Eunkyung Park
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View article: Thermal Fault-Tolerant Asymmetric Dual-Winding Motors in Integrated Electric Braking System for Autonomous Vehicles
Thermal Fault-Tolerant Asymmetric Dual-Winding Motors in Integrated Electric Braking System for Autonomous Vehicles Open
A conventional dual-winding (DW) motor has two internal windings consisting of a master part and a slave part, each connected to a different electronic control unit (ECU) to realize a redundant system. However, existing DW motors have a pr…
View article: Thermal runaway prevention through scalable fabrication of safety reinforced layer in practical Li-ion batteries
Thermal runaway prevention through scalable fabrication of safety reinforced layer in practical Li-ion batteries Open
View article: Developing a Data Visualization Program for Transfusion Appropriateness Management
Developing a Data Visualization Program for Transfusion Appropriateness Management Open
View article: Figure S8 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S8 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Interaction of Prox1 and RTA was not detected in K-BECs
View article: Figure S3 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S3 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Productive Lytic Replication in LECs, but not in BECs
View article: Figure S5 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S5 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Lytic DNA replication is essential for persistent KSHV infection in LECs
View article: Figure S7 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S7 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Quantitation of the expressions of the viral genes and Prox1
View article: Figure S1 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S1 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Sustained KSHV maintenance and massive cell death in KSHV-infected LECs
View article: Data from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Data from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Kaposi sarcoma is the most common cancer in human immunodeficiency virus–positive individuals and is caused by Kaposi sarcoma–associated herpesvirus (KSHV). It is believed that a small number of latently infected Kaposi sarcoma tumor cells…
View article: Figure S3 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S3 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Productive Lytic Replication in LECs, but not in BECs
View article: Figure S6 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S6 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Productive KSHV Lytic Replication Is Necessary for Sustained Infection in LECs
View article: Figure S6 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus
Figure S6 from The Lymphatic Cell Environment Promotes Kaposi Sarcoma Development by Prox1-Enhanced Productive Lytic Replication of Kaposi Sarcoma Herpes Virus Open
Productive KSHV Lytic Replication Is Necessary for Sustained Infection in LECs
View article: Supplemental Figures from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Figures from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplemental Figures. Fig S1:Expression of HEY1 and HEY2 in thyroid cancers vs. normal thyroid tissues. Fig S2:PROX1 mislocalization in TPC-1 cells does not depend on CRM1, energy or temperature. Fig S3: T3 promotes PROX1 nuclear exclusion…
View article: Supplementary Table 2 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor
Supplementary Table 2 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor Open
PDF file - 65K, Regulation of the expression of various nuclear receptors by KSHV in BECs
View article: Supplemental Table1 from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Table1 from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
A concise summary of the publically available transcriptome data sets that were used to generate Figure 1 in the main text.
View article: Supplemental Figures from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Figures from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplemental Figures. Fig S1:Expression of HEY1 and HEY2 in thyroid cancers vs. normal thyroid tissues. Fig S2:PROX1 mislocalization in TPC-1 cells does not depend on CRM1, energy or temperature. Fig S3: T3 promotes PROX1 nuclear exclusion…
View article: Supplementary Figure Legends from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplementary Figure Legends from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplementary Figure Legends
View article: Supplementary Table 1 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor
Supplementary Table 1 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor Open
PDF file - 91K, Regulation of the expression of the RGS genes by KSHV in BECs
View article: Supplemental Methods from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Methods from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplemental Methods
View article: Supplementary Table 1 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor
Supplementary Table 1 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor Open
PDF file - 91K, Regulation of the expression of the RGS genes by KSHV in BECs
View article: Data from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Data from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Papillary thyroid cancer (PTC) is one of the most common endocrine malignancies associated with significant morbidity and mortality. Although multiple studies have contributed to a better understanding of the genetic alterations underlying…
View article: Supplemental Methods from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Methods from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplemental Methods
View article: Supplementary Figure Legends from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplementary Figure Legends from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Supplementary Figure Legends
View article: Supplemental Table1 from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Supplemental Table1 from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
A concise summary of the publically available transcriptome data sets that were used to generate Figure 1 in the main text.
View article: Supplementary Table 2 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor
Supplementary Table 2 from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor Open
PDF file - 65K, Regulation of the expression of various nuclear receptors by KSHV in BECs
View article: Data from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells
Data from Aberrant Activation of Notch Signaling Inhibits PROX1 Activity to Enhance the Malignant Behavior of Thyroid Cancer Cells Open
Papillary thyroid cancer (PTC) is one of the most common endocrine malignancies associated with significant morbidity and mortality. Although multiple studies have contributed to a better understanding of the genetic alterations underlying…
View article: Data from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor
Data from Lymphatic Reprogramming by Kaposi Sarcoma Herpes Virus Promotes the Oncogenic Activity of the Virus-Encoded G-protein–Coupled Receptor Open
Kaposi sarcoma, the most common cancer in HIV-positive individuals, is caused by endothelial transformation mediated by the Kaposi sarcoma herpes virus (KSHV)-encoded G-protein–coupled receptor (vGPCR). Infection of blood vascular endothel…
View article: A Study on Transmedial Storytelling on HMD-based Virtual Reality Games
A Study on Transmedial Storytelling on HMD-based Virtual Reality Games Open
본 논문의 목적은 2010년대 전반에 걸쳐 나타난 HMD 기반 가상현실 게임 콘텐츠의 변화상을 스토리텔링 관점에서 논하는 데 있다. 이에 본 논문에서는 데이비드 허먼이 제시한 테제, 안티테제, 진테제의 논의를 빌어 가상현실 게임 콘텐츠에 대한 올바른 접근법을 모색하였고, 그 중 진테제의 명제가 가상현실 게임 콘텐츠 분석에 가장 타당한 접근법이라는 결론에 이르렀다. 진테제 관점에서 볼 때, 가상현실 게임의 서사 담론의 특징은 …
View article: Subconjunctival Lymphatics Respond to VEGFC and Anti-Metabolites in Rabbit and Mouse Eyes
Subconjunctival Lymphatics Respond to VEGFC and Anti-Metabolites in Rabbit and Mouse Eyes Open
Subconjunctival lymphatics are pharmacologically responsive to both VEGFC and antimetabolites in two animal models studied using different methodologies. These results may be important for bleb-forming glaucoma surgeries or ocular drug del…
View article: Piezo1-Regulated Mechanotransduction Controls Flow-Activated Lymphatic Expansion
Piezo1-Regulated Mechanotransduction Controls Flow-Activated Lymphatic Expansion Open
Background: Mutations in PIEZO1 (Piezo type mechanosensitive ion channel component 1) cause human lymphatic malformations. We have previously uncovered an ORAI1 (ORAI calcium release-activated calcium modulator 1)-mediated mechanotransduct…