Juliette Hamroune
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View article: The epigenomic landscape of bronchial epithelial cells reveals the establishment of trained immunity
The epigenomic landscape of bronchial epithelial cells reveals the establishment of trained immunity Open
Innate immune memory, also called trained immunity, refers to the ability of innate immune cells to gain memory characteristics after transient stimulation, resulting in a nonspecific modified inflammatory response upon secondary remote ch…
View article: DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
View article: The super-enhancer landscape reflects molecular subgroups of adrenocortical carcinoma
The super-enhancer landscape reflects molecular subgroups of adrenocortical carcinoma Open
Adrenocortical carcinoma (ACC) is a rare cancer of the adrenal gland with generally very unfavourable outcome. Two molecular subgroups, C1A and C1B, have been previously identified with a significant association with patient survival. In t…
View article: DNA ultra-sensitive quantification, a technology for studying HIV unintegrated linear DNA
DNA ultra-sensitive quantification, a technology for studying HIV unintegrated linear DNA Open
View article: Additional file 8 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 8 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 8: Table S7 related to content in Fig. 4: Differential expression and methylation analysis as a result of sample treatment with AZA (5-azacytidine) in HUVEC.
View article: Additional file 3 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 3 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 3: Table S2 related to content in Methods: Partial correlation analysis for modeling interactions between increased cell proliferation, expression of DNMTs and CIMP status (TCGA data set). DNMT expression is measured in log…
View article: Additional file 5 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 5 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 5: Table S4 related to content in Fig. 4: Differential expression analysis using DESeq2 as a result of sample treatment with AZA (5-azacytidine).
View article: Additional file 4 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 4 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 4: Table S3 related to content in Fig. 3: Characteristics of the ACC cohort used to perform immunohistochemistry experiments using CD3 as a marker of T cells including 6 hCIMP and 6 lCIMP tumors.
View article: Additional file 6 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 6 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 6: Table S5 related to content in Fig. 4: Differential expression and methylation analysis as a result of sample treatment with AZA (5-azacytidine) in H295R.
View article: Additional file 2 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 2 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 2: Table S1 related to content in Fig. 1: Genes coding for factors known to be involved in DNA methylation or demethylation.
View article: Additional file 9 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 9 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 9: Table S8 related to content in Fig. 4: Pathways enrichment analysis on genes exhibiting significantly lower promoter methylation and significantly higher expression upon 5 µM 5-azacytidine treatment.
View article: Additional file 10 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 10 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 10: Table S9 related to content in Fig. 4: Short description of genes common between those induced by AZA through promoter demethylation and those repressed in hCIMP tumors through promoter methylation.
View article: Additional file 7 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 7 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 7: Table S6 related to content in Fig. 4: Differential expression and methylation analysis as a result of sample treatment with AZA (5-azacytidine) in MUC1.
View article: Additional file 12 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 12 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 12: Table S11 related to content in Methods: Demographic and clinical data of the ACC TCGA cohort.
View article: Additional file 13 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 13 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 13: Table S12 related to content in Methods: Demographic and clinical data of the ENSAT TCGA cohort.
View article: Additional file 11 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma
Additional file 11 of DNA hypermethylation driven by DNMT1 and DNMT3A favors tumor immune escape contributing to the aggressiveness of adrenocortical carcinoma Open
Additional file 11: Table S10 related to content in Fig. 4: Pathways enrichment analysis on genes exhibiting significantly higher promoter methylation and significantly lower expression in the H295R than in the MUC1 cells.
View article: PP 1.39 – 00086 The level of cell activation is associated with the pre-integrative latency of HIV linear DNA
PP 1.39 – 00086 The level of cell activation is associated with the pre-integrative latency of HIV linear DNA Open
View article: Transcriptome and Methylome Analysis Reveal Complex Cross-Talks between Thyroid Hormone and Glucocorticoid Signaling at Xenopus Metamorphosis
Transcriptome and Methylome Analysis Reveal Complex Cross-Talks between Thyroid Hormone and Glucocorticoid Signaling at Xenopus Metamorphosis Open
Background: Most work in endocrinology focus on the action of a single hormone, and very little on the cross-talks between two hormones. Here we characterize the nature of interactions between thyroid hormone and glucocorticoid signaling d…
View article: Autoimmunity affecting the biliary tract fuels the immunosurveillance of cholangiocarcinoma
Autoimmunity affecting the biliary tract fuels the immunosurveillance of cholangiocarcinoma Open
Cholangiocarcinoma (CCA) results from the malignant transformation of cholangiocytes. Primary sclerosing cholangitis (PSC) and primary biliary cholangitis (PBC) are chronic diseases in which cholangiocytes are primarily damaged. Although P…
View article: Dynamic methylation of histone H3K18 in differentiating Theileria parasites
Dynamic methylation of histone H3K18 in differentiating Theileria parasites Open
View article: A genetic variant controls interferon-β gene expression in human myeloid cells by preventing C/EBP-β binding on a conserved enhancer
A genetic variant controls interferon-β gene expression in human myeloid cells by preventing C/EBP-β binding on a conserved enhancer Open
Interferon β (IFN-β) is a cytokine that induces a global antiviral proteome, and regulates the adaptive immune response to infections and tumors. Its effects strongly depend on its level and timing of expression. Therefore, the transcripti…
View article: Dynamic methylation of histone H3K18 in differentiating Theileria parasites
Dynamic methylation of histone H3K18 in differentiating Theileria parasites Open
Lysine methylation on histone tails impacts genome regulation and cell fate determination in many developmental processes. Apicomplexa intracellular parasites cause major diseases and they have developed complex life cycles with fine-tuned…
View article: Regulated expression and function of the GABAB receptor in human pancreatic beta cell line and islets
Regulated expression and function of the GABAB receptor in human pancreatic beta cell line and islets Open
G protein-coupled receptors are seven transmembrane signaling molecules that are involved in a wide variety of physiological processes. They constitute a large protein family of receptors with almost 300 members detected in human pancreati…
View article: GPR50-Ctail cleavage and nuclear translocation: a new signal transduction mode for G protein-coupled receptors
GPR50-Ctail cleavage and nuclear translocation: a new signal transduction mode for G protein-coupled receptors Open
View article: A novel, ultra-sensitive technology for quantifying the HIV unintegrated linear DNA responsible for pre-integrative latency
A novel, ultra-sensitive technology for quantifying the HIV unintegrated linear DNA responsible for pre-integrative latency Open