Hugues Ripoche
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View article: Data from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Data from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Purpose: Neurofibromatosis type 1 (NF1) is an autosomal dominant disorder with a complex variety of clinical manifestations. The hallmark of NF1 is the onset of heterogeneous (dermal or plexiform) benign neurofibromas. Plexiform neurofibro…
View article: Supplementary Tables 1-4 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Tables 1-4 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Tables 1-4 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Supplementary Figure Legends 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Figure Legends 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Figure Legends 1-3 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Supplementary Figures 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Figures 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Figures 1-3 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Supplementary Figures 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Figures 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Figures 1-3 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Data from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Data from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Purpose: Neurofibromatosis type 1 (NF1) is an autosomal dominant disorder with a complex variety of clinical manifestations. The hallmark of NF1 is the onset of heterogeneous (dermal or plexiform) benign neurofibromas. Plexiform neurofibro…
View article: Supplementary Tables 1-4 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Tables 1-4 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Tables 1-4 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Supplementary Figure Legends 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
Supplementary Figure Legends 1-3 from Microarray-Based Identification of <i>Tenascin C</i> and <i>Tenascin XB</i>, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1 Open
Supplementary Figure Legends 1-3 from Microarray-Based Identification of Tenascin C and Tenascin XB, Genes Possibly Involved in Tumorigenesis Associated with Neurofibromatosis Type 1
View article: Data from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Data from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Non–small cell lung cancer (NSCLC) with activating mutations in the epidermal growth factor receptor (EGFR) responds to EGFR tyrosine kinase inhibitors such as erlotinib. However, secondary somatic EGFR mutations (e.g., T790M) confer resis…
View article: Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Movie 1 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Data from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Data from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
MicroRNAs (miRNA) are noncoding RNAs that regulate multiple cellular processes, including proliferation and apoptosis. We used microarray technology to identify miRNAs that were upregulated by non–small cell lung cancer (NSCLC) A549 cells …
View article: Data from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Data from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
MicroRNAs (miRNA) are noncoding RNAs that regulate multiple cellular processes, including proliferation and apoptosis. We used microarray technology to identify miRNAs that were upregulated by non–small cell lung cancer (NSCLC) A549 cells …
View article: Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
View article: Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
View article: Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Data from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Data from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Non–small cell lung cancer (NSCLC) with activating mutations in the epidermal growth factor receptor (EGFR) responds to EGFR tyrosine kinase inhibitors such as erlotinib. However, secondary somatic EGFR mutations (e.g., T790M) confer resis…
View article: Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Figures 1-6 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Tables 1-4 from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Supplementary Table 1 from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
View article: Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib Open
Supplementary Methods and Materials from A Novel Epidermal Growth Factor Receptor Inhibitor Promotes Apoptosis in Non–Small Cell Lung Cancer Cells Resistant to Erlotinib
View article: Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death Open
Supplementary Figure, Table and Movie Legends from miR-181a and miR-630 Regulate Cisplatin-Induced Cancer Cell Death
View article: A multi-objective based clustering for inferring BCR clonal lineages from high-throughput B cell repertoire data
A multi-objective based clustering for inferring BCR clonal lineages from high-throughput B cell repertoire data Open
The adaptive B cell response is driven by the expansion, somatic hypermutation, and selection of B cell clonal lineages. A high number of clonal lineages in a B cell population indicates a highly diverse repertoire, while clonal size distr…
View article: Comparative Transcriptomics Highlights New Features of the Iron Starvation Response in the Human Pathogen Candida glabrata
Comparative Transcriptomics Highlights New Features of the Iron Starvation Response in the Human Pathogen Candida glabrata Open
In this work, we used comparative transcriptomics to identify regulatory outliers (ROs) in the human pathogen Candida glabrata. ROs are genes that have very different expression patterns compared to their orthologs in other species. From c…
View article: JET2 Viewer: a database of predicted multiple, possibly overlapping, protein–protein interaction sites for PDB structures
JET2 Viewer: a database of predicted multiple, possibly overlapping, protein–protein interaction sites for PDB structures Open
The database JET2 Viewer, openly accessible at http://www.jet2viewer.upmc.fr/, reports putative protein binding sites for all three-dimensional (3D) structures available in the Protein Data Bank (PDB). This knowledge base was generated by …