Jesvin Samuel
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View article: Figure S2 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S2 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 2. p53 phosphorylation.
View article: Legends from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Legends from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Legends for supplementary figures
View article: Figure S3 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S3 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 3. BTK and apoptosis.
View article: Figure S1 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S1 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 1.p53 expression and phosphorylation.
View article: Legends from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Legends from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Legends for supplementary figures
View article: Figure S3 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S3 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 3. BTK and apoptosis.
View article: Figure S2 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S2 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 2. p53 phosphorylation.
View article: Data from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Data from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
p53 is a tumor suppressor that prevents the emergence of transformed cells by inducing apoptosis or senescence, among other responses. Its functions are regulated tightly by posttranslational modifications. Here we show that Bruton's tyros…
View article: Data from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Data from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
p53 is a tumor suppressor that prevents the emergence of transformed cells by inducing apoptosis or senescence, among other responses. Its functions are regulated tightly by posttranslational modifications. Here we show that Bruton's tyros…
View article: Figure S1 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
Figure S1 from BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
Supplementary Figure 1.p53 expression and phosphorylation.
View article: Pro‐survival signal inhibition by <scp>CDK</scp> inhibitor dinaciclib in Chronic Lymphocytic Leukaemia
Pro‐survival signal inhibition by <span>CDK</span> inhibitor dinaciclib in Chronic Lymphocytic Leukaemia Open
Summary Dinaciclib is a cyclin‐dependent kinase inhibitor with clinical potential in different cancers, including chronic lymphocytic leukaemia ( CLL ). In order to better understand its cytotoxic action, we characterized its effects on si…
View article: BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses
BTK Modulates p53 Activity to Enhance Apoptotic and Senescent Responses Open
p53 is a tumor suppressor that prevents the emergence of transformed cells by inducing apoptosis or senescence, among other responses. Its functions are regulated tightly by posttranslational modifications. Here we show that Bruton's tyros…
View article: Translational studies in B-cell malignancies : studies on TP53 and BRAF
Translational studies in B-cell malignancies : studies on TP53 and BRAF Open
This thesis contains two distinct parts:\nCurrent models of Chronic Lymphocytic Leukaemia (CLL) pathogenesis invoke\nspecialised anatomical microenvironments that harbour proliferating cells. Such\nproliferating CLL cells are more resistan…