Ericka Randazzo
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View article: Rare Diseases That Impersonate One Another: <scp>X‐Linked</scp> Hypophosphatemia and <scp>Tumor‐Induced</scp> Osteomalacia, a Retrospective Analysis of Discriminating Features
Rare Diseases That Impersonate One Another: <span>X‐Linked</span> Hypophosphatemia and <span>Tumor‐Induced</span> Osteomalacia, a Retrospective Analysis of Discriminating Features Open
Tumor‐induced osteomalacia (TIO) is a rare paraneoplastic disease characterized by frequent fractures, bone pain, muscle weakness, and affected gait. The rarity of TIO and similar presentation to other phosphate‐wasting disorders contribut…
View article: EPEN-05. MUTATIONAL ANALYSIS OF THE C11ORF95 DOMAIN AND SINGLE-CELL RNA-SEQ PROFILE OF A MOUSE MODEL OF SUPRATENTORIAL EPENDYMOMA
EPEN-05. MUTATIONAL ANALYSIS OF THE C11ORF95 DOMAIN AND SINGLE-CELL RNA-SEQ PROFILE OF A MOUSE MODEL OF SUPRATENTORIAL EPENDYMOMA Open
We used a recently developed mouse model to better understand the cellular and molecular determinants of tumors driven by the oncogenic fusion protein C11orf95-RELA. Our approach makes use of in utero electroporation and a binary transposa…
View article: TMOD-32. MUTATIONAL ANALYSIS OF THE C11orf95 DOMAIN AND SINGLE CELL RNASEQ PROFILE OF A MOUSE MODEL OF SUPRATENTORIAL EPENDYMOMA
TMOD-32. MUTATIONAL ANALYSIS OF THE C11orf95 DOMAIN AND SINGLE CELL RNASEQ PROFILE OF A MOUSE MODEL OF SUPRATENTORIAL EPENDYMOMA Open
We used a recently developed mouse model to better understand the cellular and molecular determinants of tumors driven by the oncogenic fusion protein C11orf95-RELA. Our approach makes use of in utero electroporation and a binary transposa…
View article: NF-κB Subunits p50 and RelA Contribute to Oncogenesis in a Mouse Model of ST-EPN
NF-κB Subunits p50 and RelA Contribute to Oncogenesis in a Mouse Model of ST-EPN Open
The NF- κB pathway has been demonstrated to be upregulated in supratentorial ependymoma (ST-EPN). Whether the different NF- κB transcription factors contribute to ST-EPN tumorigenesis however, has not been investigated. We assessed the deg…
View article: PDTM-29. STRUCTURE FUNCTION AND CELL TYPE DETERMINANTS OF C11orf95-RELA DRIVEN TUMORS IN MICE
PDTM-29. STRUCTURE FUNCTION AND CELL TYPE DETERMINANTS OF C11orf95-RELA DRIVEN TUMORS IN MICE Open
We used a new mouse model to better understand the cellular and molecular determinants of tumors driven by the C11orf95-RELA fusion. Our approach makes use of in utero electroporation and a binary transposase system to introduce human C11o…
View article: Notch Inhibitors and the BET Inhibitor JQ-1 Decrease the Growth of Primary Tumor Cells Derived from a Novel Mouse Model of C11orf95-RELA Induced Brain Tumor
Notch Inhibitors and the BET Inhibitor JQ-1 Decrease the Growth of Primary Tumor Cells Derived from a Novel Mouse Model of C11orf95-RELA Induced Brain Tumor Open
Brain tumors are the most common childhood solid malignancy, and because of remarkable advances in treating many cancers outside of the brain, they have become the leading cause of cancer mortality in children. Ependymomas are a class of b…