Libing Song
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View article: PPM1G promotes chemoresistance in triple negative breast cancer by enhancing YAP signaling
PPM1G promotes chemoresistance in triple negative breast cancer by enhancing YAP signaling Open
Triple-negative breast cancer (TNBC) is an aggressive subtype of breast cancer with the poorest prognosis. The frequent development of chemoresistance is a major therapeutic challenge, yet the underlying mechanisms remain unclear. Here, we…
View article: PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis
PRODH2-Mediated Metabolism in the Bone Microenvironment Promotes Breast Cancer Metastasis Open
Breast cancer frequently metastasizes to the bone, but treatment options for bone metastatic breast cancer are limited. Amino acid metabolism is reprogrammed in the bone metastatic microenvironment, suggesting that it could represent a the…
View article: The Involvement of RIPK1 in Alopecia Areata
The Involvement of RIPK1 in Alopecia Areata Open
We have previously demonstrated that receptor-interacting serine threonine kinase 1 (RIPK1) is expressed in hair follicles and regulates the hair cycle. In a mouse model, RIPK1 inhibitors also accelerated the telogen-to-anagen transition a…
View article: RNF31 induces paclitaxel resistance by sustaining ALYREF cytoplasmic–nuclear shuttling in human triple‐negative breast cancer
RNF31 induces paclitaxel resistance by sustaining ALYREF cytoplasmic–nuclear shuttling in human triple‐negative breast cancer Open
Background Resistance to paclitaxel‐based chemotherapy is the major obstacle in triple‐negative breast cancer (TNBC) treatment. However, overcoming paclitaxel resistance remains an unsolved problem. The present study aimed to determine whe…
View article: Targeting the SPC25/RIOK1/MYH9 Axis to Overcome Tumor Stemness and Platinum Resistance in Epithelial Ovarian Cancer
Targeting the SPC25/RIOK1/MYH9 Axis to Overcome Tumor Stemness and Platinum Resistance in Epithelial Ovarian Cancer Open
In epithelial ovarian cancer (EOC), platinum resistance, potentially mediated by cancer stem cells (CSCs), often leads to relapse and treatment failure. Here, the role of spindle pole body component 25 (SPC25) as a key determinant promotin…
View article: Supplementary Figures and Legends, Supplementary Tables from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis
Supplementary Figures and Legends, Supplementary Tables from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis Open
S1. BASP1 downregulation is associated with poor prognosis in glioma patients. S2. Low expression of BASP1 is associated with poor prognosis in CGGA dataset. S3. Dysregulation of BASP1 alters chemoresistance of glioma cells to TMZ treatmen…
View article: Supplementary Figures and Legends, Supplementary Tables from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis
Supplementary Figures and Legends, Supplementary Tables from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis Open
S1. BASP1 downregulation is associated with poor prognosis in glioma patients. S2. Low expression of BASP1 is associated with poor prognosis in CGGA dataset. S3. Dysregulation of BASP1 alters chemoresistance of glioma cells to TMZ treatmen…
View article: Data from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis
Data from Downregulation of BASP1 promotes temozolomide resistance in gliomas via epigenetic activation of the FBXO32/NF-κB/MGMT axis Open
The chemoresistance of temozolomide (TMZ)-based therapy is a serious limitation for lasting effective treatment of gliomas, while the underlying mechanisms remain unclear. In this study, we showed that downregulation of BASP1 correlated ne…
View article: Supplementary Data 1 from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma
Supplementary Data 1 from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma Open
Supplementary material
View article: Data from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma
Data from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma Open
Purpose:Microvascular invasion (MVI) is a major unfavorable prognostic factor for intrahepatic metastasis and postoperative recurrence of hepatocellular carcinoma (HCC). However, the intervention and preoperative prediction for MVI remain …
View article: Supplementary Data 1 from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma
Supplementary Data 1 from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma Open
Supplementary material
View article: Data from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma
Data from Infiltrative Vessel Co-optive Growth Pattern Induced by IQGAP3 Overexpression Promotes Microvascular Invasion in Hepatocellular Carcinoma Open
Purpose:Microvascular invasion (MVI) is a major unfavorable prognostic factor for intrahepatic metastasis and postoperative recurrence of hepatocellular carcinoma (HCC). However, the intervention and preoperative prediction for MVI remain …
View article: Special RCA based sensitive point‐of‐care detection of HPV mRNA for cervical cancer screening
Special RCA based sensitive point‐of‐care detection of HPV mRNA for cervical cancer screening Open
Developing the sensitive point‐of‐care testing (POCT) of oncogenic nucleic acids from human papillomavirus (HPV) infection is essential in preventing cervical cancer, especially in resource‐limited settings. Rolling circle amplification (R…
View article: Supplementary Tables S4 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S4 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
The original sequencing results of all differentially expressed circRNAs in SCP2 and MDA-MB-231 parental cells
View article: Supplementary Information from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Information from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Supplementary Materials and Methods and Supplemental Figures and Figure legends
View article: Supplementary Tables S5 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S5 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Potential trans-regulatory factors were enriched on the S100A4 and LGALS3 promoter in MDA-MB-231/circMMP2(6, 7) cells
View article: Supplementary Tables S5 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S5 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Potential trans-regulatory factors were enriched on the S100A4 and LGALS3 promoter in MDA-MB-231/circMMP2(6, 7) cells
View article: Data from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Data from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
The bone is the most common site of distant metastasis of breast cancer, which leads to serious skeletal complications and mortality. Understanding the mechanisms underlying breast cancer bone metastasis would provide potential strategies …
View article: Supplementary Tables S2 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S2 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Univariate and Multivariate Analyses of Various Prognostic Parameters in Breast Cancer Patients by Cox-regression Analysis
View article: Supplementary Tables S1 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S1 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Clinicopathological Characteristics of Clinical Samples and Expression of CircMMP2(6, 7) and PRMT5 in Breast Cancer
View article: Supplementary Tables S3 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S3 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Primers, Probes, siRNA, shRNA and ASOs oligonucleotides
View article: Supplementary Tables S1 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S1 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Clinicopathological Characteristics of Clinical Samples and Expression of CircMMP2(6, 7) and PRMT5 in Breast Cancer
View article: Supplementary Tables S3 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S3 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Primers, Probes, siRNA, shRNA and ASOs oligonucleotides
View article: Supplementary Tables S2 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis
Supplementary Tables S2 from CircMMP2(6,7) Cooperates with β-Catenin and PRMT5 to Disrupt Bone Homeostasis and Promote Breast Cancer Bone Metastasis Open
Univariate and Multivariate Analyses of Various Prognostic Parameters in Breast Cancer Patients by Cox-regression Analysis