George D. Maynard
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View article: Nogo receptor decoy promotes recovery and corticospinal growth in non-human primate spinal cord injury
Nogo receptor decoy promotes recovery and corticospinal growth in non-human primate spinal cord injury Open
After CNS trauma such as spinal cord injury, the ability of surviving neural elements to sprout axons, reorganize neural networks and support recovery of function is severely restricted, contributing to chronic neurological deficits. Among…
View article: Rescue of Transgenic Alzheimer’s Pathophysiology by Polymeric Cellular Prion Protein Antagonists
Rescue of Transgenic Alzheimer’s Pathophysiology by Polymeric Cellular Prion Protein Antagonists Open
(Cell Reports 26, 145–158.e1–e8; January 2, 2019) In the originally published version of this article, author name Zahra K. Naderi was misspelled in the author list. This has now been corrected. The authors regret this error. Rescue of Tra…
View article: Rescue of Transgenic Alzheimer’s Pathophysiology by Polymeric Cellular Prion Protein Antagonists
Rescue of Transgenic Alzheimer’s Pathophysiology by Polymeric Cellular Prion Protein Antagonists Open
Cellular prion protein (PrPC) binds the scrapie conformation of PrP (PrPSc) and oligomeric β-amyloid peptide (Aβo) to mediate transmissible spongiform encephalopathy (TSE) and Alzheimer's disease (AD), respectively. W…
View article: Oro-gastric (og) administration of NGD CRF<sub>1</sub> antagonist, NGD 98-2 or NGD 9002 reduces acute water avoidance stress (WAS)-induced stimulation of colonic propulsive motor function in rats.
Oro-gastric (og) administration of NGD CRF<sub>1</sub> antagonist, NGD 98-2 or NGD 9002 reduces acute water avoidance stress (WAS)-induced stimulation of colonic propulsive motor function in rats. Open
Vehicle or NGD 98-2 (A) or NGD 9002 (B) at 3, 10 and 30 mg/kg was administered po and 180 min later (for NGD 98-2 group) or 60 min later (for NGD 9002 group), rats were exposed to WAS for 60 min. FPO was monitored during the 60 min stress …