Françoise Pujol
YOU?
Author Swipe
View article: Mitochondrial Ribosomal Protein MRPS15 Is a Component of Cytosolic Ribosomes and Regulates Translation in Stressed Cardiomyocytes
Mitochondrial Ribosomal Protein MRPS15 Is a Component of Cytosolic Ribosomes and Regulates Translation in Stressed Cardiomyocytes Open
Regulation of mRNA translation is a crucial step in controlling gene expression in stressed cells, impacting many pathologies, including heart ischemia. In recent years, ribosome heterogeneity has emerged as a key control mechanism driving…
View article: 15-Lipoxygenase promotes resolution of inflammation in lymphedema by controlling Treg cell function through IFN-β
15-Lipoxygenase promotes resolution of inflammation in lymphedema by controlling Treg cell function through IFN-β Open
Lymphedema (LD) is characterized by the accumulation of interstitial fluid, lipids and inflammatory cell infiltrate in the limb. Here, we find that LD tissues from women who developed LD after breast cancer exhibit an inflamed gene express…
View article: Supplemental Figure 3 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 3 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S3. Podoplanin staining of lymphatic vessels
View article: Supplemental Figure 7 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 7 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S7. Tumor growth after NSAID treatment
View article: Supplemental Figure 1 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 1 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S1. Method to quantify lymphatic vessels diameter
View article: Supplemental Figure 1 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 1 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S1. Method to quantify lymphatic vessels diameter
View article: Supplemental Figure 5 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 5 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S5. VEGF-D 5'UTR mRNA exhibits two structural forms that allow the binding of different proteins
View article: Supplemental Figure 8 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 8 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S8. Schematic representation of VEGF-D synthesis in tumor cells
View article: Supplemental Figure 7 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 7 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S7. Tumor growth after NSAID treatment
View article: Supplemental Figure 4 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 4 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S4. Lymphangiogenesis and tumor gowth are not affected by 4T1 or 67NR lentiviral transduction
View article: Supplemental Figure 6 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 6 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S6. Polysome profiling of 4T1 cells
View article: Supplementary Figure Legend from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplementary Figure Legend from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Supplementary figure legend
View article: Supplemental Figure 2 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 2 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S2. Expression of endogenous VEGF-D in mice tissues
View article: Data from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Data from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
The vascular endothelial growth factor VEGF-D promotes metastasis by inducing lymphangiogenesis and dilatation of the lymphatic vasculature, facilitating tumor cell extravasion. Here we report a novel level of control for VEGF-D expression…
View article: Supplemental Figure 8 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 8 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S8. Schematic representation of VEGF-D synthesis in tumor cells
View article: Supplemental Figure 2 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 2 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S2. Expression of endogenous VEGF-D in mice tissues
View article: Supplemental Figure 4 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 4 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S4. Lymphangiogenesis and tumor gowth are not affected by 4T1 or 67NR lentiviral transduction
View article: Supplemental Figure 6 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 6 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S6. Polysome profiling of 4T1 cells
View article: Data from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Data from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
The vascular endothelial growth factor VEGF-D promotes metastasis by inducing lymphangiogenesis and dilatation of the lymphatic vasculature, facilitating tumor cell extravasion. Here we report a novel level of control for VEGF-D expression…
View article: Supplemental Figure 5 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 5 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S5. VEGF-D 5'UTR mRNA exhibits two structural forms that allow the binding of different proteins
View article: Supplemental Figure 3 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplemental Figure 3 from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Figure S3. Podoplanin staining of lymphatic vessels
View article: Supplementary Figure Legend from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis
Supplementary Figure Legend from Nucleolin Promotes Heat Shock–Associated Translation of VEGF-D to Promote Tumor Lymphangiogenesis Open
Supplementary figure legend
View article: Apelin-VEGF-C mRNA delivery as therapeutic for the treatment of secondary lymphedema
Apelin-VEGF-C mRNA delivery as therapeutic for the treatment of secondary lymphedema Open
Secondary lymphedema (LD) corresponds to a severe lymphatic dysfunction leading to the accumulation of fluid and fibrotic adipose tissue in a limb. Here, we identified apelin (APLN) as a powerful molecule for regenerating lymphatic functio…
View article: Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia
Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia Open
Internal ribosome entry sites (IRESs) drive translation initiation during stress. In response to hypoxia, (lymph)angiogenic factors responsible for tissue revascularization in ischemic diseases are induced by the IRES-dependent mechanism. …
View article: 15-Lipoxygenase drives inflammation resolution in lymphedema by controlling PPARg+Treg cell population trafficking
15-Lipoxygenase drives inflammation resolution in lymphedema by controlling PPARg+Treg cell population trafficking Open
Lymphedema (LD) is characterized by the accumulation of protein-rich interstitial fluid, lipids and a significant inflammatory cell infiltrate in the limb. It causes a significant morbidity and is a common disabling disease affecting more …
View article: Author response: Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia
Author response: Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia Open
Article Figures and data Abstract Editor's evaluation Introduction Results Discussion Materials and methods Data availability References Decision letter Author response Article and author information Metrics Abstract Internal ribosome entr…
View article: Coordinating Effect of VEGFC and Oleic Acid Participates to Tumor Lymphangiogenesis
Coordinating Effect of VEGFC and Oleic Acid Participates to Tumor Lymphangiogenesis Open
In cancer, the lymphatic system is hijacked by tumor cells that escape from primary tumor and metastasize to the sentinel lymph nodes. Tumor lymphangiogenesis is stimulated by the vascular endothelial growth factors-C (VEGFC) after binding…
View article: Coordinating Effect of VEGFC and Oleic Acid Drives Tumor Lymphangiogenesis
Coordinating Effect of VEGFC and Oleic Acid Drives Tumor Lymphangiogenesis Open
In cancer, the lymphatic system is hijacked by tumor cells to escape from primary tumor and to metastasize to the sentinel lymph nodes. Tumor lymphangiogenesis is stimulated by the vascular endothelial growth factors-C (VEGFC) after bindin…
View article: Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia
Long non-coding RNA Neat1 and paraspeckle components are translational regulators in hypoxia Open
SUMMARY Internal ribosome entry sites (IRESs) drive translation initiation during stress. In response to hypoxia, (lymph)angiogenic factors responsible for tissue revascularization in ischemic diseases are induced by the IRES-dependent mec…