Jeff Wyckoff
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View article: Supplementary Figure S4 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S4 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S4. Deletion of LERER in MenaINV affects tumor growth in a xenograft model.
View article: Supplementary Figure S3 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S3 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S3. TCGA analysis supplementary analysis and characterization of MenaINV-specific monoclonal antibody.
View article: Supplementary Figure S3 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S3 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S3. TCGA analysis supplementary analysis and characterization of MenaINV-specific monoclonal antibody.
View article: Supplementary Figure S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S1. Effects on haptotaxis are independent of effects on cell velocity.
View article: Supplementary Figure S5 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S5 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S5. Mena isoform expression does not affect Integrin alphav, beta1 or FAK levels.
View article: Data from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Data from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Fibronectin (FN) is a major component of the tumor microenvironment, but its role in promoting metastasis is incompletely understood. Here, we show that FN gradients elicit directional movement of breast cancer cells, in vitro and in vivo.…
View article: Supplementary Figure S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S1. Effects on haptotaxis are independent of effects on cell velocity.
View article: Data from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Data from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Fibronectin (FN) is a major component of the tumor microenvironment, but its role in promoting metastasis is incompletely understood. Here, we show that FN gradients elicit directional movement of breast cancer cells, in vitro and in vivo.…
View article: Supplementary Figure S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S2. MenaINV drives haptotaxis at high FN concentrations.
View article: Supplementary Figure S4 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S4 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S4. Deletion of LERER in MenaINV affects tumor growth in a xenograft model.
View article: Supplementary Figure Legends from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure Legends from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure Legends
View article: Supplementary Figure S6 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S6 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S6. MenaINV-driven ECM reorganization in vitro and in vivo.
View article: Supplementary Video S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Video S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Video S1. MenaINV drives haptotaxis in vivo.
View article: Supplementary Figure S6 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S6 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S6. MenaINV-driven ECM reorganization in vitro and in vivo.
View article: Supplementary Video S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Video S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Video S2. MenaINV drives haptotaxis via its interaction with alpha5beta1 in vivo.
View article: Supplementary Figure Legends from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure Legends from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure Legends
View article: Supplementary Video S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Video S1 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Video S1. MenaINV drives haptotaxis in vivo.
View article: Supplementary Figure S5 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S5 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S5. Mena isoform expression does not affect Integrin alphav, beta1 or FAK levels.
View article: Supplementary Figure S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Figure S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Figure S2. MenaINV drives haptotaxis at high FN concentrations.
View article: Supplementary Video S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Supplementary Video S2 from Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Supplementary Video S2. MenaINV drives haptotaxis via its interaction with alpha5beta1 in vivo.
View article: Tumor Cell-Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Tumor Cell-Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Fibronectin (FN) is a major component of the tumor microenvironment, but its role in promoting metastasis is incompletely understood. Here we show that FN gradients elicit directional movement of breast cancer cells, in vitro and in vivo. …
View article: Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression
Tumor Cell–Driven Extracellular Matrix Remodeling Drives Haptotaxis during Metastatic Progression Open
Fibronectin (FN) is a major component of the tumor microenvironment, but its role in promoting metastasis is incompletely understood. Here, we show that FN gradients elicit directional movement of breast cancer cells, in vitro and in vivo.…