Luana Casetti
YOU?
Author Swipe
View article: Supplementary Figures 1 - 5 from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Figures 1 - 5 from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 589K, shS5A, shS5B and shS5A/B differentially affect STAT5 expression in CML and normal stem/progenitor cells (S1); Characteristics of CML cells following addition of the indicated shRNA encoding lentiviral particles (S2); shS5A…
View article: Supplementary Figure Legends from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Figure Legends from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 32K
View article: Data from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Data from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
STAT5 fulfills essential roles in hematopoietic stem cell (HSC) self-renewal and chronic myeloid leukemia (CML), a prototypical stem cell malignancy. However, the specific contributions of the two related genes STAT5A and STAT5B have not b…
View article: Supplementary Figures 1 - 5 from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Figures 1 - 5 from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 589K, shS5A, shS5B and shS5A/B differentially affect STAT5 expression in CML and normal stem/progenitor cells (S1); Characteristics of CML cells following addition of the indicated shRNA encoding lentiviral particles (S2); shS5A…
View article: Supplementary Methods from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Methods from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 34K, Includes lentiviral constructs and transduction, immunoblots and antibodies, RNA extraction data, and supplementary references.
View article: Data from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Data from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
STAT5 fulfills essential roles in hematopoietic stem cell (HSC) self-renewal and chronic myeloid leukemia (CML), a prototypical stem cell malignancy. However, the specific contributions of the two related genes STAT5A and STAT5B have not b…
View article: Supplementary Methods from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Methods from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 34K, Includes lentiviral constructs and transduction, immunoblots and antibodies, RNA extraction data, and supplementary references.
View article: Supplementary Figure Legends from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells
Supplementary Figure Legends from Differential Contributions of STAT5A and STAT5B to Stress Protection and Tyrosine Kinase Inhibitor Resistance of Chronic Myeloid Leukemia Stem/Progenitor Cells Open
PDF file - 32K
View article: Hematopoietic niche drives FLT3-ITD acute myeloid leukemia resistance to quizartinib <i>via</i> STAT5-and hypoxia-dependent upregulation of AXL
Hematopoietic niche drives FLT3-ITD acute myeloid leukemia resistance to quizartinib <i>via</i> STAT5-and hypoxia-dependent upregulation of AXL Open
Internal tandem duplication in Fms-like tyrosine kinase 3 (FLT3-ITD) is the most frequent mutation observed in acute myeloid leukemia (AML) and correlates with poor prognosis. FLT3 tyrosine kinase inhibitors are promising for targeted ther…