Emanuele Cocucci
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View article: Garp/LRRC32 defines the erythroid-megakaryocyte fate decision and enables β-globin program activation
Garp/LRRC32 defines the erythroid-megakaryocyte fate decision and enables β-globin program activation Open
Erythroid commitment from multipotent progenitors is a tightly regulated process, yet the molecular drivers during the earliest stages, prior to erythropoietin (Epo) responsiveness, remain incompletely understood. GARP (Glycoprotein A Repe…
View article: Exploring the Spatial Limits of Extracellular Vesicles‐Mediated Intercellular Communication
Exploring the Spatial Limits of Extracellular Vesicles‐Mediated Intercellular Communication Open
Extracellular vesicles (EVs) are biological nanovectors that retain molecular signatures of their cells of origin and mediate intercellular communication, resulting in ideal platforms for the development of diagnostic tools and bio‐inspire…
View article: Genomewide Screen Identifies Peroxisomal Role in APOL1 Podocytopathy
Genomewide Screen Identifies Peroxisomal Role in APOL1 Podocytopathy Open
The G1 and G2 variants of the APOL1 gene increase the risk of chronic kidney disease (CKD) in individuals of African descent. In the presence of secondary stressors such as inflammation and hypoxia, these gain-of-function variants can indu…
View article: Nucleolin acute degradation reveals novel functions in cell cycle progression and division in TNBC
Nucleolin acute degradation reveals novel functions in cell cycle progression and division in TNBC Open
Nucleoli are large nuclear sub-compartments where vital processes, such as ribosome assembly, take place. Technical obstacles still limit our understanding of the biological functions of nucleolar proteins in cell homeostasis and cancer pa…
View article: Minimal information for studies of extracellular vesicles (MISEV2023): From basic to advanced approaches
Minimal information for studies of extracellular vesicles (MISEV2023): From basic to advanced approaches Open
Extracellular vesicles (EVs), through their complex cargo, can reflect the state of their cell of origin and change the functions and phenotypes of other cells. These features indicate strong biomarker and therapeutic potential and have ge…
View article: Editorial: Recent advances in measuring and controlling synaptic communication
Editorial: Recent advances in measuring and controlling synaptic communication Open
EDITORIAL article Front. Cell. Neurosci., 21 September 2023Sec. Cellular Neurophysiology Volume 17 - 2023 | https://doi.org/10.3389/fncel.2023.1289874
View article: Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
View article: Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
View article: Data from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Data from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Multiple myeloma cells aberrantly express surface antigens compared with normal plasma cells. Among others, CD56 is present at variable levels in approximately 70% of patients with multiple myeloma; however, very little is known about CD56…
View article: Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
View article: Data from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Data from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Multiple myeloma cells aberrantly express surface antigens compared with normal plasma cells. Among others, CD56 is present at variable levels in approximately 70% of patients with multiple myeloma; however, very little is known about CD56…
View article: Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma Open
Supplementary Figure from Redefining CD56 as a Biomarker and Therapeutic Target in Multiple Myeloma
View article: Supplementary Figure 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on Akt1 in circulating EVs
View article: Supplementary Table 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Table 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Supplementary Table 2: Master regulator analysis (MRA), using TF-target interaction information collected from public databases, including the Biomolecular Interaction Network database (BIND), EdgeExpress database (EEDB), Transcriptional R…
View article: Supplementary Figure 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on Akt1 in circulating EVs
View article: Supplementary Figure 4 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 4 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on Akt1 activation by LO
View article: Supplementary Figure 3 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 3 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on MYC activation by LO
View article: Supplementary Figure 4 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 4 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on Akt1 activation by LO
View article: Supplementary Figure 3 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 3 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on MYC activation by LO
View article: Supplementary Figure 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on LO internalization
View article: Supplementary Table 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Table 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Supplementary Table 1: RT-qPCR primers sequences.
View article: Supplementary Table 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Table 1 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Supplementary Table 1: RT-qPCR primers sequences.
View article: Data from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Data from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Communication between cancer cells and the tumor microenvironment results in the modulation of complex signaling networks that facilitate tumor progression. Here, we describe a new mechanism of intercellular communication originating from …
View article: Supplementary Figure 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Figure 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional data on LO internalization
View article: Supplementary Table 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplementary Table 2 from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Supplementary Table 2: Master regulator analysis (MRA), using TF-target interaction information collected from public databases, including the Biomolecular Interaction Network database (BIND), EdgeExpress database (EEDB), Transcriptional R…
View article: Data from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Data from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Communication between cancer cells and the tumor microenvironment results in the modulation of complex signaling networks that facilitate tumor progression. Here, we describe a new mechanism of intercellular communication originating from …
View article: Supplemental Methods from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer
Supplemental Methods from MYC Mediates Large Oncosome-Induced Fibroblast Reprogramming in Prostate Cancer Open
Additional Experimental Procedure Information