Frédéric Vigant YOU? Author Swipe

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Supplementary Table 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Table 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Supplementary Figure 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Figure 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Data from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

We have reported previously that the interaction of α_E(CD103)β₇ integrin, expressed on a CD8⁺ tumor-infiltrating lymphocyte (TIL) clone but not on a peripheral blood lymphocyte (PBL) counterpart, with the e…

Supplementary Figure 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Figure 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Supplementary Table 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Table 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Supplementary Table 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Table 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Data from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

We have reported previously that the interaction of α_E(CD103)β₇ integrin, expressed on a CD8⁺ tumor-infiltrating lymphocyte (TIL) clone but not on a peripheral blood lymphocyte (PBL) counterpart, with the e…

Supplementary Figure 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Figure 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Supplementary Figure 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Figure 2 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Supplementary Table 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention Open

Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert , et al. · 2023

Supplementary Table 1 from Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention

Efficient and Robust <i>Paramyxoviridae</i> Reverse Genetics Systems Open

Shannon M. Beaty, Arnold Park, Sohui T. Won, Patrick Hong, Michael J. Lyons , et al. · 2017

The ability to manipulate the genome of paramyxoviruses and evaluate the effects of these changes at the phenotypic level is a powerful tool for the investigation of specific aspects of the viral life cycle and viral pathogenesis. However,…

Inhibition of an Aquatic Rhabdovirus Demonstrates Promise of a Broad-Spectrum Antiviral for Use in Aquaculture Open

Bethany F. Balmer, Rachel L. Powers, Ting-Hu Zhang, Jihye Lee, Frédéric Vigant , et al. · 2016

Many enveloped viruses cause devastating disease in aquaculture, resulting in significant economic impact. LJ001 is a broad-spectrum antiviral compound that inhibits enveloped virus infections by specifically targeting phospholipids in the…

Cross-reactive and cross-neutralizing activity of human mumps antibodies against a novel mumps virus from bats Open

Shannon M. Beaty, Raffael Nachbagauer, Ariana Hirsh, Frédéric Vigant, James Duehr , et al. · 2016

To evaluate the antigenic relationship between bat mumps virus (BMV) and the JL5 vaccine strain of mumps virus (MuVJL5), we rescued a chimeric virus bearing the F and HN glycoproteins of BMV in the background of a recombinant JL5 genome (r…

The Microtubule Inhibitor Podofilox Inhibits an Early Entry Step of Human Cytomegalovirus Open

Tobias Cohen, Toni M. Schwarz, Frédéric Vigant, Thomas J. Gardner, Rosmel E. Hernandez , et al. · 2016

Human cytomegalovirus is a ubiquitous β-herpesvirus that infects many different cell types through an initial binding to cell surface receptors followed by a fusion event at the cell membrane or endocytic vesicle. A recent high-throughput …

NiV-C has a direct interaction with Tsg101. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

(A) HA-tagged NiV-C co-immunoprecipitates endogenous Tsg101 from 293T cell lysates, as does HA-tagged Vps28, a known cellular interacting partner of Tsg101. (B) NiV-C was truncated as indicated, with all constructs retaining …

NiV-C interacts with NiV-M to promote budding. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

(A) NiV-C-HA and NiV-M were transfected as indicated in 293T cells, and lysates were immunoprecipitated with anti-NiV-M as described in Materials and Methods. NiV-C was detected with anti-HA. (B) NiV-C-HA and NiV-M were trans…

The C proteins of other known henipaviruses also enhance the budding of their cognate matrix proteins. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

(A) Phylogeny (based on the relatively conserved nucleocapsid protein, neighbor-joining tree from Clustal Omega, visualized in FigTree) for the henipaviruses as well as the “henipa-like” Mojiang virus (MojPV) and representative para…

Nipah virus budding is ESCRT-dependent. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Olivier Pernet, T. Won Sohui , et al. · 2016

(A) Wild-type (WT) and dominant-negative (DN) Vps4A-inducible 293 cells were infected with Gaussia luciferase-expressing NiV at a multiplicity of infection (MOI) of 2. Cells were induced with doxycycline (dox) post-infection,…

Model of NiV-C interactions with ESCRT. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

Vps28 acts as an adaptor between ESCRT-I and ESCRT-II. The NTD of Vps28 interacts with ESCRT-I via Tsg101-CTD, while the CTD of Vps28 interacts with ESCRT-II possibly via hVps36. The CTD of NiV-C also interacts with Tsg101-CTD, while the p…

Tsg101 is required for the NiV-C enhancement of budding and efficient release of live NiV. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

(A) NiV-C was minimally truncated at its C-terminus as indicated, with all constructs retaining a C-terminal HA tag. (B) Truncation of only 7 residues (159tr) from NiV-C results in loss of interaction with Tsg101. (C) …

NiV-M budding is enhanced by NiV-C in an ESCRT-dependent manner. Open

Arnold Park, Yun Tatyana, Frédéric Vigant, Pernet Olivier, T. Won Sohui , et al. · 2016

(A) A schematic of the NiV genome is shown, with the NiV genes arrayed between the genomic termini, the 3’ leader (3’le) and 5’ trailer (5’tr). NiV-V and -W are produced by the stochastic inclusion of additional G nucleotides in the…

Nipah Virus C Protein Recruits Tsg101 to Promote the Efficient Release of Virus in an ESCRT-Dependent Pathway Open

Arnold Park, Tatyana Yun, Frédéric Vigant, Olivier Pernet, Sohui T. Won , et al. · 2016

The budding of Nipah virus, a deadly member of the Henipavirus genus within the Paramyxoviridae, has been thought to be independent of the host ESCRT pathway, which is critical for the budding of many enveloped viruses. This conclusion was…

ISG15 deficiency and increased viral resistance in humans but not mice Open

Scott D. Speer, Zhi Li, Sofija Buta, Béatrice Payelle‐Brogard, Qian Li , et al. · 2016

Pseudotyping Serotype 5 Adenovirus with the Fiber from Other Serotypes Uncovers a Key Role of the Fiber Protein in Adenovirus 5-Induced Thrombocytopenia Open

Najat Raddi, Frédéric Vigant, Orianne Wagner‐Ballon, Stéphane Giraudier, Jerome Custers , et al. · 2016

Adenovirus (Ad) infection in humans is associated with inflammatory responses and thrombocytopenia. Although several studies were conducted in mice models to understand molecular and cellular mechanisms of Ad-induced inflammatory responses…

Sendai virus, an RNA virus with no risk of genomic integration, delivers CRISPR/Cas9 for efficient gene editing Open

Arnold Park, Patrick Hong, Sohui T. Won, Patricia A. Thibault, Frédéric Vigant , et al. · 2016

Broad-spectrum antivirals against viral fusion Open

Frédéric Vigant, Nuno C. Santos, Benhur Lee · 2015

Hexon Modification to Improve the Activity of Oncolytic Adenovirus Vectors against Neoplastic and Stromal Cells in Pancreatic Cancer Open

Tanja Lucas, Karim Benihoud, Frédéric Vigant, Christoph Q. Schmidt, Christoph Q. Schmidt , et al. · 2015

Primary pancreatic carcinoma has an unfavourable prognosis and standard treatment strategies mostly fail in advanced cases. Virotherapy might overcome this resistance to current treatment modalities. However, data from clinical studies wit…

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