Kylie A. McLaughlin
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View article: Supplementary Figure Legends from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure Legends from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Figure legends for Supplementary Figures
View article: Data from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Data from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Resistance to radiotherapy due to insufficient cancer cell death is a significant cause of treatment failure in non–small cell lung cancer (NSCLC). The endogenous caspase-8 inhibitor FLIP is a critical regulator of cell death that is frequ…
View article: Supplementary Figure 1 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 1 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Response of H460 and A549 cells to IR
View article: Supplementary Figure Legends from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure Legends from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Figure legends for Supplementary Figures
View article: Supplementary Figure 3 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 3 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
IR-induced changes in cell surface expression of death receptors
View article: Supplementary Figure 3 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 3 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
IR-induced changes in cell surface expression of death receptors
View article: Supplementary Figure 2 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 2 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Caspase-8 dependent apoptosis induced by IR
View article: Supplementary Figure 4 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 4 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Combination of SAHA with IR in H460 and A549 cells using different treatment schedules
View article: Supplementary Figure 1 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 1 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Response of H460 and A549 cells to IR
View article: Supplementary Figure 4 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 4 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Combination of SAHA with IR in H460 and A549 cells using different treatment schedules
View article: Supplementary Figure 2 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Supplementary Figure 2 from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Caspase-8 dependent apoptosis induced by IR
View article: Data from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer
Data from FLIP: A Targetable Mediator of Resistance to Radiation in Non–Small Cell Lung Cancer Open
Resistance to radiotherapy due to insufficient cancer cell death is a significant cause of treatment failure in non–small cell lung cancer (NSCLC). The endogenous caspase-8 inhibitor FLIP is a critical regulator of cell death that is frequ…
View article: Targeting nucleotide metabolism enhances the efficacy of anthracyclines and anti-metabolites in triple-negative breast cancer
Targeting nucleotide metabolism enhances the efficacy of anthracyclines and anti-metabolites in triple-negative breast cancer Open
View article: Metadata record for the manuscript: Targeting nucleotide metabolism enhances the efficacy of anthracyclines and anti-metabolites in triple-negative breast cancer
Metadata record for the manuscript: Targeting nucleotide metabolism enhances the efficacy of anthracyclines and anti-metabolites in triple-negative breast cancer Open
Summary This metadata record provides details of the data supporting the claims of the related manuscript: “Targeting nucleotide metabolism enhances the efficacy of anthracyclines and anti-metabolites in triple-negative breast cance…