS w…
View article: Supplementary Figure 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 106K, Supplementary Figure 1. Transient transfection of alphavbeta6 in primary myoepithelial cells.
View article: Supplementary Methods from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Methods from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 91K
View article: Supplementary Figure 2 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Figure 2 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 108K, Supplementary Figure 2. alphavbeta6 functional assays in the beta6-1089 cell line using RNAi.
View article: Supplementary Methods from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Methods from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 91K
View article: Supplementary Table 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence
Supplementary Table 1 from Altered Microenvironment Promotes Progression of Preinvasive Breast Cancer: Myoepithelial Expression of αvβ6 Integrin in DCIS Identifies High-risk Patients and Predicts Recurrence Open
PDF file - 55K, Supplementary Table 1. Details relating to; treatment received, patient age, hormone replacement therapy, mean follow up and pathological characteristics for cases and controls.
View article: Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12
Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12 Open
Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12
View article: Supplementary Figure 5 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
Supplementary Figure 5 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling Open
Supplementary Figure 5 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
View article: Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling Open
Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
View article: Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling Open
Supplementary Figure 2 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
View article: Supplementary Figure 1 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
Supplementary Figure 1 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling Open
Supplementary Figure 1 from Tissue Inhibitor of Metalloproteinases-1 Promotes Liver Metastasis by Induction of Hepatocyte Growth Factor Signaling
View article: Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12
Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12 Open
Supplementary Figure 2 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12
View article: Supplementary Figure 1 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12
Supplementary Figure 1 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12 Open
Supplementary Figure 1 from Tenascin-C Stimulates Glioma Cell Invasion through Matrix Metalloproteinase-12