Fanyuan Zeng
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View article: 770 Immune landscape of cardiovascular mortality in primary prostate cancer patients following leuprolide treatment, a non-canonical immunotherapy
770 Immune landscape of cardiovascular mortality in primary prostate cancer patients following leuprolide treatment, a non-canonical immunotherapy Open
View article: Targeting the VIP-VPAC Pathway in Melanoma Models Inhibits Tumor Growth and Liver Metastasis
Targeting the VIP-VPAC Pathway in Melanoma Models Inhibits Tumor Growth and Liver Metastasis Open
Uveal melanoma (UVM) is resistant to immune checkpoint therapy and chemotherapy, resulting in high mortality rates, primarily due to liver metastases. While vasoactive intestinal peptide (VIP) signaling has been identified as an immune che…
View article: 732 Vasoactive intestinal peptide (VIP) increases during tumor ontology in KPC GEMM mice
732 Vasoactive intestinal peptide (VIP) increases during tumor ontology in KPC GEMM mice Open
View article: 1347 VIP Biology drive prognosis in AML patients
1347 VIP Biology drive prognosis in AML patients Open
View article: Figure S1 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S1 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S4 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S4 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S6 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S6 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Supplementary Methods and Materials from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Supplementary Methods and Materials from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Methods and Materials
View article: Data from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Data from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Pancreatic ductal adenocarcinoma (PDAC) harbors a complex tumor microenvironment, and cross-talk among cells in the tumor microenvironment can contribute to drug resistance and relapse. Vasoactive intestinal peptide (VIP) is overexpressed …
View article: Figure S3 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S3 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S2 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S2 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S1 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S1 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Supplementary Methods and Materials from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Supplementary Methods and Materials from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Methods and Materials
View article: Figure S6 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S6 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S3 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S3 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S2 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S2 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S5 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S5 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Data from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Data from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Pancreatic ductal adenocarcinoma (PDAC) harbors a complex tumor microenvironment, and cross-talk among cells in the tumor microenvironment can contribute to drug resistance and relapse. Vasoactive intestinal peptide (VIP) is overexpressed …
View article: Figure S5 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S5 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: Figure S4 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
Figure S4 from VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Supplementary Figure
View article: VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer
VPAC2 Receptor Signaling Promotes Growth and Immunosuppression in Pancreatic Cancer Open
Pancreatic ductal adenocarcinoma (PDAC) harbors a complex tumor microenvironment, and cross-talk among cells in the tumor microenvironment can contribute to drug resistance and relapse. Vasoactive intestinal peptide (VIP) is overexpressed …
View article: CT-based peritumoral radiomics nomogram on prediction of response and survival to induction chemotherapy in locoregionally advanced nasopharyngeal carcinoma
CT-based peritumoral radiomics nomogram on prediction of response and survival to induction chemotherapy in locoregionally advanced nasopharyngeal carcinoma Open
Purpose The study aims to harness the value of radiomics models combining intratumoral and peritumoral features obtained from pretreatment CT to predict treatment response as well as the survival of LA-NPC(locoregionally advanced nasophary…
View article: VPAC2 receptor signaling promotes pancreatic cancer cell growth and decreases the immunogenicity of the tumor microenvironment
VPAC2 receptor signaling promotes pancreatic cancer cell growth and decreases the immunogenicity of the tumor microenvironment Open
Identifying mechanisms underlying tumor growth and immune resistance is needed to treat pancreatic ductal adenocarcinoma (PDAC) effectively. The complexity of the tumor microenvironment (TME) suggests that the crosstalk between cells in th…
View article: A cost-effectiveness analysis of toripalimab plus TP versus TP for the first-line treatment of advanced esophageal cancer
A cost-effectiveness analysis of toripalimab plus TP versus TP for the first-line treatment of advanced esophageal cancer Open
Objective : We evaluated the cost-effectiveness of Toripalimab plus TP in treating patients with esophageal cancer in this study. Perspective : the Chinese payers. Methods : A Markov model was developed from the Chinese payers’ perspective…
View article: The interplay of circulating tumor DNA and chromatin modification, therapeutic resistance, and metastasis
The interplay of circulating tumor DNA and chromatin modification, therapeutic resistance, and metastasis Open
Peripheral circulating free DNA (cfDNA) is DNA that is detected in plasma or serum fluid with a cell-free status. For cancer patients, cfDNA not only originates from apoptotic cells but also from necrotic tumor cells and disseminated tumor…