Liqin Du
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View article: Impact of Drinking Water Supplemented with Complex Acidifiers on Production Performance, Egg Quality, Physiological and Biochemical Indicators, and Microbial Flora of BIAN Chickens
Impact of Drinking Water Supplemented with Complex Acidifiers on Production Performance, Egg Quality, Physiological and Biochemical Indicators, and Microbial Flora of BIAN Chickens Open
This study investigated the effects of dietary supplementation with composite acidifying agents containing 2-hydroxy-4-methylthiobutyric acid (≥30.0%), lactic acid (≥24.2%), and phosphoric acid (≥23.8%) on production performance, egg quali…
View article: Comparative analysis of meat quality and flavor profiles between Bian chicken and Cobb broiler using FTIR and GC×GC-TOF MS technologies
Comparative analysis of meat quality and flavor profiles between Bian chicken and Cobb broiler using FTIR and GC×GC-TOF MS technologies Open
This study systematically compared meat quality characteristics and flavor profiles between the local Shanxi breed Bian chicken (BIAN) and the commercial Cobb broiler (CB) using a comprehensive analytical approach. In terms of physical pro…
View article: Comparison of the Metabolic and Flavor Characteristics of the Egg Yolks of BIAN Chicken and Hy-Line Brown Chicken Using LC-MS and GC × GC-TOF MS Techniques
Comparison of the Metabolic and Flavor Characteristics of the Egg Yolks of BIAN Chicken and Hy-Line Brown Chicken Using LC-MS and GC × GC-TOF MS Techniques Open
Objectives: This study systematically compared the differences in egg quality between the BIAN chicken, an indigenous breed of Shanxi Province, and the Hy-Line Brown, a commercial breed, through the integration of non-targeted metabolomics…
View article: Effects of Substitution of Wheat Straw by Giant Reed on Growth Performance, Serum Biochemical Parameters, Nutrient Digestibility, and Antioxidant Properties of Sheep
Effects of Substitution of Wheat Straw by Giant Reed on Growth Performance, Serum Biochemical Parameters, Nutrient Digestibility, and Antioxidant Properties of Sheep Open
The development and utilization of unconventional forage resources is crucial to alleviating the current situation of shortage of forage resources. Giant reed (Arundo donax) is a promising forage resource from the Poaceae family, one of th…
View article: Emerging pathogens <i>Aerococcus urinae</i> and <i>Aerococcus sanguinicola</i> from a Canadian tertiary care hospital
Emerging pathogens <i>Aerococcus urinae</i> and <i>Aerococcus sanguinicola</i> from a Canadian tertiary care hospital Open
Background: Aerococcus urinae and Aerococcus sanguinicola are emerging pathogens linked with urinary tract infections. We present a case series of A. urinae and A. sanguinicola isolates characterizing the…
View article: Using glycerol-inducible expression system to overexpressed maltooligosaccharide-forming α-amylase in Bacillus subtilis
Using glycerol-inducible expression system to overexpressed maltooligosaccharide-forming α-amylase in Bacillus subtilis Open
In order to meet the desire of maltopentaose (G5) in industrial application, we developed a glycerol-inducible expression system in Bacillus subtilis to overexpress maltooligosaccharide-forming α-amylase from Bacillus cereus ATCC 14579 (Bc…
View article: Discovery of a Novel β-xylosidase with Xylanase Activity and Its Application in the Production of Xylitol from Corncob Xylan
Discovery of a Novel β-xylosidase with Xylanase Activity and Its Application in the Production of Xylitol from Corncob Xylan Open
Although β-xylosidases with xylanase activity are preferential for the hydrolysis of xylan and production of xylitol, reports on their use are scarce. In this study, a multifunctional β-xylosidase (XYL4) was identified. In addition to β-xy…
View article: Highly efficient bioconversion of icariin to icaritin by whole-cell catalysis
Highly efficient bioconversion of icariin to icaritin by whole-cell catalysis Open
View article: Supplementary Table 4 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 4 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S4. PANTHER and mSigDB analysis of siELF4 down- and up-regulated genes.
View article: Supplementary Table 1 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 1 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S1. Cherry picker siRNA list and Primer pairs utilized for qRT-PCR.
View article: Supplementary Table 7 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 7 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S7. mSigDB analysis of overlap between miR-124 targets and siELF4 downregulated genes.
View article: Supplementary Table 1 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 1 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S1. Cherry picker siRNA list and Primer pairs utilized for qRT-PCR.
View article: Supplementary Figure Legends from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Figure Legends from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Figure S1. Knockdown efficiency 48 h after reverse transfection. Figure S2. miR-124 directly binds ELF4 and regulates ELF4 expression. Figure S3. ELF4 ectopic expression antagonizes the effect of miR-124 on differentiation of KELLY cells. …
View article: Supplementary Table 2 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 2 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S2. siELF4 differentially expressed genes.
View article: Supplementary Table 5 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 5 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S5. miR-124 target list (mNSC Study and miRTarBase).
View article: Supplementary Table 4 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 4 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S4. PANTHER and mSigDB analysis of siELF4 down- and up-regulated genes.
View article: Supplementary Table 7 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 7 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S7. mSigDB analysis of overlap between miR-124 targets and siELF4 downregulated genes.
View article: Supplementary Table 2 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 2 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S2. siELF4 differentially expressed genes.
View article: Supplementary Figure Legends from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Figure Legends from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Figure S1. Knockdown efficiency 48 h after reverse transfection. Figure S2. miR-124 directly binds ELF4 and regulates ELF4 expression. Figure S3. ELF4 ectopic expression antagonizes the effect of miR-124 on differentiation of KELLY cells. …
View article: Supplementary Table 3 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 3 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S3. Survival analysis of Versteeg and Seeger cohorts.
View article: Supplementary Table 3 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 3 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S3. Survival analysis of Versteeg and Seeger cohorts.
View article: Data from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Data from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
13-Cis-retinoic acid (RA) is typically used in postremission maintenance therapy in patients with neuroblastoma. However, side effects and recurrence are often observed. We investigated the use of miRNAs as a strategy to replace RA …
View article: Supplementary Table 6 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 6 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S6. Overlap between miR-124 targets and siELF4 down-regulated genes.
View article: Supplementary Table 6 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 6 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S6. Overlap between miR-124 targets and siELF4 down-regulated genes.
View article: Supplementary Table 5 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Supplementary Table 5 from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
Table S5. miR-124 target list (mNSC Study and miRTarBase).
View article: Data from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State
Data from <i>ELF4</i> Is a Target of miR-124 and Promotes Neuroblastoma Proliferation and Undifferentiated State Open
13-Cis-retinoic acid (RA) is typically used in postremission maintenance therapy in patients with neuroblastoma. However, side effects and recurrence are often observed. We investigated the use of miRNAs as a strategy to replace RA …
View article: Supplementary Figure 3 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling
Supplementary Figure 3 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling Open
Supplementary Figure 3. Transcriptional profiling of HBEC30KT and HBEC30KT-shTP53 identifies context-dependent de-repression of the miR-17~92 targetome after p53 loss driven by target genes of the miR-17-92 polycistron.
View article: Data from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling
Data from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling Open
Lung cancer is the leading cause of cancer-related fatalities. Recent success developing genotypically targeted therapies, with potency only in well-defined subpopulations of tumors, suggests a path to improving patient survival. We used a…
View article: Supplementary Figure 1 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling
Supplementary Figure 1 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling Open
Supplementary Figure 1. Activity of miR-92a related inhibitors.
View article: Supplementary Figure 1 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling
Supplementary Figure 1 from Genetic Mutation of p53 and Suppression of the miR-17∼92 Cluster Are Synthetic Lethal in Non–Small Cell Lung Cancer due to Upregulation of Vitamin D Signaling Open
Supplementary Figure 1. Activity of miR-92a related inhibitors.