Jonathan Krystkiewicz
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View article: Supplemental figures from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Supplemental figures from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplemental data file contains Table S1, S2 and Figures S1-S7
View article: Data from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Data from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Evolutionarily conserved selenoprotein O (SELENOO) catalyzes a posttranslational protein modification known as AMPylation that is essential for the oxidative stress response in bacteria and yeast. Given that oxidative stress experienced in…
View article: Table S3 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Table S3 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Data table of AMPylated substrates identified by mass spectrometry analysis.
View article: Figure S6 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S6 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S6 depicts markers of subcellular fractionation used to isolate crude mitochondria.
View article: Figure S2 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S2 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S2 correlates SelenoO expression with survival and metastasis
View article: Figure S1 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S1 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S1 shows expressions of SelenoO in cancer subtypes
View article: Figure S7 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S7 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S7 shows mass spectra of SdhA and Aco2
View article: Figure S4 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S4 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S4 depicts characterization of SelenoO deficient cell lines
View article: Figure S3 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S3 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S3 depicts expression of SelenoO in cell lines used in this study
View article: Figure S5 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Figure S5 from Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Supplementary Figure S5 shows redox metabolite levels in wildtype and SelenoO deficient cell lines.
View article: Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity
Selenoprotein O Promotes Melanoma Metastasis and Regulates Mitochondrial Complex II Activity Open
Evolutionarily conserved selenoprotein O (SELENOO) catalyzes a posttranslational protein modification known as AMPylation that is essential for the oxidative stress response in bacteria and yeast. Given that oxidative stress experienced in…
View article: Pathogenic mitochondrial DNA mutations inhibit melanoma metastasis
Pathogenic mitochondrial DNA mutations inhibit melanoma metastasis Open
Mitochondrial DNA (mtDNA) mutations are frequent in cancer, yet their precise role in cancer progression remains debated. To functionally evaluate the impact of mtDNA variants on tumor growth and metastasis, we developed an enhanced cytopl…
View article: Short-term post-fast refeeding enhances intestinal stemness via polyamines
Short-term post-fast refeeding enhances intestinal stemness via polyamines Open