Fumitaka Kamachi
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View article: Supplementary Figure S3 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S3 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S3. TLR2 specific response by LTA.
View article: Supplementary Figure S5 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S5 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S5. DCA and LTA promote obesity-associated HCC.
View article: Supplementary Figure S9 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S9 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S9. EP4 antagonist inhibit the growth of HFD-induced liver cancer.
View article: Supplementary Figure S8 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S8 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S8. The analysis of cells in the liver of DMBA-treated, HFD-fed mice.
View article: Supplementary Figure S4 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S4 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S4. DCA and LTA promote obesity-associated HCC.
View article: Supplementary Figure S5 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S5 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S5. DCA and LTA promote obesity-associated HCC.
View article: Supplementary Figure S2 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S2 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S2. Detection of cellular senescence in hepatic stellate cells.
View article: Supplementary Figure S2 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S2 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S2. Detection of cellular senescence in hepatic stellate cells.
View article: Supplementary Figure S9 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S9 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S9. EP4 antagonist inhibit the growth of HFD-induced liver cancer.
View article: Supplementary Figure S3 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity
Supplementary Figure S3 from Gut Microbiota Promotes Obesity-Associated Liver Cancer through PGE<sub>2</sub>-Mediated Suppression of Antitumor Immunity Open
Supplementary Figure S3. TLR2 specific response by LTA.
View article: Downregulation of cytoplasmic DNases is implicated in cytoplasmic DNA accumulation and SASP in senescent cells
Downregulation of cytoplasmic DNases is implicated in cytoplasmic DNA accumulation and SASP in senescent cells Open
Accumulating evidence indicates that the senescence-associated secretory phenotype (SASP) contributes to many aspects of physiology and disease. Thus, controlling the SASP will have tremendous impacts on our health. However, our understand…