Emmanuelle Viré
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View article: Characterisation and prion transmission study in mice with genetic reduction of sporadic Creutzfeldt-Jakob disease risk gene Stx6
Characterisation and prion transmission study in mice with genetic reduction of sporadic Creutzfeldt-Jakob disease risk gene Stx6 Open
View article: Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Data from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Data from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Tumors can escape from immunity by repressing leukocyte adhesion molecule expression on tumor endothelial cells and by rendering endothelial cells unresponsive to inflammatory activation. This endothelial cell anergy is induced by angiogen…
View article: Data from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Data from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Tumor angiogenesis requires intricate regulation of gene expression in endothelial cells. We recently showed that DNA methyltransferase (DNMT) and histone deacetylase (HDAC) inhibitors directly repress endothelial cell growth and tumor ang…
View article: Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
View article: Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Supplementary Tables 1-3 from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
View article: Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 3 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
View article: Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 2 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Data from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Data from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Tumor angiogenesis requires intricate regulation of gene expression in endothelial cells. We recently showed that DNA methyltransferase (DNMT) and histone deacetylase (HDAC) inhibitors directly repress endothelial cell growth and tumor ang…
View article: Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Data 1 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Methods from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Data from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Data from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Tumors can escape from immunity by repressing leukocyte adhesion molecule expression on tumor endothelial cells and by rendering endothelial cells unresponsive to inflammatory activation. This endothelial cell anergy is induced by angiogen…
View article: Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 4 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells Open
Supplementary Table 5 from Identification of Epigenetically Silenced Genes in Tumor Endothelial Cells
View article: Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications Open
Supplementary Materials and Methods from Epigenetic Regulation of Tumor Endothelial Cell Anergy: Silencing of Intercellular Adhesion Molecule-1 by Histone Modifications
View article: Characterisation and prion transmission study in mice with genetic reduction of sporadic Creutzfeldt-Jakob Disease risk gene<i>Stx6</i>
Characterisation and prion transmission study in mice with genetic reduction of sporadic Creutzfeldt-Jakob Disease risk gene<i>Stx6</i> Open
Sporadic Creutzfeldt-Jakob disease (sCJD), the most common human prion disease, is thought to occur when the cellular prion protein (PrP C ) spontaneously misfolds and assembles into prion fibrils, culminating in fatal neurodegeneration. I…
View article: Epigenetic age acceleration is associated with oligodendrocyte proportions in MSA and control brain tissue
Epigenetic age acceleration is associated with oligodendrocyte proportions in MSA and control brain tissue Open
Aims Epigenetic clocks are widely applied as surrogates for biological age in different tissues and/or diseases, including several neurodegenerative diseases. Despite white matter (WM) changes often being observed in neurodegenerative dise…
View article: DNA methylation analysis of archival lymphoreticular tissues in Creutzfeldt–Jakob disease
DNA methylation analysis of archival lymphoreticular tissues in Creutzfeldt–Jakob disease Open
View article: Prion protein gene mutation detection using long-read Nanopore sequencing
Prion protein gene mutation detection using long-read Nanopore sequencing Open
View article: Kroll, Dimitriadis, et al., 2022. Prion protein gene mutation detection using long-read Nanopore sequencing
Kroll, Dimitriadis, et al., 2022. Prion protein gene mutation detection using long-read Nanopore sequencing Open
Version uploaded 17/04/2022. Data for François Kroll*, Athanasios Dimitriadis*, Tracy Campbell, Lee Darwent, John Collinge, Simon Mead, Emmanuelle Viré. 2022. *co-first authors Prion protein gene mutation detectio…
View article: Kroll et al., 2022. Prion protein gene mutation detection using long-read Nanopore sequencing
Kroll et al., 2022. Prion protein gene mutation detection using long-read Nanopore sequencing Open
Version uploaded 09/04/2022. Data and code for Kroll, F. et al. 2022. Prion protein gene mutation detection using long-read Nanopore sequencing. https://doi.org/10.1101/2022.03.06.22271294 Present Zenodo archive contains heavier d…
View article: Gene expression and epigenetic markers of prion diseases
Gene expression and epigenetic markers of prion diseases Open