Dawei Wang
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View article: Supplementary Table 2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of significantly altered transcripts from RNA Seq analysis
View article: Supplementary Data Legends from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Data Legends from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Supplementary Figures 1 -7 and Supplementary Tables 1 - 3 legends
View article: Fig. S6 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S6 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S6 shows ingenuity pathway analysis for canonical pathways and upstream regulators
View article: Fig. S4 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S4 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S4 shows transcript levels of Col1a1, western blots for Desmin, a-SMA and b-tubulin, Transcript levels of TGFb
View article: Fig. S3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S3 shows flow cytometry data for CD45, gating strategy and transcript levels of CD68 and TLR4
View article: Supplementary Table 1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of Differentially Expressed Genes from RNA Seq analysis
View article: Fig. S5 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S5 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S5 shows images of liver nodules, transcript levels of AFP
View article: Fig. S2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S2 shows body weight changes, liver weight changes, Transcript levels of Il1b and H&E staining for showing KC population
View article: Fig. S7 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S7 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S7 shows western blots for p-AKT/AKT, p-S6/S6, b tubulin, p-ERK/ERK, P-p-38/p-38, b catenin, p62 and b tubulin
View article: Fig. S7 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S7 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S7 shows western blots for p-AKT/AKT, p-S6/S6, b tubulin, p-ERK/ERK, P-p-38/p-38, b catenin, p62 and b tubulin
View article: Fig. S2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S2 shows body weight changes, liver weight changes, Transcript levels of Il1b and H&E staining for showing KC population
View article: Supplementary Table 3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of primers used for quantitative real time PCR
View article: Supplementary Table 1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of Differentially Expressed Genes from RNA Seq analysis
View article: Fig. S3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S3 shows flow cytometry data for CD45, gating strategy and transcript levels of CD68 and TLR4
View article: Supplementary Table 3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 3 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of primers used for quantitative real time PCR
View article: Supplementary Table 2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Table 2 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
List of significantly altered transcripts from RNA Seq analysis
View article: Fig. S1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S1 shows the experimental plan of the study, IHC staining for Ripk3, Transcript levels of Mlkl and Ripk3, Western blots for Cleaved and Total Caspase 3
View article: Fig. S6 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S6 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S6 shows ingenuity pathway analysis for canonical pathways and upstream regulators
View article: Fig. S1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S1 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S1 shows the experimental plan of the study, IHC staining for Ripk3, Transcript levels of Mlkl and Ripk3, Western blots for Cleaved and Total Caspase 3
View article: Supplementary Data Legends from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Supplementary Data Legends from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Supplementary Figures 1 -7 and Supplementary Tables 1 - 3 legends
View article: Fig. S4 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S4 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Fig S4 shows transcript levels of Col1a1, western blots for Desmin, a-SMA and b-tubulin, Transcript levels of TGFb
View article: Fig. S5 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Fig. S5 from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Figure S5 shows images of liver nodules, transcript levels of AFP
View article: Data from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis
Data from Absence of either Ripk3 or Mlkl reduces incidence of hepatocellular carcinoma independent of liver fibrosis Open
Nonalcoholic fatty liver disease (NAFLD) is one of the etiologies that contribute to hepatocellular carcinoma (HCC), and chronic inflammation is one of the proposed mediators of HCC. Since necroptosis is a cell death pathway that induces i…
View article: Non-Necroptotic Roles of MLKL in Diet-Induced Obesity, Liver Pathology, and Insulin Sensitivity: Insights from a High-Fat, High-Fructose, High-Cholesterol Diet Mouse Model
Non-Necroptotic Roles of MLKL in Diet-Induced Obesity, Liver Pathology, and Insulin Sensitivity: Insights from a High-Fat, High-Fructose, High-Cholesterol Diet Mouse Model Open
Chronic inflammation is a key player in metabolic dysfunction-associated fatty liver disease (MAFLD) progression. Necroptosis, an inflammatory cell death pathway, is elevated in MAFLD patients and mouse models, yet its role is unclear due …
View article: Non-Necroptotic Roles of MLKL in Diet-Induced Obesity, Liver Pathology, and Insulin Sensitivity: Insights from a High Fat, High Fructose, High Cholesterol Diet Mouse Model
Non-Necroptotic Roles of MLKL in Diet-Induced Obesity, Liver Pathology, and Insulin Sensitivity: Insights from a High Fat, High Fructose, High Cholesterol Diet Mouse Model Open
Chronic inflammation is a key player in metabolic dysfunction-associated fatty liver disease (MAFLD) progression. Necroptosis, an inflammatory cell death pathway, is elevated in MAFLD patients and mouse models, yet its role is unclear due …
View article: [Diagnostic value of low versus high titers of MOG-IgG and their clinical implications in myelin oligodendrocyte glycoprotein antibody-associated disease: a retrospective singlecenter study].
[Diagnostic value of low versus high titers of MOG-IgG and their clinical implications in myelin oligodendrocyte glycoprotein antibody-associated disease: a retrospective singlecenter study]. Open
Low titers and high titers of MOG-IgG have similar diagnostic value for MOGAD. The serum titers of MOG-IgG are probably associated with the disease severity and stress levels of the patients with MOGAD.
View article: Fig. S6 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis
Fig. S6 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis Open
Fig S6 shows ingenuity pathway analysis for canonical pathways and upstream regulators
View article: Fig. S4 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis
Fig. S4 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis Open
Fig S4 shows transcript levels of Col1a1, western blots for Desmin, a-SMA and b-tubulin, Transcript levels of TGFb
View article: Supplementary Table 3 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis
Supplementary Table 3 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis Open
List of primers used for quantitative real time PCR
View article: Fig. S4 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis
Fig. S4 from Absence of Either <i>Ripk3</i> or <i>Mlkl</i> Reduces Incidence of Hepatocellular Carcinoma Independent of Liver Fibrosis Open
Fig S4 shows transcript levels of Col1a1, western blots for Desmin, a-SMA and b-tubulin, Transcript levels of TGFb