Jueheng Wu
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View article: ZIKV infection causes placental inflammation through activating PANoptosis
ZIKV infection causes placental inflammation through activating PANoptosis Open
Infection by the Zika virus (ZIKV) during pregnancy can cause congenital Zika syndrome (CZS) or other central nervous system conditions in infants, underscoring the importance of understanding the role of ZIKV-induced placental damage in t…
View article: Data from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma
Data from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma Open
Early-stage lung adenocarcinoma generally has a favorable prognosis. However, more than 30% of early-stage lung adenocarcinoma cases relapse within 5 years of initial treatment, even after complete removal of the primary tumor. Identificat…
View article: Supplementary Data from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma
Supplementary Data from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma Open
Supplementary Figure 1 to 7, and Supplementary Materials and Methods
View article: Supplementary Table 1 from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma
Supplementary Table 1 from The Glycosyltransferase XYLT1 Activates NF-κB Signaling to Promote Metastasis of Early-Stage Lung Adenocarcinoma Open
Supplementary Table 1. Clinical Information of Tissues from Early-Stage LUAD Patients
View article: The impact of glucose metabolism on inflammatory processes in sepsis-induced acute lung injury
The impact of glucose metabolism on inflammatory processes in sepsis-induced acute lung injury Open
Acute lung injury (ALI) is a prevalent and critical complication of sepsis, marked by high incidence and mortality rates, with its pathogenesis still not being fully elucidated. Recent research has revealed a significant correlation betwee…
View article: Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer
Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer Open
Purpose: The present study was to examine the effect of sphingosine kinase-1 (SPHK1) on chemotherapeutics-induced apoptosis in non–small cell lung cancer (NSCLC) cells, which is relatively insensitive to chemotherapy, and its clinical sign…
View article: Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer
Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer Open
Purpose: The present study was to examine the effect of sphingosine kinase-1 (SPHK1) on chemotherapeutics-induced apoptosis in non–small cell lung cancer (NSCLC) cells, which is relatively insensitive to chemotherapy, and its clinical sign…
View article: Supplementary Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer
Supplementary Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer Open
Supplementary Figures S1-S5; Supplementary Tables S1-S4.
View article: Supplementary Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer
Supplementary Data from Sphingosine Kinase-1 Enhances Resistance to Apoptosis through Activation of PI3K/Akt/NF-κB Pathway in Human Non–Small Cell Lung Cancer Open
Supplementary Figures S1-S5; Supplementary Tables S1-S4.
View article: LincRNA01703 Facilitates CD81+ Exosome Secretion to Inhibit Lung Adenocarcinoma Metastasis via the Rab27a/SYTL1/CD81 Complex
LincRNA01703 Facilitates CD81+ Exosome Secretion to Inhibit Lung Adenocarcinoma Metastasis via the Rab27a/SYTL1/CD81 Complex Open
Metastasis, a major cause of cancer-related mortality worldwide, frequently occurs early in the diagnosis of lung adenocarcinoma (LUAD). However, the precise molecular mechanisms governing the aggressive metastatic behavior of LUAD remain …
View article: Epidemiology, genetic characteristics, and association with meteorological factors of human metapneumovirus infection in children in southern China: A 10-year retrospective study
Epidemiology, genetic characteristics, and association with meteorological factors of human metapneumovirus infection in children in southern China: A 10-year retrospective study Open
HMPV is an important respiratory pathogen that causes acute respiratory tract infections in children in southern China, particularly in children ≤5 years old. The prevalence peak of HMPV in this area appeared in spring, and the predominant…
View article: m<sup>6</sup>A‐Dependent Modulation via IGF2BP3/MCM5/Notch Axis Promotes Partial EMT and LUAD Metastasis
m<sup>6</sup>A‐Dependent Modulation via IGF2BP3/MCM5/Notch Axis Promotes Partial EMT and LUAD Metastasis Open
The importance of mRNA N6‐methyladenosine (m 6 A) modification during tumor metastasis is controversial as it plays distinct roles in different biological contexts. Moreover, how cancer cell plasticity is shaped by m 6 A modification is in…
View article: PHF14 enhances DNA methylation of SMAD7 gene to promote TGF-β-driven lung adenocarcinoma metastasis
PHF14 enhances DNA methylation of SMAD7 gene to promote TGF-β-driven lung adenocarcinoma metastasis Open
Aberrant activation of TGF-β signaling plays a pivotal role in cancer metastasis and progression. However, molecular mechanisms underlying the dysregulation of TGF-β pathway remain to be understood. Here, we found that SMAD7, a direct down…
View article: Supplementary Figure 2 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Supplementary Figure 2 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
Overexpression of miR-452 reduces glioma stem-like traits.
View article: Supplementary Figure 3 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 3 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 231 KB, Overexpression of miR-138 inhibits the malignant properties of ESCC cells.
View article: Supplementary Figure 4 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 4 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 39 KB, Silencing miR-138 sustains NF-�B activity.
View article: Supplementary Figure 1 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 1 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 25 KB, miR-138 expression is downregulated in ESCC.
View article: Supplementary Figure 5 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Supplementary Figure 5 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
Overexpression of miR-452 decreases glioma cell tumorigenesis in vivo.
View article: Supplementary Figure 7 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 7 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 21 KB, miR-138 directly suppresses multiple lipid rafts components.
View article: Supplementary Figure 2 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 2 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 234 KB, Downregulation of miR-138 promotes the aggressiveness of ESCC cells in vitro.
View article: Supplementary Figure 3 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Supplementary Figure 3 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
miR-452 directly targets Bmi-1, LEF1 and TCF4.
View article: Supplementary Figure 3 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 3 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 231 KB, Overexpression of miR-138 inhibits the malignant properties of ESCC cells.
View article: Supplementary Information from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Information from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 173 KB, Supplementary Materials and Methods, Supplementary Figure Legends, and Supplementary References
View article: Supplementary Figure 1 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 1 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 25 KB, miR-138 expression is downregulated in ESCC.
View article: Supplementary Figure 7 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 7 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 21 KB, miR-138 directly suppresses multiple lipid rafts components.
View article: Data from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Data from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
Purpose: miR-452 is reported to be required for neural crest stem cell differentiation during neural crest development. However, the biologic role of miR-452 in gliomas remains unclear. The aim of the present study was to evaluate the effe…
View article: Supplementary Figure 2 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Supplementary Figure 2 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
Overexpression of miR-452 reduces glioma stem-like traits.
View article: Supplementary Figure 1 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas
Supplementary Figure 1 from Downregulation of miR-452 Promotes Stem-Like Traits and Tumorigenicity of Gliomas Open
Reduced miR-452 expression in gliomas correlates with poor prognosis.
View article: Supplementary Figure 5 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma
Supplementary Figure 5 from Downregulation of miR-138 Sustains NF-κB Activation and Promotes Lipid Raft Formation in Esophageal Squamous Cell Carcinoma Open
PDF file - 88 KB, Blockade of the NF-�B pathway via overexpressing I�B�-mut inhibits miR-138 downregulation-induced ESCC progression.