Jérèmy Willekens
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View article: Supplementary Table 1 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia
Supplementary Table 1 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia Open
Timing of CSF collection relative to Leukemia Treatment Phases for patients treated on DFCI protocol 16-001
View article: Supplementary Figure 2 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia
Supplementary Figure 2 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia Open
Linear regression between CSF 8OHdG and CSF tau at each of five timepoints.
View article: Supplementary Figure 1 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia
Supplementary Figure 1 from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia Open
Exploratory analysis of the effect of Cranial Radiation on CSF 8OHdG among patients on DFCI 16-001 and 11-001.
View article: Data from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia
Data from Treatment-Related Changes in Cerebrospinal Fluid Markers of Oxidative Stress and Neurodegeneration during Therapy for Childhood Acute Lymphoblastic Leukemia Open
Background:Survivors of childhood acute lymphoblastic leukemia (ALL) frequently exhibit treatment-related neurocognitive impairment, although there is substantial interpatient variability in this outcome. Analysis of biomarkers that reflec…
View article: The cytoplasmic domains of the CNNM family of transmembrane proteins modulate the ion channel-kinase TRPM7
The cytoplasmic domains of the CNNM family of transmembrane proteins modulate the ion channel-kinase TRPM7 Open
View article: Doxorubicin causes cognitive impairment and alters gut microbiota in both male and female juvenile rats
Doxorubicin causes cognitive impairment and alters gut microbiota in both male and female juvenile rats Open
Many survivors of childhood cancer experience significant side effects leading to inferior quality of life like chemotherapy-induced cognitive impairment (CICI). A growing body of literature implies that the gut-brain axis modulates neurol…
View article: Cross‐Talk between miRNAs from the <i>Dlk1‐Dio3</i> Locus and Histone Methylation to Protect Male Cerebellum from Methyl Donor Deficiency
Cross‐Talk between miRNAs from the <i>Dlk1‐Dio3</i> Locus and Histone Methylation to Protect Male Cerebellum from Methyl Donor Deficiency Open
Scope Disruption of the one carbon metabolism during development, i.e., following a gestational vitamin B9 and B12 deficiencies, is involved in birth defects and brain development delay. Using a rat nutritional model, consisting of pups bo…
View article: Impact of Zinc Transport Mechanisms on Embryonic and Brain Development
Impact of Zinc Transport Mechanisms on Embryonic and Brain Development Open
The trace element zinc (Zn) binds to over ten percent of proteins in eukaryotic cells. Zn flexible chemistry allows it to regulate the activity of hundreds of enzymes and influence scores of metabolic processes in cells throughout the body…
View article: Early methyl donor deficiency in cerebellum : molecular and epigenetic mechanisms
Early methyl donor deficiency in cerebellum : molecular and epigenetic mechanisms Open
View article: Carence précoce en donneurs de méthyles dans le cervelet : mécanismes moléculaires et épigénétiques
Carence précoce en donneurs de méthyles dans le cervelet : mécanismes moléculaires et épigénétiques Open
Early methyl-donor deficiencies (e.g. B9 and B12 vitamins) can lead to congenital disabilities. They are behind developmental abnormalities of the brain, and are associated with the development of neurological and neurodegenerative disease…