Francesca R. Dejure
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View article: Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality
Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality Open
View article: Correction to: CIP2A regulates MYC translation (via its 5′UTR) in colorectal cancer
Correction to: CIP2A regulates MYC translation (via its 5′UTR) in colorectal cancer Open
View article: Author Correction: Reprogramming of host glutamine metabolism during Chlamydia trachomatis infection and its key role in peptidoglycan synthesis
Author Correction: Reprogramming of host glutamine metabolism during Chlamydia trachomatis infection and its key role in peptidoglycan synthesis Open
View article: Additional file 2 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality
Additional file 2 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality Open
Additional file 2.
View article: Additional file 4 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality
Additional file 4 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality Open
Additional file 4.
View article: Additional file 3 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality
Additional file 3 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality Open
Additional file 3.
View article: Additional file 1 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality
Additional file 1 of Uncovering cancer vulnerabilities by machine learning prediction of synthetic lethality Open
Additional file 1.
View article: Exploiting the metabolic dependencies of the broad amino acid transporter SLC6A14
Exploiting the metabolic dependencies of the broad amino acid transporter SLC6A14 Open
Tumor cells typically enhance their metabolic capacity to sustain their higher rate of growth and proliferation. One way to elevate the nutrient intake into cancer cells is to increase the expression of genes encoding amino acid transporte…
View article: CIP2A regulates MYC translation (via its 5′UTR) in colorectal cancer
CIP2A regulates MYC translation (via its 5′UTR) in colorectal cancer Open
Background Deregulated expression of MYC is a driver of colorectal carcinogenesis, suggesting that decreasing MYC expression may have significant therapeutic value. CIP2A is an oncogenic factor that regulates MYC expression. CIP2A is overe…
View article: Reprogramming of host glutamine metabolism during Chlamydia trachomatis infection and its key role in peptidoglycan synthesis
Reprogramming of host glutamine metabolism during Chlamydia trachomatis infection and its key role in peptidoglycan synthesis Open
View article: A central role of glutamine in <i>Chlamydia</i> infection
A central role of glutamine in <i>Chlamydia</i> infection Open
Obligate intracellular bacteria like Chlamydia trachomatis undergo a complex developmental cycle between infectious non-replicative (EBs) and non-infectious replicative (RBs) forms. EBs shortly after entering a host cell transform to RBs, …
View article: Golgi stress mediates redox imbalance and ferroptosis in human cells
Golgi stress mediates redox imbalance and ferroptosis in human cells Open
View article: Investigation of the role of MYC as a stress responsive protein
Investigation of the role of MYC as a stress responsive protein Open
The transcription factor MYC is deregulated in over 70% of all human tumors and, in its oncogenic form, plays a major role in the cancer metabolic reprogramming, promoting the uptake of nutrients in order to sustain the biosynthetic needs …
View article: The <i>MYC</i> mRNA 3′‐UTR couples RNA polymerase II function to glutamine and ribonucleotide levels
The <i>MYC</i> mRNA 3′‐UTR couples RNA polymerase II function to glutamine and ribonucleotide levels Open